Biography and Research Information
OverviewAI-generated summary
Melissa M. Clemens investigates the mechanisms underlying drug-induced liver injury, with a focus on acetaminophen toxicity. Her research has explored the role of exogenous phosphatidic acid in mitigating liver damage in mice by activating hepatic interleukin-6 signaling. This work also examines inter-organ crosstalk as a factor in liver regeneration and injury response. Clemens has published 12 papers, with a total of 332 citations and an h-index of 8. She has collaborated with researchers at the University of Arkansas for Medical Sciences and the University of Arkansas at Fayetteville on shared publications. Her work has been identified by Medical Subject Headings including Animals, Liver, Chemical and Drug Induced Liver Injury, Liver Regeneration, Acetaminophen, and Mice.
Metrics
- h-index: 8
- Publications: 12
- Citations: 334
Selected Publications
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Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk (2021)
Collaboration Network
Top Collaborators
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
- Exogenous phosphatidic acid reduces acetaminophen-induced liver injury in mice by activating hepatic interleukin-6 signaling through inter-organ crosstalk
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