Syed F. Ali Data-verified

Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.

High Impact

Researcher

Last publication 2025 Last refreshed 2026-05-16

faculty

75 h-index 591 pubs 20,874 cited

Biography and Research Information

OverviewAI-generated summary

Syed F. Ali's research has focused on understanding the complex mechanisms underlying neurological disorders, particularly those resulting from trauma and substance abuse. His work investigates the impact of traumatic brain injury (TBI) on the central nervous system, examining molecular and cellular changes such as blood-brain barrier breakdown, astrocyte reactivity, and alterations in the cortical transcriptome and proteome. These studies aim to elucidate pathways involving neurogenesis, inflammation, and cell death in the context of TBI.

Further research by Ali explores the role of the endocannabinoid system, specifically the mitochondrial cannabinoid receptor 1 (mtCB1R), in central nervous system function. He has also investigated the behavioral and glial responses to acute substance abuse paradigms, contributing to the understanding of drug-induced changes in emotional behavior. His scholarly output includes work on predicting outcomes for severe subdural hematomas and case reports on cardiovascular complications and adverse drug reactions, reflecting a broad interest in clinical toxicology and emergency medicine.

Ali holds a distinguished record with an h-index of 75 and over 20,000 citations across nearly 600 publications. He has collaborated with researchers at the University of Arkansas at Fayetteville, including Kartik Balachandran, Jeffrey C. Wolchok, Celeste Dunn, and Sara M. Venier.

Metrics

  • h-index: 75
  • Publications: 591
  • Citations: 20,874

Selected Publications

  • Retraction notice to “On the in vivo early toxic properties of Aβ25–35 peptide in the rat hippocampus: Involvement of the Receptor-for-Advanced Glycation-End-Products and changes in gene expression” [Neurotoxicology and Teratology 33 (2011) 288–296] (2025)
  • Retraction notice to “On the early toxic effect of quinolinic acid: Involvement of RAGE” [Neurosci. Lett. 474(2) (2010) 74–78] (2024)
  • The Endocannabinoid System in the Central Nervous System: Emphasis on the Role of the Mitochondrial Cannabinoid Receptor 1 (mtCB1R) (2022)
  • Functional Analysis of the Cortical Transcriptome and Proteome Reveal Neurogenesis, Inflammation, and Cell Death after Repeated Traumatic Brain Injury <i>In vivo</i> (2022)
    4 citations DOI OpenAlex
  • Autophagy and protein aggregation as a mechanism of dopaminergic degeneration in a primary human dopaminergic neuronal model (2022)
    2 citations DOI OpenAlex
  • Blood–Brain Barrier Breakdown and Astrocyte Reactivity Evident in the Absence of Behavioral Changes after Repeated Traumatic Brain Injury (2021)
    21 citations DOI OpenAlex
  • The Endocannabinoid System in the Central Nervous System: Emphasis on the Role of the Mitochondrial Cannabinoid Receptor 1 (mtCB1R) (2021)
    5 citations DOI OpenAlex
  • Acute MDPV Binge Paradigm on Mice Emotional Behavior and Glial Signature (2021)
    4 citations DOI OpenAlex
  • Neurotoxicity of thallium: Old issues and new developments (2021)
    1 citation DOI OpenAlex

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Collaboration Network

216 Collaborators 97 Institutions 17 Countries

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