Biography and Research Information
OverviewAI-generated summary
David S. Henry's research investigates physiological mechanisms across various biological systems. His work includes studies on the disruption of lymph vessel contractions and lymph flow in hypertensive rats, as well as the role of interleukin-1 receptor antagonist in inhibiting arthrofibrosis in a post-traumatic knee immobilization model. Henry has also examined the implications of beta-blocker therapy and collateral artery function in rat models of ischemic stroke. Additionally, his research has assessed the retinal toxicity and sealing strength of tissue adhesives.
His publications also extend to the potential link between phosphodiesterase-5 inhibitor use and Alzheimer's disease and related dementias in older adults, suggesting the need for further clinical trials. Henry collaborates with researchers at the University of Arkansas for Medical Sciences, including Amanda J. Stolarz and Sung W. Rhee, with whom he has co-authored multiple publications. His scholarship metrics include an h-index of 5, with 13 total publications and 67 total citations.
Metrics
- h-index: 5
- Publications: 13
- Citations: 69
Selected Publications
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Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats (2024)
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Hypertension Induces Contractile Dysfunction in Rat Mesenteric Lymph Vessels (2022)
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Assessment of the Retinal Toxicity and Sealing Strength of Tissue Adhesives (2021)
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Mechanisms of Increased Infarct Volume in a Rat Model of Ischemic Stroke: Implications for Leptomeningeal Collateral Artery Function and Beta Blocker Therapy (2021)
Collaboration Network
Top Collaborators
- Mechanisms of Increased Infarct Volume in a Rat Model of Ischemic Stroke: Implications for Leptomeningeal Collateral Artery Function and Beta Blocker Therapy
- Hypertension Induces Contractile Dysfunction in Rat Mesenteric Lymph Vessels
- Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats
- Mechanisms of Increased Infarct Volume in a Rat Model of Ischemic Stroke: Implications for Leptomeningeal Collateral Artery Function and Beta Blocker Therapy
- Hypertension Induces Contractile Dysfunction in Rat Mesenteric Lymph Vessels
- Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats
- Hypertension Induces Contractile Dysfunction in Rat Mesenteric Lymph Vessels
- Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats
- Mechanisms of Increased Infarct Volume in a Rat Model of Ischemic Stroke: Implications for Leptomeningeal Collateral Artery Function and Beta Blocker Therapy
- Interleukin-1 receptor antagonist inhibits arthrofibrosis in a post-traumatic knee immobilization model
- Interleukin-1 receptor antagonist inhibits arthrofibrosis in a post-traumatic knee immobilization model
- Interleukin-1 receptor antagonist inhibits arthrofibrosis in a post-traumatic knee immobilization model
- Interleukin-1 receptor antagonist inhibits arthrofibrosis in a post-traumatic knee immobilization model
- Interleukin-1 receptor antagonist inhibits arthrofibrosis in a post-traumatic knee immobilization model
- Assessment of the Retinal Toxicity and Sealing Strength of Tissue Adhesives
- Assessment of the Retinal Toxicity and Sealing Strength of Tissue Adhesives
- Assessment of the Retinal Toxicity and Sealing Strength of Tissue Adhesives
- Assessment of the Retinal Toxicity and Sealing Strength of Tissue Adhesives
- A case-control study of phosphodiesterase-5 inhibitor use and Alzheimer’s disease and related dementias among male and female patients aged 65 years and older supporting the need for a phase III clinical trial
- Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats
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