Jeffrey M. Sifford
Researcher
Also affiliated: St. Jude Children's Research Hospital (2017–2020)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Jeffrey M. Sifford investigates host-pathogen interactions, with a focus on viral infections and their impact on cellular processes. His research has explored the intrinsic activation of p53 as a mechanism to restrict gammaherpesvirus-driven B cell expansion during the establishment of latency. Sifford's work has been published in peer-reviewed journals, and he has contributed to 11 publications with a total of 839 citations, holding an h-index of 7. He has collaborated with researchers at the University of Arkansas for Medical Sciences, including Jason S. Stumhofer, J. Craig Forrest, Mark Manzano, and Shana M. Owens, with whom he shares multiple publications. Sifford's recent activity indicates ongoing research in his areas of expertise.
Metrics
- h-index: 7
- Publications: 11
- Citations: 879
Selected Publications
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Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment (2025)
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p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated <i>IgH/c-Myc</i> Translocations (2020)
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Conditional mutagenesis in vivo reveals cell type- and infection stage-specific requirements for LANA in chronic MHV68 infection (2018)
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Murine Gammaherpesvirus 68 LANA and SOX Homologs Counteract ATM-Driven p53 Activity during Lytic Viral Replication (2015)
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Phosphoproteomic Analyses Reveal Signaling Pathways That Facilitate Lytic Gammaherpesvirus Replication (2013)
Collaboration Network
Top Collaborators
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
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