Iad Alhallak
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Researcher
Also affiliated: Memorial Hermann (2025–2026); Harvard University (2022–2025); Massachusetts General Hospital (2022–2025); Memorial Hermann–Texas Medical Center (2026); Conway School of Landscape Design (2022); The University of Texas Medical Branch at Galveston (2025); The University of Texas Health Science Center at Houston (2025–2026)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Iad Alhallak's research focuses on metabolic pathways and their influence on disease development, particularly in the context of obesity and liver disease. Recent work has investigated the role of Malic Enzyme 1 (ME1) in promoting adiposity and hepatic steatosis, and its connection to circulating insulin and leptin levels in obese female mice. Alhallak has also explored the impact of diet on endometriosis development in mice and the regulation of breast adipose tissue. In parallel, his work has examined immune system involvement in human immunodeficiency virus (HIV) infection, specifically arterial infiltration by macrophages and its relation to noncalcified plaque and systemic immune activation.
Further investigations have delved into the molecular mechanisms of oncogenesis, studying the function of Krüppel-like Factors (KLFs) in suppressing oxidative stress and inflammation in hepatocellular carcinoma. Alhallak's scholarship includes 30 publications with an h-index of 9 and 242 total citations. Key collaborators include Rosalia C. M. Simmen, Frank A. Simmen, Charles M. Quick, and Garrett L. Graham, all from the University of Arkansas for Medical Sciences.
Metrics
- h-index: 9
- Publications: 31
- Citations: 247
Selected Publications
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26-CCC-10096-ACC CHEMOTHERAPY-INDUCED CARDIOTOXICITY VS LEFT BUNDLE BRANCH BLOCK CARDIOMYOPATHY: A DIAGNOSTIC AND MANAGEMENT CHALLENGE (2026)
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Krüppel-like Factor-9 and Krüppel-like Factor-13: Highly Related, Multi-Functional, Transcriptional Repressors and Activators of Oncogenesis (2023)
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Prior Statin vs In-Hospital Statin Usage in Severe COVID-19: Review and Meta-Analysis (2023)
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Malic Enzyme 1 (ME1) Promotes Adiposity and Hepatic Steatosis and Induces Circulating Insulin and Leptin in Obese Female Mice (2023)
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A Pilot Study on the Co-existence of Diabetes and Endometriosis in Reproductive-Age Women: Potential for Endometriosis Progression (2023)
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Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet (2022)
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Modelling the adsorption of phospholipid vesicles to a silicon dioxide surface using Langmuir kinetics (2022)
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Lesion Genotype Modifies High-Fat Diet Effects on Endometriosis Development in Mice (2021)
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Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10 (2021)
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Co-Morbidity of Type 1 Diabetes Promotes Endometriosis Status-A Pilot Study (2021)
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Metformin regulation of progesterone receptor isoform‑B expression in human endometrial cancer cells is glucose‑dependent (2020)
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Malic enzyme 1 (ME1) in the biology of cancer: it is not just intermediary metabolism (2020)
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Impact of Student-Run Free Clinics in Urgent Care (2020)
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SUN-126 Distinct Molecular Phenotypes of Non-Diseased Breast Adipose Tissue of Pre-Menopausal Obese and Non-Obese Women May Underlie Differing Breast Cancer Risks (2020)
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Metformin Promotes Anti-tumor Biomarkers in Human Endometrial Cancer Cells (2020)
Collaboration Network
Top Collaborators
- Malic Enzyme 1 (ME1) Promotes Adiposity and Hepatic Steatosis and Induces Circulating Insulin and Leptin in Obese Female Mice
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
- Krüppel-like Factor-9 and Krüppel-like Factor-13: Highly Related, Multi-Functional, Transcriptional Repressors and Activators of Oncogenesis
- A Pilot Study on the Co-existence of Diabetes and Endometriosis in Reproductive-Age Women: Potential for Endometriosis Progression
- Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10
Showing 5 of 7 shared publications
- Malic Enzyme 1 (ME1) Promotes Adiposity and Hepatic Steatosis and Induces Circulating Insulin and Leptin in Obese Female Mice
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
- Krüppel-like Factor-9 and Krüppel-like Factor-13: Highly Related, Multi-Functional, Transcriptional Repressors and Activators of Oncogenesis
- Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10
- Lesion Genotype Modifies High-Fat Diet Effects on Endometriosis Development in Mice
- A Pilot Study on the Co-existence of Diabetes and Endometriosis in Reproductive-Age Women: Potential for Endometriosis Progression
- Co-Morbidity of Type 1 Diabetes Promotes Endometriosis Status-A Pilot Study
- A Pilot Study on the Co-existence of Diabetes and Endometriosis in Reproductive-Age Women: Potential for Endometriosis Progression
- Co-Morbidity of Type 1 Diabetes Promotes Endometriosis Status-A Pilot Study
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
- Lesion Genotype Modifies High-Fat Diet Effects on Endometriosis Development in Mice
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
- Lesion Genotype Modifies High-Fat Diet Effects on Endometriosis Development in Mice
- Malic Enzyme 1 (ME1) Promotes Adiposity and Hepatic Steatosis and Induces Circulating Insulin and Leptin in Obese Female Mice
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
- Prior Statin vs In-Hospital Statin Usage in Severe COVID-19: Review and Meta-Analysis
- 26-CCC-10096-ACC CHEMOTHERAPY-INDUCED CARDIOTOXICITY VS LEFT BUNDLE BRANCH BLOCK CARDIOMYOPATHY: A DIAGNOSTIC AND MANAGEMENT CHALLENGE
- Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10
- Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10
- Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10
- Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10
- Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10
- Modelling the adsorption of phospholipid vesicles to a silicon dioxide surface using Langmuir kinetics
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
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