Forouh Kalantari
Associate Professor
faculty
Radiation Oncology, College of Medicine
Research Areas
Biography and Research Information
OverviewAI-generated summary
Forouh Kalantari, Associate Professor in the Department of Radiation Oncology at the University of Arkansas for Medical Sciences, investigates mechanisms underlying cancer progression and treatment. Her recent work focuses on the role of cystathionine gamma-lyase in regulating hypoxia-inducible factor 1-alpha expression, a pathway implicated in the progression of clear cell ovarian cancer. Kalantari also explores vulnerabilities in cancer cells, as demonstrated by research identifying KEAP1 perturbation as a weakness in ARID1A-deficient cells. Additionally, her research extends to the evaluation of radiation oncology residency training, examining resident and faculty concordance in target delineation and contouring as a method for performance assessment. She has also contributed to studies on the dosimetric analysis of treatment planning systems for spine SBRT and the evaluation of linear accelerators for radiation therapy. Kalantari has a h-index of 4 with 16 publications and 177 citations, and she frequently collaborates with researchers at her institution, including Faraz Kalantari, Gary D. Lewis, and Fen Xia.
Metrics
- h-index: 8
- Publications: 59
- Citations: 177
Selected Publications
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SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells (2026)
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Interstitial Brachytherapy for Soft Tissue Sarcomas: Outcomes and Patterns of Failure Patterns from a Single Institution Experience (2025)
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Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells (2025)
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Spectral CT Imaging for the Visualization of Hyaluronic Acid Based Rectal Spacers prior to Radiation Therapy (2024)
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A Prospective Study Measuring Resident and Faculty Contour Concordance: A Potential Tool for Quantitative Assessment of Residents’ Performance in Contouring and Target Delineation in Radiation Oncology Residency (2023)
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Analysis of the Cancer Imaging Archive for Patterns of Locoregional Failure in Relation to the Mucous Membranes among Patients with Head and Neck Squamous Cell Carcinoma: Potential Organ at Risk that can be Spared? (2023)
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Cleaning the dose falloff with low modulation in SBRT lung plans (2023)
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A Prospective Study Measuring Resident/Faculty Contour Concordance: A Potential Tool for Quantitative Assessment of Residents’ Performance in Contouring and Target Delineation (2023)
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Design and performance evaluation of a slit‐slat camera for 2D prompt gamma imaging in proton therapy monitoring: A Monte Carlo simulation study (2023)
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BEAM MATCHING EVALUATION OF TWO SIMILAR LINEAR ACCELERATORS (2023)
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Analysis of Resident/Faculty Contour Concordance: A Potential Tool for Quantitative Assessment of Residents’ Performance in Target Volume Delineation (2022)
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Measuring resident/faculty contour concordance as a potential tool for quantitative assessment of residents' performance in target volume delineation: a feasibility study (2022)
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A phantom-based study and clinical implementation of brainlab’s treatment planning system for radiosurgical treatments of arteriovenous malformations (2022)
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A dosimetric comparative analysis of Brainlab elements and Eclipse RapidArc for spine SBRT treatment planning (2022)
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Derivation and validation of a sensitivity formula for knife-edge slit gamma camera: A theoretical and Monte Carlo simulation study (2021)1 citation OpenAlex
Collaboration Network
Top Collaborators
- Genome-Wide CRISPR Screen Identifies KEAP1 Perturbation as a Vulnerability of ARID1A-Deficient Cells
- Abstract 4967: Investigating the therapeutic efficacy of EO3001 in clear cell carcinoma of the ovary
- Abstract 2182: The evolution of high grade serous ovarian cancer under the pressure of non-cytotoxic and cytotoxic treatment
- Abstract 2482: On the verge of metastasis: CTH defines a cell-state-dependent adaptive response to microenvironmental stresses
- Abstract PR002: Proteomic profiling of endometrial carcinomas
Showing 5 of 8 shared publications
- Cystathionine gamma‐lyase‐mediated hypoxia inducible factor 1‐alpha expression drives clear cell ovarian cancer progression
- Abstract 4967: Investigating the therapeutic efficacy of EO3001 in clear cell carcinoma of the ovary
- Abstract 2482: On the verge of metastasis: CTH defines a cell-state-dependent adaptive response to microenvironmental stresses
- Abstract A037: Exploring the therapeutic effectiveness of EO3001 in the treatment of clear cell ovarian cancers with ARID1A mutations
- Abstract 4298: Investigating the potential of EO3001 as a therapeutic agent for clear cell ovarian cancers harboring ARID1A mutations
- Cystathionine gamma‐lyase‐mediated hypoxia inducible factor 1‐alpha expression drives clear cell ovarian cancer progression
- Abstract 2182: The evolution of high grade serous ovarian cancer under the pressure of non-cytotoxic and cytotoxic treatment
- Abstract 2482: On the verge of metastasis: CTH defines a cell-state-dependent adaptive response to microenvironmental stresses
- Abstract A037: Exploring the therapeutic effectiveness of EO3001 in the treatment of clear cell ovarian cancers with ARID1A mutations
- O15: Human endometrial organoid model implicates G6PD-dependant metabolism as a potential targetable pathway in ARID1A mutant gynecological cancers
- Cystathionine gamma‐lyase‐mediated hypoxia inducible factor 1‐alpha expression drives clear cell ovarian cancer progression
- Abstract 2482: On the verge of metastasis: CTH defines a cell-state-dependent adaptive response to microenvironmental stresses
- Abstract PR002: Proteomic profiling of endometrial carcinomas
- O15: Human endometrial organoid model implicates G6PD-dependant metabolism as a potential targetable pathway in ARID1A mutant gynecological cancers
- Proteomic analysis uncovers biological diversity in molecularly defined endometrial carcinomas
- Cystathionine gamma‐lyase‐mediated hypoxia inducible factor 1‐alpha expression drives clear cell ovarian cancer progression
- Abstract 2482: On the verge of metastasis: CTH defines a cell-state-dependent adaptive response to microenvironmental stresses
- Abstract PR002: Proteomic profiling of endometrial carcinomas
- O15: Human endometrial organoid model implicates G6PD-dependant metabolism as a potential targetable pathway in ARID1A mutant gynecological cancers
- Proteomic analysis uncovers biological diversity in molecularly defined endometrial carcinomas
- Genome-Wide CRISPR Screen Identifies KEAP1 Perturbation as a Vulnerability of ARID1A-Deficient Cells
- Cystathionine gamma‐lyase‐mediated hypoxia inducible factor 1‐alpha expression drives clear cell ovarian cancer progression
- Genome-wide CRISPR screen identifies KEAP1 perturbation as a vulnerability of ARID1A-deficient cells
- Abstract 2482: On the verge of metastasis: CTH defines a cell-state-dependent adaptive response to microenvironmental stresses
- Abstract 4967: Investigating the therapeutic efficacy of EO3001 in clear cell carcinoma of the ovary
- Abstract A037: Exploring the therapeutic effectiveness of EO3001 in the treatment of clear cell ovarian cancers with ARID1A mutations
- Abstract 4298: Investigating the potential of EO3001 as a therapeutic agent for clear cell ovarian cancers harboring ARID1A mutations
- Abstract 4967: Investigating the therapeutic efficacy of EO3001 in clear cell carcinoma of the ovary
- Abstract A037: Exploring the therapeutic effectiveness of EO3001 in the treatment of clear cell ovarian cancers with ARID1A mutations
- Abstract 4298: Investigating the potential of EO3001 as a therapeutic agent for clear cell ovarian cancers harboring ARID1A mutations
- Abstract 2182: The evolution of high grade serous ovarian cancer under the pressure of non-cytotoxic and cytotoxic treatment
- Abstract A037: Exploring the therapeutic effectiveness of EO3001 in the treatment of clear cell ovarian cancers with ARID1A mutations
- Abstract 4298: Investigating the potential of EO3001 as a therapeutic agent for clear cell ovarian cancers harboring ARID1A mutations
- Cystathionine gamma‐lyase‐mediated hypoxia inducible factor 1‐alpha expression drives clear cell ovarian cancer progression
- Abstract 2482: On the verge of metastasis: CTH defines a cell-state-dependent adaptive response to microenvironmental stresses
- O15: Human endometrial organoid model implicates G6PD-dependant metabolism as a potential targetable pathway in ARID1A mutant gynecological cancers
- Abstract PR002: Proteomic profiling of endometrial carcinomas
- O15: Human endometrial organoid model implicates G6PD-dependant metabolism as a potential targetable pathway in ARID1A mutant gynecological cancers
- Proteomic analysis uncovers biological diversity in molecularly defined endometrial carcinomas
- Cystathionine gamma‐lyase‐mediated hypoxia inducible factor 1‐alpha expression drives clear cell ovarian cancer progression
- Abstract PR002: Proteomic profiling of endometrial carcinomas
- Proteomic analysis uncovers biological diversity in molecularly defined endometrial carcinomas
- Cystathionine gamma‐lyase‐mediated hypoxia inducible factor 1‐alpha expression drives clear cell ovarian cancer progression
- Abstract PR002: Proteomic profiling of endometrial carcinomas
- Proteomic analysis uncovers biological diversity in molecularly defined endometrial carcinomas
- Genome-Wide CRISPR Screen Identifies KEAP1 Perturbation as a Vulnerability of ARID1A-Deficient Cells
- Cystathionine gamma‐lyase‐mediated hypoxia inducible factor 1‐alpha expression drives clear cell ovarian cancer progression
- O15: Human endometrial organoid model implicates G6PD-dependant metabolism as a potential targetable pathway in ARID1A mutant gynecological cancers
- Abstract 4967: Investigating the therapeutic efficacy of EO3001 in clear cell carcinoma of the ovary
- Abstract 4298: Investigating the potential of EO3001 as a therapeutic agent for clear cell ovarian cancers harboring ARID1A mutations
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