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Likely match
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-06
J
James B. Raymick
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Also affiliated: United States Food and Drug Administration (2015–2026)
18 h-index
38 pubs
1,119 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
James Raymick's research centers on neurodegenerative diseases, particularly Alzheimer's disease, and the role of metals and diet in its progression. His work investigates the localization of amyloid plaques and tangles in brain tissue, utilizing techniques such as Congo Red labeling and modified K114 methods. Raymick has explored the potential link between high-fat diets and the expression of Alzheimer's-related genes in the enteric mucosa of rat models. He has also evaluated the affinity of styrylbenzene analogs for binding parenchymal plaques and tangles in human Alzheimer's patients. Furthermore, his research includes the application of proteomics to identify circulating biomarkers of neurotoxicity, offering fluidic endpoints for central nervous system toxicity in rodent models. Raymick has published 37 papers, accumulating 1,074 citations and an h-index of 18. He has collaborated with several researchers at the National Center for Toxicological Research, including Sumit Sarkar on six shared publications.
Metrics
- h-index: 18
- Publications: 38
- Citations: 1,119
Selected Publications
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A new method for visualizing cerebrovascular vessels (2026)
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Potential link of high fat diet and mRNA expression of Alzheimer's disease-related genes in the enteric mucosa of a rat model of Alzheimer's disease (2025)
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Evaluation and Characterization of Modified K114 Method to Localize Plaques in Rodent and Plaques and Tangles in Human Brain Tissue (2024)
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Evaluation of Styrylbenzene analog- FSB and its affinity to bind parenchymal plaques and tangles in patients of Alzheimer’s disease (2022)
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Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity (2021)
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Role of metals in Alzheimer’s disease (2021)
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In vivo demonstration of Congo Red labeled amyloid plaques via perfusion in the Alzheimer disease rat model (2021)
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Tauroursodeoxycholic acid (TUDCA) is neuroprotective in a chronic mouse model of Parkinson’s disease (2020)
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Modification of methods to use Congo-red stain to simultaneously visualize amyloid plaques and tangles in human and rodent brain tissue sections (2020)
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Impaired Amyloid Beta Clearance and Brain Microvascular Dysfunction are Present in the Tg-SwDI Mouse Model of Alzheimer’s Disease (2020)
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P4‐064: EVALUATION OF TAU NEUROPATHOLOGY AND OTHER KEY PROTEINS IN THE OLFACTORY BULB AND ITS CORRELATION WITH HIPPOCAMPUS IN HUMAN ALZHEIMER'S DISEASE (2019)
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Downregulation of 14-3-3 Proteins in Alzheimer’s Disease (2019)
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High Contrast and Resolution Labeling of Amyloid Plaques in Tissue Sections from APP-PS1 Mice and Humans with Alzheimer’s Disease with the Zinc Chelator HQ-O: Practical and Theoretical Considerations (2019)
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A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies (2019)
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Characterization of Serum Exosomes from a Transgenic Mouse Model of Alzheimer’s Disease (2019)
Collaboration Network
Top Collaborators
Sumit Sarkar
National Center for Toxicological Research
24 shared publications
In database
- Neuroprotective and Therapeutic Strategies against Parkinson’s Disease: Recent Perspectives
- Role of metals in Alzheimer’s disease
- Downregulation of 14-3-3 Proteins in Alzheimer’s Disease
- Tauroursodeoxycholic acid (TUDCA) is neuroprotective in a chronic mouse model of Parkinson’s disease
- Combination therapy with lenalidomide and nanoceria ameliorates CNS autoimmunity
Showing 5 of 24 shared publications
Joseph P. Hanig
Center for Drug Evaluation and Research (US)
11 shared publications
- Tauroursodeoxycholic acid (TUDCA) is neuroprotective in a chronic mouse model of Parkinson’s disease
- Neuroprotective effects of acetyl-l-carnitine (ALC) in a chronic MPTP-induced Parkinson’s disease mouse model: Endothelial and microglial effects
- Modification of methods to use Congo-red stain to simultaneously visualize amyloid plaques and tangles in human and rodent brain tissue sections
- Changes in the metabolome and microRNA levels in biological fluids might represent biomarkers of neurotoxicity: A trimethyltin study
- Identification of altered microRNAs in serum of a mouse model of Parkinson’s disease
Showing 5 of 11 shared publications
Elvis Cuevas
National Center for Toxicological Research (US)
9 shared publications
- Downregulation of 14-3-3 Proteins in Alzheimer’s Disease
- Tauroursodeoxycholic acid (TUDCA) is neuroprotective in a chronic mouse model of Parkinson’s disease
- Modification of methods to use Congo-red stain to simultaneously visualize amyloid plaques and tangles in human and rodent brain tissue sections
- Impaired Amyloid Beta Clearance and Brain Microvascular Dysfunction are Present in the Tg-SwDI Mouse Model of Alzheimer’s Disease
- Changes in the metabolome and microRNA levels in biological fluids might represent biomarkers of neurotoxicity: A trimethyltin study
Showing 5 of 9 shared publications
Bonnie Robinson
National Center for Toxicological Research
8 shared publications
In database
- Tauroursodeoxycholic acid (TUDCA) is neuroprotective in a chronic mouse model of Parkinson’s disease
- Neuroprotective effects of acetyl-l-carnitine (ALC) in a chronic MPTP-induced Parkinson’s disease mouse model: Endothelial and microglial effects
- Impaired Amyloid Beta Clearance and Brain Microvascular Dysfunction are Present in the Tg-SwDI Mouse Model of Alzheimer’s Disease
- Changes in the metabolome and microRNA levels in biological fluids might represent biomarkers of neurotoxicity: A trimethyltin study
- Identification of altered microRNAs in serum of a mouse model of Parkinson’s disease
Showing 5 of 8 shared publications
Héctor Rosas-Hernández
National Center for Toxicological Research
6 shared publications
In database
- Modification of methods to use Congo-red stain to simultaneously visualize amyloid plaques and tangles in human and rodent brain tissue sections
- Impaired Amyloid Beta Clearance and Brain Microvascular Dysfunction are Present in the Tg-SwDI Mouse Model of Alzheimer’s Disease
- Changes in the metabolome and microRNA levels in biological fluids might represent biomarkers of neurotoxicity: A trimethyltin study
- Identification of altered microRNAs in serum of a mouse model of Parkinson’s disease
- Characterization of Serum Exosomes from a Transgenic Mouse Model of Alzheimer’s Disease
Showing 5 of 6 shared publications
Qiang Gu
National Center for Toxicological Research
5 shared publications
In database
- Downregulation of 14-3-3 Proteins in Alzheimer’s Disease
- Downregulation of 14-3-3 Proteins in a Kainic Acid-Induced Neurotoxicity Model
- ERK/MAP Kinase Activation is Evident in Activated Microglia of the Striatum and Substantia Nigra in an Acute and Chronically-Induced Mouse Model of Parkinson’s Disease
- Decreased Mcl-1 protein level in the striatum of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-treated mice
- A new method for visualizing cerebrovascular vessels
Srinivasulu Chigurupati
United States Food and Drug Administration (US)
4 shared publications
- Tauroursodeoxycholic acid (TUDCA) is neuroprotective in a chronic mouse model of Parkinson’s disease
- Combination therapy with lenalidomide and nanoceria ameliorates CNS autoimmunity
- Identification of altered microRNAs in serum of a mouse model of Parkinson’s disease
- Corrigendum to “Combination therapy with lenalidomide and nanoceria ameliorates CNS autoimmunity”, [Exp. Neurol. 273 (2015), 151–160]
Merle G. Paule
4 shared publications
In database
- Downregulation of 14-3-3 Proteins in a Kainic Acid-Induced Neurotoxicity Model
- Decreased Mcl-1 protein level in the striatum of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-treated mice
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
Jyotshna Kanungo
National Center for Toxicological Research
3 shared publications
In database
- Downregulation of 14-3-3 Proteins in Alzheimer’s Disease
- Downregulation of 14-3-3 Proteins in a Kainic Acid-Induced Neurotoxicity Model
- A new method for visualizing cerebrovascular vessels
Zhen He
National Center for Toxicological Research (US)
3 shared publications
- Changes in the metabolome and microRNA levels in biological fluids might represent biomarkers of neurotoxicity: A trimethyltin study
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
Serguei Liachenko
National Center for Toxicological Research
3 shared publications
In database
- Changes in the metabolome and microRNA levels in biological fluids might represent biomarkers of neurotoxicity: A trimethyltin study
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
Sherry A. Ferguson
3 shared publications
- Tauroursodeoxycholic acid (TUDCA) is neuroprotective in a chronic mouse model of Parkinson’s disease
- Changes in the metabolome and microRNA levels in biological fluids might represent biomarkers of neurotoxicity: A trimethyltin study
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
James P. O’Callaghan
National Institute for Occupational Safety and Health (US)
3 shared publications
- Changes in the metabolome and microRNA levels in biological fluids might represent biomarkers of neurotoxicity: A trimethyltin study
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
Susan M. Burks
National Center for Toxicological Research
3 shared publications
In database
- Tauroursodeoxycholic acid (TUDCA) is neuroprotective in a chronic mouse model of Parkinson’s disease
- Neuroprotective effects of acetyl-l-carnitine (ALC) in a chronic MPTP-induced Parkinson’s disease mouse model: Endothelial and microglial effects
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
Sharay E. Setti
National Center for Toxicological Research (US)
3 shared publications
- In vivo demonstration of Congo Red labeled amyloid plaques via perfusion in the Alzheimer disease rat model
- Evaluation and Characterization of Modified K114 Method to Localize Plaques in Rodent and Plaques and Tangles in Human Brain Tissue
- Evaluation of Styrylbenzene analog- FSB and its affinity to bind parenchymal plaques and tangles in patients of Alzheimer’s disease
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