Philip R. Mayeux
Researcher
Also affiliated: Tulane University (1987–1989); Arkansas Children's Hospital (2016); Medical University of South Carolina (1988–1991); Baylor College of Medicine (2016); University of Arkansas Medical Center (1997–2014); Tulane Medical Center (1985–1988); Dutch Health Care Inspectorate (2016); Xilinx (United States) (2016); MicroVision (United States) (2016); Fresenius (Germany) (2016); Nierstichting (2016); AM–Pharma (Netherlands) (2016); Bio-Medical Science (South Korea) (2016); Texas Children's Hospital (2016); Institute of Pharmacology (2009–2010); Baxter (United States) (2016)
Faculty Researcher
Research Areas
Biomedical Subjects
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Biography and Research Information
OverviewAI-generated summary
Philip R. Mayeux's research interests include behavioral and psychological studies, with a focus on health sciences research and education. His work has explored various aspects of health disparities and outcomes, as well as behavioral health and interventions. Mayeux has also been involved in studies related to child and adolescent health, investigating factors that influence well-being and development within this population.
Metrics
- h-index: 38
- Publications: 95
- Citations: 6,199
Selected Publications
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Microvascular Dysfunction in the Critically Ill (2020)
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Retraction Note: Sepsis-induced elevation in plasma serotonin facilitates endothelial hyperpermeability (2019)
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Female mice exhibit less renal mitochondrial injury but greater mortality using a comorbid model of experimental sepsis. (2018)
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Rolipram Improves Outcome in a Rat Model of Infant Sepsis-Induced Cardiorenal Syndrome (2017)
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Development of Renal Dysfunction in an Infant Model of Polymicrobial Sepsis (2016)
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Preclinical Evaluation of the Phosphodiesterase‐4 Inhibitor, Rolipram, in an Infant Model of Sepsis‐Induced Cardiorenal Syndrome (2016)
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Delayed Mitogen-Activated Protein Kinase/Extracellular Signal–Regulated Kinase Inhibition by Trametinib Attenuates Systemic Inflammatory Responses and Multiple Organ Injury in Murine Sepsis* (2016)
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Could Biomarkers Direct Therapy for the Septic Patient? (2016)
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RETRACTED ARTICLE: Sepsis-induced elevation in plasma serotonin facilitates endothelial hyperpermeability (2016)
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Inactivation of renal mitochondrial respiratory complexes and manganese superoxide dismutase during sepsis: mitochondria-targeted antioxidant mitigates injury (2014)
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Targeting mitochondrial oxidants may facilitate recovery of renal function during infant sepsis (2014)
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Pharmacologic Targeting of Sphingosine-1-Phosphate Receptor 1 Improves the Renal Microcirculation during Sepsis in the Mouse (2014)
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Rolipram Improves Renal Perfusion and Function during Sepsis in the Mouse (2013)
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Preclinical evaluation of the mitochondria‐targeted antioxidant mitoquinone to treat sepsis‐induced acute kidney injury (2013)
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Sirtuin 1 enzyme activity and autophagy proteins are increased in the kidney during murine sepsis (2012)
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