Sydney Hilgenbrink
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Researcher
Graduate Student Researcher
Research Areas
Biography and Research Information
OverviewAI-generated summary
Sydney Hilgenbrink's research investigates the mechanisms underlying muscle dysfunction associated with pancreatic cancer-induced cachexia. Her work has explored alterations in gait mechanics during the development of this condition, as indicated by her pilot study. Hilgenbrink also studies strategies to target mitochondrial dynamics, specifically examining the protective effects of OPA1 overexpression and BNIP3 knockdown against muscle dysfunction induced by pancreatic cancer. Her current research activity is noted as recent.
Metrics
- Publications: 2
Selected Publications
-
Alterations in gait mechanics during the development of pancreatic cancer-induced cachexia: a pilot study (2026)
-
Targeting mitochondrial dynamics via OPA1 overexpression and BNIP3 knockdown confers protection against pancreatic cancer induced muscle dysfunction (2026)
Collaboration Network
Top Collaborators
- Targeting mitochondrial dynamics via OPA1 overexpression and BNIP3 knockdown confers protection against pancreatic cancer induced muscle dysfunction
- Alterations in gait mechanics during the development of pancreatic cancer-induced cachexia: a pilot study
- Targeting mitochondrial dynamics via OPA1 overexpression and BNIP3 knockdown confers protection against pancreatic cancer induced muscle dysfunction
- Alterations in gait mechanics during the development of pancreatic cancer-induced cachexia: a pilot study
- Targeting mitochondrial dynamics via OPA1 overexpression and BNIP3 knockdown confers protection against pancreatic cancer induced muscle dysfunction
- Alterations in gait mechanics during the development of pancreatic cancer-induced cachexia: a pilot study
- Targeting mitochondrial dynamics via OPA1 overexpression and BNIP3 knockdown confers protection against pancreatic cancer induced muscle dysfunction
- Targeting mitochondrial dynamics via OPA1 overexpression and BNIP3 knockdown confers protection against pancreatic cancer induced muscle dysfunction
- Targeting mitochondrial dynamics via OPA1 overexpression and BNIP3 knockdown confers protection against pancreatic cancer induced muscle dysfunction
- Alterations in gait mechanics during the development of pancreatic cancer-induced cachexia: a pilot study
Similar Researchers
Based on overlapping research topics