Ashim K. Bagchi
Researcher
Formerly Arkansas Affiliated with UAMS through 2024; recent publications list Cardiovascular Institute Hospital, University of Manitoba.
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Ashim K. Bagchi's research focuses on the molecular mechanisms underlying cardiovascular diseases, particularly those induced by chemotherapy agents like doxorubicin. His work investigates the roles of oxidative stress, endoplasmic reticulum (ER) stress, and apoptotic pathways in the development of doxorubicin-induced cardiomyopathy. Bagchi has published studies examining how factors such as Interleukin-10 and empagliflozin can mitigate these damaging effects by modulating ER stress pathways. His research also extends to other forms of heart failure, including studies on the protective effects of olive oil against cardiac remodeling following myocardial infarction in rats and the role of oxidative stress versus lipids in monocrotaline-induced pulmonary hypertension. Bagchi has a scholarly h-index of 18 with 1,290 citations across 67 publications. He has collaborated with Amanda J. Stolarz, Rushita A. Bagchi, Soumiya Pal, and Jawahar L. Mehta on multiple projects.
Metrics
- h-index: 18
- Publications: 67
- Citations: 1,290
Selected Publications
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Obesity, histone acetylation, and insulin resistance (2024)
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List of contributors (2024)
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Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats (2024)
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Proteomics Approach to Identify Anthracycline-induced Cardiotoxicity Mechanisms in Human Cardiac Fibroblasts (2024)
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JoVE Video Dataset (2024)
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Real-Time Evaluation of Absolute, Cytosolic, Free Ca<sup>2+</sup> and Corresponding Contractility in Isolated, Pressurized Lymph Vessels (2024)
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Doxorubicin‑induced cardiomyopathy is mitigated by empagliflozin via the modulation of endoplasmic reticulum stress pathways (2024)
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The cardioprotective effects of Empagliflozin in Doxorubicin-induced endoplasmic reticulum (ER) stress (2023)
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Lipidomic Predictors of Coronary No-Reflow (2023)
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Olive oil protects against progression of heart failure by inhibiting remodeling of heart subsequent to myocardial infarction in rats (2022)
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NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases (2022)
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The Structural Basis of Effective LOX-1 Inhibition (2022)
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Interleukin-10 Mitigates Doxorubicin-Induced Endoplasmic Reticulum Stress as Well as Cardiomyopathy (2022)
Collaboration Network
Top Collaborators
- Interleukin-10 Mitigates Doxorubicin-Induced Endoplasmic Reticulum Stress as Well as Cardiomyopathy
- Olive oil protects against progression of heart failure by inhibiting remodeling of heart subsequent to myocardial infarction in rats
- Doxorubicin‑induced cardiomyopathy is mitigated by empagliflozin via the modulation of endoplasmic reticulum stress pathways
- Lipidomic Predictors of Coronary No-Reflow
- The cardioprotective effects of Empagliflozin in Doxorubicin-induced endoplasmic reticulum (ER) stress
- Interleukin-10 Mitigates Doxorubicin-Induced Endoplasmic Reticulum Stress as Well as Cardiomyopathy
- Olive oil protects against progression of heart failure by inhibiting remodeling of heart subsequent to myocardial infarction in rats
- Doxorubicin‑induced cardiomyopathy is mitigated by empagliflozin via the modulation of endoplasmic reticulum stress pathways
- The cardioprotective effects of Empagliflozin in Doxorubicin-induced endoplasmic reticulum (ER) stress
- Interleukin-10 Mitigates Doxorubicin-Induced Endoplasmic Reticulum Stress as Well as Cardiomyopathy
- Olive oil protects against progression of heart failure by inhibiting remodeling of heart subsequent to myocardial infarction in rats
- Doxorubicin‑induced cardiomyopathy is mitigated by empagliflozin via the modulation of endoplasmic reticulum stress pathways
- The cardioprotective effects of Empagliflozin in Doxorubicin-induced endoplasmic reticulum (ER) stress
- Real-Time Evaluation of Absolute, Cytosolic, Free Ca<sup>2+</sup> and Corresponding Contractility in Isolated, Pressurized Lymph Vessels
- JoVE Video Dataset
- Proteomics Approach to Identify Anthracycline-induced Cardiotoxicity Mechanisms in Human Cardiac Fibroblasts
- Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats
- Real-Time Evaluation of Absolute, Cytosolic, Free Ca<sup>2+</sup> and Corresponding Contractility in Isolated, Pressurized Lymph Vessels
- JoVE Video Dataset
- Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats
- Proteomics Approach to Identify Anthracycline-induced Cardiotoxicity Mechanisms in Human Cardiac Fibroblasts
- List of contributors
- Obesity, histone acetylation, and insulin resistance
- Proteomics Approach to Identify Anthracycline-induced Cardiotoxicity Mechanisms in Human Cardiac Fibroblasts
- List of contributors
- Obesity, histone acetylation, and insulin resistance
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
- The Structural Basis of Effective LOX-1 Inhibition
- The Structural Basis of Effective LOX-1 Inhibition
- The Structural Basis of Effective LOX-1 Inhibition
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
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