Biography and Research Information
OverviewAI-generated summary
Landon B. Gatrell's research investigates cellular mechanisms related to aging, bone development, and tissue repair. His work has explored the role of the NAD salvage pathway in skeletal development in mice and the impact of mitochondrial oxidative stress and autophagy on bone mechanoresponsiveness in aging. Gatrell has also studied the effects of chemotherapy on bone mass and the contribution of cellular senescence to fibrotic repair in preclinical models. His publications include research on single-cell RNA sequencing of mesenchymal populations to identify pathways altered in age-associated osteoarthritis and the development of a transgenic rat model for assessing chondrogenesis <i>in vivo</i>. Gatrell collaborates with researchers at the University of Arkansas for Medical Sciences, including Aaron Warren and Maria Almeida.
Metrics
- h-index: 2
- Publications: 4
- Citations: 16
Selected Publications
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The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone (2025)
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The adverse effects of chemotherapy on bone mass are not prevented by senolytics (2025)
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Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation (2025)
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Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness (2025)
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Single cell RNA sequencing of mesenchymal populations from murine knees reveals distinct pathways altered in age-associated versus post-traumatic osteoarthritis (2024)
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CHRONIC SENESCENCE CONTRIBUTES TO FIBROCARTILAGE REPAIR IN A PRECLINICAL MODEL OF BONE MARROW STIMULATION (2024)
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The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice (2023)
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A Transgenic Rat for Noninvasive Assessment of Chondrogenesis <i>in Vivo</i> (2021)
Collaboration Network
Top Collaborators
Ryan M. Porter
University of Arkansas Medical Center (US)
3 shared publications
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
- A Transgenic Rat for Noninvasive Assessment of Chondrogenesis <i>in Vivo</i>
- Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation
Elisabeth Ferreira
University of Arkansas Medical Center
2 shared publications
In database
- A Transgenic Rat for Noninvasive Assessment of Chondrogenesis <i>in Vivo</i>
- Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation
Luke Childress
University of Arkansas Medical Center (US)
2 shared publications
- A Transgenic Rat for Noninvasive Assessment of Chondrogenesis <i>in Vivo</i>
- Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation
Hong Wu
University of Arkansas Medical Center (US)
2 shared publications
- A Transgenic Rat for Noninvasive Assessment of Chondrogenesis <i>in Vivo</i>
- Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation
Aaron Warren
University of Arkansas for Medical Sciences
2 shared publications
In database
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
- The adverse effects of chemotherapy on bone mass are not prevented by senolytics
Md Mohsin Ali
University of Arkansas for Medical Sciences (US)
2 shared publications
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
- The adverse effects of chemotherapy on bone mass are not prevented by senolytics
Intawat Nookaew
University of Arkansas Medical Center
2 shared publications
In database
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
- Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation
Maria Almeida
2 shared publications
In database
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
- The adverse effects of chemotherapy on bone mass are not prevented by senolytics
Olivia Reyes‐Castro
University of Arkansas for Medical Sciences
1 shared publication
In database
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
Adriana Lelis Carvalho
University of Coimbra
1 shared publication
In database
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
Ha‐Neui Kim
University of Arkansas for Medical Sciences
1 shared publication
In database
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
Ernestina Schipani
University of Pennsylvania (US)
1 shared publication
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
Charles A. O’Brien
University of Arkansas for Medical Sciences
1 shared publication
In database
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
Roy Morello
University of Arkansas for Medical Sciences
1 shared publication
In database
- The NAD salvage pathway in mesenchymal cells is indispensable for skeletal development in mice
Pilar Simmons
University of Arkansas for Medical Sciences
1 shared publication
In database
- The adverse effects of chemotherapy on bone mass are not prevented by senolytics
Similar Researchers
Based on overlapping research topics
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