Wuying Du
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Also affiliated: Sun Yat-sen University (2018); Sun Yat-sen University Cancer Center (2014–2018); State Key Laboratory of Oncology in South China (2014–2018)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Wuying Du's research investigates molecular mechanisms underlying disease progression and potential therapeutic interventions. Recent publications include work on how SIRT2 influences murine melanoma progression by inhibiting natural killer cells and how nicotinamide riboside alleviates cisplatin-induced peripheral neuropathy through SIRT2 activation. Du has also contributed to studies on the synergistic effects of IFNγ and antibodies in enhancing immune responses against *Chlamydia* dissemination and reinfections in the female reproductive tract. Collaborations include shared publications with researchers at the University of Arkansas for Medical Sciences such as Lin‐Xi Li, Miguel A. B. Mercado, Priyangi A. Malaviarachchi, and Mousumi Patra. Du's scholarly output is reflected in an h-index of 7, with 14 total publications and over 310 citations.
Metrics
- h-index: 7
- Publications: 14
- Citations: 315
Selected Publications
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IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections (2022)
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IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections (2022)
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Nicotinamide riboside alleviates cisplatin-induced peripheral neuropathy via SIRT2 activation (2022)
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SIRT2 promotes murine melanoma progression through natural killer cell inhibition (2021)
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SIRT2 Promotes Murine Melanoma Progression Through Natural Killer Cell Inhibition (2020)
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Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract (2020)
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Depletion of senescent-like neuronal cells alleviates cisplatin-induced peripheral neuropathy in mice (2020)
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Innate IFNγ is essential for systemic Chlamydia control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract (2020)
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Sirt2-associated transcriptome modifications in cisplatin-induced neuronal injury (2020)
Collaboration Network
Top Collaborators
- Depletion of senescent-like neuronal cells alleviates cisplatin-induced peripheral neuropathy in mice
- SIRT2 promotes murine melanoma progression through natural killer cell inhibition
- Sirt2-associated transcriptome modifications in cisplatin-induced neuronal injury
- Nicotinamide riboside alleviates cisplatin-induced peripheral neuropathy via SIRT2 activation
- SIRT2 Promotes Murine Melanoma Progression Through Natural Killer Cell Inhibition
- Depletion of senescent-like neuronal cells alleviates cisplatin-induced peripheral neuropathy in mice
- SIRT2 promotes murine melanoma progression through natural killer cell inhibition
- Sirt2-associated transcriptome modifications in cisplatin-induced neuronal injury
- SIRT2 Promotes Murine Melanoma Progression Through Natural Killer Cell Inhibition
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic Chlamydia control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
- IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic Chlamydia control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
- IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic Chlamydia control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
- IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections
- Depletion of senescent-like neuronal cells alleviates cisplatin-induced peripheral neuropathy in mice
- SIRT2 promotes murine melanoma progression through natural killer cell inhibition
- Nicotinamide riboside alleviates cisplatin-induced peripheral neuropathy via SIRT2 activation
- SIRT2 Promotes Murine Melanoma Progression Through Natural Killer Cell Inhibition
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic Chlamydia control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
- SIRT2 promotes murine melanoma progression through natural killer cell inhibition
- Nicotinamide riboside alleviates cisplatin-induced peripheral neuropathy via SIRT2 activation
- SIRT2 Promotes Murine Melanoma Progression Through Natural Killer Cell Inhibition
- Depletion of senescent-like neuronal cells alleviates cisplatin-induced peripheral neuropathy in mice
- Sirt2-associated transcriptome modifications in cisplatin-induced neuronal injury
- SIRT2 promotes murine melanoma progression through natural killer cell inhibition
- SIRT2 Promotes Murine Melanoma Progression Through Natural Killer Cell Inhibition
- SIRT2 promotes murine melanoma progression through natural killer cell inhibition
- SIRT2 Promotes Murine Melanoma Progression Through Natural Killer Cell Inhibition
- Sirt2-associated transcriptome modifications in cisplatin-induced neuronal injury
- Depletion of senescent-like neuronal cells alleviates cisplatin-induced peripheral neuropathy in mice
- Depletion of senescent-like neuronal cells alleviates cisplatin-induced peripheral neuropathy in mice
- Depletion of senescent-like neuronal cells alleviates cisplatin-induced peripheral neuropathy in mice
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