Zhiqiang Qin
Associate Professor
Also affiliated: Qingdao University (2017–2018); Zhejiang Sci-Tech University (2023); National Institute for Parasitic Diseases (2014); Medical University of South Carolina (2010–2011); Chinese Center For Disease Control and Prevention (2014); MUSC Hollings Cancer Center (2010); Nanjing Maternity and Child Health Care Hospital (2020); Ministry of Education (2012); The People's Hospital of Guangxi Zhuang Autonomous Region (2021–2025); Qingdao Municipal Hospital (2018); Jiangsu Cancer Hospital (2016); Institute of Forensic Science (2021–2023); Jiangsu Province Hospital (2016–2021); Nanjing Jiangning Hospital (2019); Zhejiang University (2013); Henan Medical University (2013); Nanjing Medical University (2012–2023)
Faculty Researcher
Pathology, College of Medicine
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Zhiqiang Qin's research investigates the molecular mechanisms underlying disease progression, with a particular focus on cancer and oxidative stress. He has published work examining the role of microRNAs and signaling pathways, such as the Keap1/Nrf2 pathway, in disease development and response to injury. His studies include investigating the NLRP3 inflammasome's involvement in the malignant progression of prostate cancer and the function of USP15 in testicular oxidative stress injury. Qin also applies computational methods to biological and forensic problems, including the use of deep learning for sex estimation from skeletal remains and the development of algorithms for inferring drowning site locations from diatom populations.
His work contributes to understanding disease mechanisms and exploring potential therapeutic targets. Qin's research group leads investigations into these areas, with scholarship metrics including an h-index of 24, 85 total publications, and 1,830 total citations. He has collaborated with researchers at the University of Arkansas for Medical Sciences, including Steven R. Post, Lu Dai, Jungang Chen, and Jiaojiao Fan.
Metrics
- h-index: 25
- Publications: 84
- Citations: 1,866
Selected Publications
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Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation (2026)
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KSHV and Human Diseases: Beyond KS, PEL and MCD (2026)
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Abstract 2987: Role of EIF4G1 network in non-small cell lung cancers (NSCLC) pathogenesis and development of targeted therapy (2025)
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Infection of human induced pluripotent stem cells by an oncogenic herpesvirus (2025)
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Abstract 4384: Role of EIF4G1 in non-small cell lung cancer pathogenesis and targeted therapy (2019)
Grants & Funding
As listed on this researcher's institutional profile.
- Seeds of Science Pilot award UAMS Internal Research Awards Principal Investigator
- Development of O'PROTACs-based Novel EIF4G1 Degraders for Treatment of Non-small Cell Lung Cancer US Department of Defense Principal Investigator
- Defining mechanisms of KSHV pathogenesis using MHV68-KSHV chimeric viruses NIH Co-Investigator
- Periodontal bacteria enhance oral KSHV pathogenesis and Kaposi's Sarcoma development in HIV + patients - Continuation NIH/Nat. Cancer Institute Principal Investigator
Collaboration Network
Top Collaborators
- Infection of human induced pluripotent stem cells by an oncogenic herpesvirus
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
- Infection of human induced pluripotent stem cells by an oncogenic herpesvirus
- Abstract 2987: Role of EIF4G1 network in non-small cell lung cancers (NSCLC) pathogenesis and development of targeted therapy
- Infection of human induced pluripotent stem cells by an oncogenic herpesvirus
- Infection of human induced pluripotent stem cells by an oncogenic herpesvirus
- Abstract 2987: Role of EIF4G1 network in non-small cell lung cancers (NSCLC) pathogenesis and development of targeted therapy
- Abstract 2987: Role of EIF4G1 network in non-small cell lung cancers (NSCLC) pathogenesis and development of targeted therapy
- KSHV and Human Diseases: Beyond KS, PEL and MCD
- KSHV and Human Diseases: Beyond KS, PEL and MCD
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
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