Zain B. Malik
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Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Zain B. Malik's research investigates the impact of exercise on cancer progression and associated molecular changes. His work, primarily conducted in mouse models, examines how exercise preconditioning influences skeletal muscle plasticity and helps preserve muscle mass during cancer development. Malik's studies also focus on suppressing the molecular mediators of cachexia, a condition characterized by significant weight loss and muscle wasting in cancer patients. His recent publications include detailed raw data sets related to muscle physiology, body weight, and metabolic markers in mice undergoing exercise interventions. Malik collaborates with Micah Goeke at the University of Arkansas at Fayetteville, with whom he has co-authored 11 publications.
Metrics
- h-index: 1
- Publications: 13
- Citations: 1
Selected Publications
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Exercise preconditioning confers skeletal muscle myometaplasticity (2026)
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Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia (2026)
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Satellite Cell Content and Muscle Memory in Retrained and Naïve Trained Skeletal Muscle (2026)Journal of the Arkansas Academy of Science OpenAlex
Collaboration Network
Top Collaborators
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise preconditioning confers skeletal muscle myometaplasticity
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise preconditioning confers skeletal muscle myometaplasticity
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise preconditioning confers skeletal muscle myometaplasticity
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise preconditioning confers skeletal muscle myometaplasticity
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise preconditioning confers skeletal muscle myometaplasticity
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise preconditioning confers skeletal muscle myometaplasticity
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
- Exercise preconditioning confers skeletal muscle myometaplasticity
- Exercise preconditioning confers skeletal muscle myometaplasticity
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