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Presence Current · Arkansas
Last published 2026
Sources OpenAlex · ORCID
Refreshed 2026-08-15

Tiffany Weinkopff

Assistant Professor

Also affiliated: University of Maryland, Baltimore (2018); Centers for Disease Control and Prevention (2014); University of Georgia (2009–2014); University of Pennsylvania (2015–2016); University of Lausanne (2012–2015)

Faculty Researcher

Microbiology & Immunology, College of Medicine

14 h-index 40 pubs 687 cited

  • Animals
  • Mice
  • Leishmaniasis, Cutaneous
  • Leishmania major
  • Mice, Inbred C57BL
  • Female
  • Humans
  • Signal Transduction
  • Macrophages
  • Endothelial Cells
  • Leishmania braziliensis
  • Disease Models, Animal
  • Neutrophil Activation
  • Elephantiasis, Filarial
  • Lymphangiogenesis

Biography and Research Information

OverviewAI-generated summary

Tiffany Weinkopff is an Assistant Professor in the Department of Microbiology & Immunology at the University of Arkansas for Medical Sciences. Her research focuses on understanding the host-pathogen interactions during parasitic infections, particularly cutaneous leishmaniasis. Weinkopff investigates how the host immune system responds to infection by *Leishmania major*, employing single-cell resolution techniques to analyze cellular heterogeneity and transcriptomic profiling in infected mice.

Her work has explored the role of signaling pathways, such as Hypoxia-Inducible Factor (HIF) signaling, in modulating macrophage function and promoting lymphangiogenesis during infection. She also studies the impact of the inflammatory microenvironment on macrophage transcriptional remodeling in leishmanial lesions. Weinkopff has published 40 papers, accumulating 679 citations and an h-index of 14. Her recent collaborations include work with Charity L. Washam, Manjunath Bettadapura, Steven Murdock, and Hayden Roys, all at the University of Arkansas for Medical Sciences.

Metrics

  • h-index: 14
  • Publications: 40
  • Citations: 687

Selected Publications

  • Guanylate-binding proteins balance iNOS/Arg-1 in myeloid cells during <i>L. major</i> infection and promote host defense to infection (2026)
    mBio DOI OpenAlex
  • Enhanced translational activity is linked to lymphatic endothelial cell activation in cutaneous leishmaniasis 4267 (2025)
    The Journal of Immunology DOI OpenAlex
  • Impaired lymphatic remodeling leads to immunopathology during non-healing cutaneous leishmaniasis 4376 (2025)
    The Journal of Immunology DOI OpenAlex
  • From defense to disease: the multifaceted role of lymphatics during infection (2025)
    The Journal of Immunology DOI OpenAlex
  • Guanylate-Binding Proteins Promote Host Defense Against <i>Leishmania major</i> by Balancing iNOS/Arg-1 in Myeloid Cells (2025)
    bioRxiv (Cold Spring Harbor Laboratory) DOI OpenAlex
  • Dysregulated lymphatic remodeling promotes immunopathology during non-healing cutaneous leishmaniasis (2025)
    bioRxiv (Cold Spring Harbor Laboratory) DOI OpenAlex
  • Enhanced Translational Activity Is Linked to Lymphatic Endothelial Cell Activation in Cutaneous Leishmaniasis (2025)
    Lymphatic Research and Biology 1 citation DOI OpenAlex
  • HIF-α signaling regulates the macrophage inflammatory response during Leishmania major infection (2025)
    Frontiers in Immunology 2 citations DOI OpenAlex
  • Platelets Modulate Leukocyte Population Composition Within Perivascular Adipose Tissue (2025)
    International Journal of Molecular Sciences 1 citation DOI OpenAlex
  • Host guanylate binding proteins participate in parasite control during Leishmania infection (2024)
    The Journal of Immunology DOI OpenAlex
  • HIF-α signaling regulates the macrophage inflammatory response during <i>Leishmania major</i> infection (2024)
    bioRxiv (Cold Spring Harbor Laboratory) DOI OpenAlex
  • Synthesis and in vitro evaluation shows disquaramide compounds are a promising class of anti-leishmanial drugs (2024)
    bioRxiv (Cold Spring Harbor Laboratory) DOI OpenAlex
  • Enhanced translational activity is linked to lymphatic endothelial cell activation in cutaneous leishmaniasis (2024)
    bioRxiv (Cold Spring Harbor Laboratory) 2 citations DOI OpenAlex
  • Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis (2024)
    Mucosal Immunology 35 citations DOI OpenAlex
  • Investigating the platelet contribution to diet induced PVAT leukocyte infiltration (2024)
    Physiology DOI OpenAlex

View all publications on OpenAlex →

Research Interests

Leishmania, leishmaniasis, lymphatics, macrophages, vascular remodeling

Grants & Funding

As listed on this researcher's institutional profile.

  • Partnerships for Biomedical Research in Arkansas - Admin Supplement NIH/Nat. Inst. of General Medical Sciences Principal Investigator
  • Partnerships for Biomedical Research in Arkansas NIH Co-Investigator
  • The Role of Myeloid Lineage Cells in Leishmaniasis NIH Principal Investigator
  • Regulation of macrophage function during acute infection with Plasmodium NIH Co-Investigator

Collaboration Network

75 Collaborators 18 Institutions 3 Countries

Top Collaborators

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