Match tier
Listed
Presence
Current · Arkansas
Last published
2024
Sources
OpenAlex · ORCID
Refreshed
2026-08-08
H
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Hayley M. Theriot investigates host-pathogen interactions, focusing on the mechanisms of bacterial infections. Her research has examined the role of interleukin-17 in neutrophil infiltration into the lungs during pneumonic plague, a severe and often fatal form of the disease caused by *Yersinia pestis*. Theriot also studies disease severity in animal models, including work on a mouse model of Down syndrome to understand heightened mortality in SARS-CoV-2 infection.
Her work involves studying specific biological responses and cellular mechanisms within disease models. Theriot collaborates with researchers at the University of Arkansas for Medical Sciences, including Priyangi A. Malaviarachchi, Lu Huang, Zijing Zhang, and Samantha D. Crane, on shared publications. Her research output includes two publications with 10 total citations and an h-index of 2.
Metrics
- h-index: 2
- Publications: 2
- Citations: 10
Selected Publications
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SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome (2024)
-
Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague (2023)
Collaboration Network
Top Collaborators
Priyangi A. Malaviarachchi
University of Arkansas for Medical Sciences
2 shared publications
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Roger D. Pechous
University of Arkansas for Medical Sciences (US)
2 shared publications
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Madeleine G. Scott
University of Arkansas for Medical Sciences
1 shared publication
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
Kenneth T. Appell
University of Arkansas for Medical Sciences
1 shared publication
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
Srijon K. Banerjee
University of Pittsburgh
1 shared publication
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
Zhuo Xing
Cancer Genetics (United States) (US)
1 shared publication
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
A. R. Douglas
Cancer Genetics (United States) (US)
1 shared publication
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Samantha D. Crane
University of Arkansas for Medical Sciences
1 shared publication
In database
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Zijing Zhang
University of Arkansas for Medical Sciences
1 shared publication
In database
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Alireza Ghaffarieh
University of Arkansas for Medical Sciences (US)
1 shared publication
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Lu Huang
University of Arkansas for Medical Sciences
1 shared publication
In database
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Yichao Yu
Cancer Genetics (United States) (US)
1 shared publication
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Xuming Zhang
University of Arkansas for Medical Sciences (US)
1 shared publication
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
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