Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-09
A
Abdelrahman Y. Fouda
Federal Grant PI
High Impact
Associate Professor with tenure
Also affiliated: Cairo University (2016–2026); University of Waterloo (2017); Medical University of South Carolina (2019); Jordan University of Science and Technology (2016); University of Georgia (2013–2019); Augusta University (2014–2023); University of Arkansas Medical Center (2022–2024); Charlie Norwood VA Medical Center (2013–2022); Augusta University Health (2020); Fuzhou University (2014–2018)
Pharmacology & Toxicology, College of Medicine
28 h-index
85 pubs
2,164 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Abdelrahman Y. Fouda investigates mechanisms of neuroprotection and vascular dysfunction, with a focus on conditions such as stroke and diabetes. His work employs animal models to study the roles of specific proteins, including arginase 1 and thioredoxin-interacting protein, in disease progression and potential therapeutic targets.
Fouda's research has been supported by federal grants from the NIH/National Eye Institute, totaling over $500,000. These grants support studies on the role of arginase 1 in retinal ischemia-reperfusion injury and the enhancement of retinal neurovascular protection through efferocytosis. His publication record includes work on cysteine oxidation in angiogenesis, minocycline in cerebral hemorrhage, and obesity-induced vascular dysfunction. He has also contributed to research on retinal ganglion cell repopulation for vision restoration.
With an h-index of 28 and over 2,100 citations across 85 publications, Fouda is recognized as a highly cited researcher. He collaborates with several colleagues at the University of Arkansas for Medical Sciences, including Esraa Shosha and Carol Morris, with whom he has co-authored numerous publications.
Metrics
- h-index: 28
- Publications: 85
- Citations: 2,164
Positions
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Associate Professor with tenure 2025–presentUniversity of Arkansas for Medical Sciences Pharmacology & Toxicology, College of Medicine Institutional directory
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Assistant Professor 2021–2025University of Arkansas for Medical Sciences Pharmacology and Toxicology ORCID
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Postdoctoral fellow 2016–2021Augusta University Vascular Biology Center ORCID
Selected Publications
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Association between primary open-angle glaucoma and retinal artery occlusions (2026)
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CD5L promotes efferocytosis and resolution of retinal ischemic injury (2026)
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Myeloid HDAC3 deletion protects against traumatic optic injury (2026)
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A metabolic switch in retinal angiogenesis: Histone deacetylase 3 (2026)
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Recent advances and future challenges in nanosystems for ocular drug delivery (2025)
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Systemic Lactate Dehydrogenase Levels as a Predictor of Progression from Non-Proliferative to Proliferative Diabetic Retinopathy (2025)
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HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis (2025)
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Myeloid HDAC3 Deletion Protects Against Traumatic Optic Injury (2025)
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Extracellular Vesicle Metabolomics Holds Promise for Adult Axon Regeneration (2025)
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Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy (2025)
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HDAC3 Drives Retinal Endothelial Cell Angiogenesis: Potential Therapeutic Implications for Retinopathy (Abstract ID: 161164) (2025)
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Disrupting the CD47/SIRPα Axis as a Novel and Translational Therapy for Stroke (Abstract ID: 161422) (2025)
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Efferocytosis and retinal clean-up: Role of histone deacetylase 3 in ischemic retinopathy (2025)
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Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology (2024)
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Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury (2024)
Federal Grants 2 $502,702 total
NIH
Contact PI
Sep 2024 - May 2029
NIH
Contact PI
Sep 2019 - Jan 2026
Collaboration Network
Top Collaborators
Esraa Shosha
Cairo University
14 shared publications
In database
- Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair
- Preclinical investigation of Pegylated arginase 1 as a treatment for retina and brain injury
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Investigation of Retinal Metabolic Function in Type 1 Diabetic Akita Mice
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
Showing 5 of 14 shared publications
Ruth B. Caldwell
Augusta University (US)
12 shared publications
- Cysteine oxidation of copper transporter CTR1 drives VEGFR2 signalling and angiogenesis
- Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair
- Preclinical investigation of Pegylated arginase 1 as a treatment for retina and brain injury
- Preclinical investigation of Pegylated arginase 1 as a treatment for retina and brain injury
- Preclinical investigation of Pegylated arginase 1 as a treatment for retina and brain injury
Showing 5 of 12 shared publications
Rami Ahmad Shahror
University of Arkansas for Medical Sciences
9 shared publications
In database
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Recent advances and future challenges in nanosystems for ocular drug delivery
- Multi-color Flow Cytometry Protocol to Characterize Myeloid Cells in Mouse Retina Research
Showing 5 of 9 shared publications
Carol Morris
University of Arkansas for Medical Sciences
9 shared publications
In database
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Multi-color Flow Cytometry Protocol to Characterize Myeloid Cells in Mouse Retina Research
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
Showing 5 of 9 shared publications
Nancy J. Rusch
University of Arkansas for Medical Sciences
6 shared publications
In database
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- Opinion: Endothelial Cells - Macrophage-Like Gatekeepers?
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
Showing 5 of 6 shared publications
Melissa Wild
University of Arkansas for Medical Sciences
6 shared publications
In database
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy
Showing 5 of 6 shared publications
Zhimin Xu
Augusta University (US)
5 shared publications
- Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair
- Preclinical investigation of Pegylated arginase 1 as a treatment for retina and brain injury
- Investigation of Retinal Metabolic Function in Type 1 Diabetic Akita Mice
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- Calbindin 2-specific deletion of arginase 2 preserves visual function after optic nerve crush
Tahira Lemtalsi
Augusta University (US)
4 shared publications
- Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair
- Preclinical investigation of Pegylated arginase 1 as a treatment for retina and brain injury
- Investigation of Retinal Metabolic Function in Type 1 Diabetic Akita Mice
- Calbindin 2-specific deletion of arginase 2 preserves visual function after optic nerve crush
Syed Zaidi
Augusta University (US)
4 shared publications
- Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair
- Preclinical investigation of Pegylated arginase 1 as a treatment for retina and brain injury
- Investigation of Retinal Metabolic Function in Type 1 Diabetic Akita Mice
- Calbindin 2-specific deletion of arginase 2 preserves visual function after optic nerve crush
S. Priya Narayanan
University of Georgia (US)
4 shared publications
- Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair
- Preclinical investigation of Pegylated arginase 1 as a treatment for retina and brain injury
- Calbindin 2-specific deletion of arginase 2 preserves visual function after optic nerve crush
- The mouse model of experimental autoimmune encephalomyelitis: Methods to study neuroinflammation in optic neuritis
Robert W. Caldwell
Augusta University (US)
4 shared publications
- Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair
- Preclinical investigation of Pegylated arginase 1 as a treatment for retina and brain injury
- Investigation of Retinal Metabolic Function in Type 1 Diabetic Akita Mice
- Calbindin 2-specific deletion of arginase 2 preserves visual function after optic nerve crush
Christian D. Mitchell
University of Arkansas for Medical Sciences
4 shared publications
In database
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- HDAC3 Drives Retinal Endothelial Cell Angiogenesis: Potential Therapeutic Implications for Retinopathy (Abstract ID: 161164)
Shengyu Mu
University of Arkansas for Medical Sciences
3 shared publications
In database
- Opinion: Endothelial Cells - Macrophage-Like Gatekeepers?
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
Modesto Rojas
Augusta University (US)
3 shared publications
- Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair
- Investigation of Retinal Metabolic Function in Type 1 Diabetic Akita Mice
- Calbindin 2-specific deletion of arginase 2 preserves visual function after optic nerve crush
Bushra Zaman
University of Arkansas for Medical Sciences
3 shared publications
In database
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
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