Match tier
Listed
Presence
Current · Arkansas
Last published
2022
Sources
OpenAlex · ORCID
Refreshed
2026-10-09
J
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Jessica I. Gann's research focuses on understanding the immune response to chlamydial infections, particularly in the context of the female reproductive tract. Her work has investigated the role of interferon-gamma (IFN-γ) in controlling systemic infections caused by *Chlamydia muridarum* in mice, highlighting the importance of innate immune pathways. Recent publications also explore how IFN-γ and antibodies can synergize to enhance protective immunity against chlamydia dissemination and reinfection. Additionally, her research has touched upon the impact of myeloma-derived exosomes and soluble factors on natural killer cell function. Gann has published five articles with a total of 34 citations and an h-index of 3. She collaborates with researchers at the University of Arkansas for Medical Sciences, including Lin‐Xi Li, Miguel A. B. Mercado, Priyangi A. Malaviarachchi, and Wuying Du.
Metrics
- h-index: 3
- Publications: 5
- Citations: 34
Selected Publications
-
IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections (2022)
-
IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections (2022)
-
Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract (2020)
-
Innate IFNγ is essential for systemic <i>Chlamydia</i> control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract (2020)
-
Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function (2016)
Collaboration Network
Top Collaborators
Priyangi A. Malaviarachchi
University of Arkansas for Medical Sciences
5 shared publications
In database
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic <i>Chlamydia</i> control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
- IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections
Miguel A. B. Mercado
University of Arkansas for Medical Sciences
4 shared publications
In database
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic <i>Chlamydia</i> control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
- IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections
Wuying Du
University of Arkansas for Medical Sciences
4 shared publications
In database
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic <i>Chlamydia</i> control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
- IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections
Lin‐Xi Li
University of Arkansas for Medical Sciences
4 shared publications
In database
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic <i>Chlamydia</i> control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
- IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections
Tarun K. Garg
University of Arkansas for Medical Sciences (US)
1 shared publication
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
Kate Stone
University of Arkansas for Medical Sciences (US)
1 shared publication
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
Veronica MacLeod
University of Arkansas for Medical Sciences (US)
1 shared publication
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
Amy D Greenway
University of Arkansas for Medical Sciences
1 shared publication
In database
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
Nisreen Akel
University of Arkansas for Medical Sciences
1 shared publication
In database
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
Ricky D. Edmondson
University of Arkansas for Medical Sciences
1 shared publication
In database
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
Faith E. Davies
University of Arkansas for Medical Sciences (US)
1 shared publication
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
Joshua Epstein
University of Arkansas for Medical Sciences (US)
1 shared publication
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
Gareth J. Morgan
University of Arkansas for Medical Sciences (US)
1 shared publication
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
Frits van Rhee
University of Arkansas for Medical Sciences
1 shared publication
In database
- Myeloma-Derived Exosomes and Soluble Factors Suppress Natural Killer Cell Function
Similar Researchers
Based on overlapping research topics
Kara A. O’Neal
University of Arkansas for Medical Sciences
Host-Pathogen Interactions
Immune Response and Inflammation
Disease Models, Animal
Sheldon L. Zeltner
University of Arkansas for Medical Sciences
Host-Pathogen Interactions
Immune Response and Inflammation
Disease Models, Animal
Alison R. Gouch
University of Arkansas for Medical Sciences
Host-Pathogen Interactions
Immune Response and Inflammation
Disease Models, Animal
Bernabe Mercado
University of Arkansas for Medical Sciences
Host-Pathogen Interactions
Immune Response and Inflammation
Disease Models, Animal
Lin‐Xi Li
University of Arkansas for Medical Sciences
Host-Pathogen Interactions
Immune Response and Inflammation
Disease Models, Animal
Marli S.P. Azevedo
National Center for Toxicological Research
Host-Pathogen Interactions
Immune Response and Inflammation
Disease Models, Animal