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Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
S
Shannon Rose
High Impact
Associate Professor
Also affiliated: Rutgers, The State University of New Jersey (1990–1991); Arkansas Children's Hospital (2009–2025); University of Minnesota (1990–1991); Scripps Health (1990–1991); Harvard University (1990–1994); University of Tennessee Health Science Center (2025); The University of Melbourne (1956); University of California, San Francisco (1990–1991); University of Arkansas Medical Center (2023); Council of Science Editors (1991); University of California, San Diego (1990–1991); Shannon Applied Biotechnology Centre (2024); Hospices Civils de Lyon (2008); Hôpital Edouard Herriot (2008); Arkansas Children's Nutrition Center (2018–2022); Scripps Clinic (1990–1991); Arkansas Department of Agriculture (2019); National Health and Medical Research Council (1956); Weizmann Institute of Science (1990–1994); University of California, Berkeley (1990–1991)
34 h-index
91 pubs
5,143 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Shannon Rose's research focuses on understanding the biological mechanisms underlying Autism Spectrum Disorder (ASD), with a particular emphasis on mitochondrial dysfunction, oxidative stress, and metabolic imbalances. Rose has investigated these areas through studies involving human subjects, including children, and utilizing cell lines derived from individuals with ASD.
Publications detail findings related to oxidative damage and inflammation in the autism brain, as well as metabolic imbalances associated with methylation dysregulation and oxidative damage in children with ASD. Further work has explored the clinical and molecular characteristics of mitochondrial dysfunction in ASD and the potential of butyrate to enhance mitochondrial function during oxidative stress in cell lines from boys with autism. Rose's research also includes studies on cellular and mitochondrial glutathione redox imbalance in lymphoblastoid cells derived from children with autism, and the impact of oxidative stress on mitochondrial function in a subset of autism lymphoblastoid cell lines. Additionally, Rose has investigated the therapeutic potential of folinic acid in improving verbal communication in children with autism.
Rose's scholarly contributions are reflected in an h-index of 34 and over 5,000 citations across 91 publications. Rose collaborates with several researchers at the University of Arkansas for Medical Sciences, including Leanna Delhey, Elisabet Børsheim, Craig Porter, and Pritmohinder S. Gill.
Metrics
- h-index: 34
- Publications: 91
- Citations: 5,143
Positions
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Associate Professor 2024–presentUniversity of Tennessee Health Science Center College of Nursing, Department of Health Promotion and Disease Prevention ORCID
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Associate Professor publications 2008–2025University of Arkansas for Medical Sciences ORCID
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Assistant Professor 2017–2024University of Arkansas for Medical Sciences Pediatrics ORCID
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Postdoctoral Fellow 2012–2017University of Arkansas for Medical Sciences Pediatrics ORCID
Selected Publications
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Mitochondrial respiratory function in human platelets: Influence of sample preparation, assay buffer, and instrumental platform (2025)
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Correlating maternal and cord-blood inflammatory markers and BDNF with human fetal brain activity recorded by magnetoencephalography: An exploratory study (2024)
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Platelets, inflammation, and purinergic receptors in chronic kidney disease (2024)
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Regulatory T cells and bioenergetics of peripheral blood mononuclear cells linked to pediatric obesity (2024)
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Correlation of fetal heart rate dynamics to inflammatory markers and brain-derived neurotrophic factor during pregnancy (2024)
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Nitrosative Stress in Autism: Supportive Evidence and Implications for Mitochondrial Dysfunction (2024)
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Circulating microRNA levels differ in the early stages of insulin resistance in prepubertal children with obesity (2022)
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Air Pollution and Maximum Temperature Are Associated with Neurodevelopmental Regressive Events in Autism Spectrum Disorder (2022)
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Metabolomic Signatures of Autism Spectrum Disorder (2022)
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Blood RNA Sequencing Indicates Upregulated BATF2 and LY6E and Downregulated ISG15 and MT2A Expression in Children with Autism Spectrum Disorder (2022)
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Integrated microRNA–mRNA Expression Profiling Identifies Novel Targets and Networks Associated with Autism (2022)
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Effect of excess weight and insulin resistance on DNA methylation in prepubertal children (2022)
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A Personalized Approach to Evaluating and Treating Autism Spectrum Disorder (2022)
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Blood RNA Sequencing Confirms Upregulated BATF2 and FCGR1A Expression in Children with Autism Spectrum Disorder (2021)
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MicroRNA Expression Profiles in Autism Spectrum Disorder: Role for miR-181 in Immunomodulation (2021)
Collaboration Network
Top Collaborators
Richard E. Frye
Southwest Autism Research & Resource Center (US)
42 shared publications
- Evidence of oxidative damage and inflammation associated with low glutathione redox status in the autism brain
- Clinical and Molecular Characteristics of Mitochondrial Dysfunction in Autism Spectrum Disorder
- Butyrate enhances mitochondrial function during oxidative stress in cell lines from boys with autism
- Folinic acid improves verbal communication in children with autism and language impairment: a randomized double-blind placebo-controlled trial
- Gastrointestinal dysfunction in autism spectrum disorder: the role of the mitochondria and the enteric microbiome
Showing 5 of 42 shared publications
Sirish C. Bennuri
Arkansas Children's Hospital (US)
27 shared publications
- Butyrate enhances mitochondrial function during oxidative stress in cell lines from boys with autism
- Modulation of mitochondrial function by the microbiome metabolite propionic acid in autism and control cell lines
- Mitochondrial and redox abnormalities in autism lymphoblastoid cells: a sibling control study
- Prenatal air pollution influences neurodevelopment and behavior in autism spectrum disorder by modulating mitochondrial physiology
- Blocking and Binding Folate Receptor Alpha Autoantibodies Identify Novel Autism Spectrum Disorder Subgroups
Showing 5 of 27 shared publications
Leanna Delhey
University of Michigan
21 shared publications
In database
- Butyrate enhances mitochondrial function during oxidative stress in cell lines from boys with autism
- Folinic acid improves verbal communication in children with autism and language impairment: a randomized double-blind placebo-controlled trial
- Modulation of mitochondrial function by the microbiome metabolite propionic acid in autism and control cell lines
- Prenatal air pollution influences neurodevelopment and behavior in autism spectrum disorder by modulating mitochondrial physiology
- Blocking and Binding Folate Receptor Alpha Autoantibodies Identify Novel Autism Spectrum Disorder Subgroups
Showing 5 of 21 shared publications
Stepan Melnyk
Arkansas Children's Hospital
20 shared publications
In database
- Evidence of oxidative damage and inflammation associated with low glutathione redox status in the autism brain
- Metabolic Imbalance Associated with Methylation Dysregulation and Oxidative Damage in Children with Autism
- Cellular and mitochondrial glutathione redox imbalance in lymphoblastoid cells derived from children with autism
- Folinic acid improves verbal communication in children with autism and language impairment: a randomized double-blind placebo-controlled trial
- Modulation of mitochondrial function by the microbiome metabolite propionic acid in autism and control cell lines
Showing 5 of 20 shared publications
Marie Tippett
Arkansas Children's Hospital
18 shared publications
In database
- Butyrate enhances mitochondrial function during oxidative stress in cell lines from boys with autism
- Folinic acid improves verbal communication in children with autism and language impairment: a randomized double-blind placebo-controlled trial
- Modulation of mitochondrial function by the microbiome metabolite propionic acid in autism and control cell lines
- Oxidative stress induces mitochondrial dysfunction in a subset of autistic lymphoblastoid cell lines
- Prenatal air pollution influences neurodevelopment and behavior in autism spectrum disorder by modulating mitochondrial physiology
Showing 5 of 18 shared publications
S. Jill James
Arkansas Children's Hospital
17 shared publications
In database
- Evidence of oxidative damage and inflammation associated with low glutathione redox status in the autism brain
- Metabolic Imbalance Associated with Methylation Dysregulation and Oxidative Damage in Children with Autism
- Cellular and mitochondrial glutathione redox imbalance in lymphoblastoid cells derived from children with autism
- Folinic acid improves verbal communication in children with autism and language impairment: a randomized double-blind placebo-controlled trial
- Abnormal Transmethylation/transsulfuration Metabolism and DNA Hypomethylation Among Parents of Children with Autism
Showing 5 of 17 shared publications
Rebecca Wynne
Arkansas Children's Hospital
12 shared publications
In database
- Butyrate enhances mitochondrial function during oxidative stress in cell lines from boys with autism
- Folinic acid improves verbal communication in children with autism and language impairment: a randomized double-blind placebo-controlled trial
- Modulation of mitochondrial function by the microbiome metabolite propionic acid in autism and control cell lines
- Mitochondrial and redox abnormalities in autism lymphoblastoid cells: a sibling control study
- Oxidative stress induces mitochondrial dysfunction in a subset of autistic lymphoblastoid cell lines
Showing 5 of 12 shared publications
Stephen G. Kahler
Arkansas Children's Hospital (US)
10 shared publications
- Metabolic Imbalance Associated with Methylation Dysregulation and Oxidative Damage in Children with Autism
- Clinical and Molecular Characteristics of Mitochondrial Dysfunction in Autism Spectrum Disorder
- Butyrate enhances mitochondrial function during oxidative stress in cell lines from boys with autism
- Modulation of mitochondrial function by the microbiome metabolite propionic acid in autism and control cell lines
- Blocking and Binding Folate Receptor Alpha Autoantibodies Identify Novel Autism Spectrum Disorder Subgroups
Showing 5 of 10 shared publications
John M. Slattery
Arkansas Children's Hospital (US)
9 shared publications
- Folinic acid improves verbal communication in children with autism and language impairment: a randomized double-blind placebo-controlled trial
- Gastrointestinal dysfunction in autism spectrum disorder: the role of the mitochondria and the enteric microbiome
- Oxidative stress induces mitochondrial dysfunction in a subset of autistic lymphoblastoid cell lines
- Blocking and Binding Folate Receptor Alpha Autoantibodies Identify Novel Autism Spectrum Disorder Subgroups
- Thyroid dysfunction in children with autism spectrum disorder is associated with folate receptor α autoimmune disorder
Showing 5 of 9 shared publications
John C. Slattery
Arkansas Children's Hospital (US)
6 shared publications
- Butyrate enhances mitochondrial function during oxidative stress in cell lines from boys with autism
- Modulation of mitochondrial function by the microbiome metabolite propionic acid in autism and control cell lines
- Intravenous immunoglobulin for the treatment of autoimmune encephalopathy in children with autism
- Multivariate techniques enable a biochemical classification of children with autism spectrum disorder versus typically‐developing peers: A comparison and validation study
- Comparison of Treatment for Metabolic Disorders Associated with Autism:Reanalysis of Three Clinical Trials
Showing 5 of 6 shared publications
Raymond F. Palmer
The University of Texas at San Antonio Health Science Center (US)
6 shared publications
- Prenatal air pollution influences neurodevelopment and behavior in autism spectrum disorder by modulating mitochondrial physiology
- Early life metal exposure dysregulates cellular bioenergetics in children with regressive autism spectrum disorder
- Mitochondria May Mediate Prenatal Environmental Influences in Autism Spectrum Disorder
- Oxidative Stress Challenge Uncovers Trichloroacetaldehyde Hydrate-Induced Mitoplasticity in Autistic and Control Lymphoblastoid Cell Lines
- Physiological mediators of prenatal environmental influences in autism spectrum disorder
Showing 5 of 6 shared publications
Oleksandra Pavliv
Arkansas Children's Hospital
5 shared publications
In database
- Evidence of oxidative damage and inflammation associated with low glutathione redox status in the autism brain
- Metabolic Imbalance Associated with Methylation Dysregulation and Oxidative Damage in Children with Autism
- Cellular and mitochondrial glutathione redox imbalance in lymphoblastoid cells derived from children with autism
- Intracellular and Extracellular Redox Status and Free Radical Generation in Primary Immune Cells from Children with Autism
- Mitochondrial respiratory function in human platelets: Influence of sample preparation, assay buffer, and instrumental platform
Eugénia Carvalho
Arkansas Children's Hospital (US)
5 shared publications
- A comparative study of mitochondrial respiration in circulating blood cells and skeletal muscle fibers in women
- Redox Imbalance and Methylation Disturbances in Early Childhood Obesity
- Circulating microRNA levels differ in the early stages of insulin resistance in prepubertal children with obesity
- Effect of excess weight and insulin resistance on DNA methylation in prepubertal children
- Mitochondrial Respiration in Female Zucker Rats: Effects of Obesity and Short-Term Metformin Treatment
Elisabet Børsheim
Arkansas Children's Hospital
5 shared publications
In database
- A comparative study of mitochondrial respiration in circulating blood cells and skeletal muscle fibers in women
- Redox Imbalance and Methylation Disturbances in Early Childhood Obesity
- Circulating microRNA levels differ in the early stages of insulin resistance in prepubertal children with obesity
- Effect of excess weight and insulin resistance on DNA methylation in prepubertal children
- Mitochondrial respiratory function in human platelets: Influence of sample preparation, assay buffer, and instrumental platform
Janet Cakir
North Carolina State University (US)
5 shared publications
- Prenatal air pollution influences neurodevelopment and behavior in autism spectrum disorder by modulating mitochondrial physiology
- Early life metal exposure dysregulates cellular bioenergetics in children with regressive autism spectrum disorder
- Mitochondria May Mediate Prenatal Environmental Influences in Autism Spectrum Disorder
- Physiological mediators of prenatal environmental influences in autism spectrum disorder
- Air Pollution and Maximum Temperature Are Associated with Neurodevelopmental Regressive Events in Autism Spectrum Disorder
Similar Researchers
Based on overlapping research topics
S. Jill James
University of Arkansas for Medical Sciences
Autism Spectrum Disorder Research
Oxidative Stress
Metabolism and Genetic Disorders
Oleksandra Pavliv
University of Arkansas for Medical Sciences
Autism Spectrum Disorder Research
Mitochondrial Function and Pathology
Oxidative Stress
Marie Tippett
University of Arkansas for Medical Sciences
Autism Spectrum Disorder Research
Mitochondrial Function and Pathology
Child and Adolescent Health
Stepan Melnyk
University of Arkansas for Medical Sciences
Autism Spectrum Disorder Research
Oxidative Stress
Metabolism and Genetic Disorders
Nükhet Aykin‐Burns
University of Arkansas for Medical Sciences
Mitochondrial Function and Pathology
Oxidative Stress
Metabolism and Genetic Disorders
Lee Ann MacMillan-Crow
University of Arkansas for Medical Sciences
Mitochondrial Function and Pathology
Oxidative Stress
Disease Mechanisms Research