Miguel A. B. Mercado

Researcher

University of Arkansas for Medical Sciences

faculty

3 h-index 10 pubs 39 cited

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Biography and Research Information

OverviewAI-generated summary

Miguel A. B. Mercado's research focuses on the immune response to <i>Chlamydia</i> infection, particularly within the female reproductive tract. His work investigates the roles of specific immune cells and molecular pathways in mediating protective immunity. Recent publications explore how Interferon-gamma (IFNγ) and antibodies, in combination, enhance immunity against <i>Chlamydia</i> dissemination and reinfection. Additionally, his research examines the function of the transcription factor BHLHE40 in driving CD4 T cell differentiation and promoting a polyfunctional T cell response necessary for combating <i>Chlamydia</i> infection in mouse models. He also studies the influence of TGFb signaling on CD4 T cell responses in this context. Mercado collaborates with several researchers at the University of Arkansas for Medical Sciences, including Lin-Xi Li and Rachel S. Palmer, with whom he has co-authored multiple publications.

Metrics

  • h-index: 3
  • Publications: 10
  • Citations: 39

Selected Publications

  • The roles of TGFb signaling in CD4 T cell responses to Chlamydia infection in the female reproductive tract 9284 (2025) DOI
  • BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia (2024) DOI
  • BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against <i>Chlamydia</i> (2023) DOI
  • Transcription factor Bhlhe40 plays a protective role during intravaginal <i>Chlamydia muridarum</i> infection in mice (2023) DOI
  • IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections (2022) DOI
  • IFNγ and antibody synergize to enhance protective immunity against <i>Chlamydia</i> dissemination and female reproductive tract reinfections (2022) DOI

Collaborators

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