Joseph J. Goellner
Research Instructor
faculty
Internal Med, College of Medicine
19 h-index
29 pubs
4,012 cited
Research Areas
Biography and Research Information
OverviewAI-generated summary
Joseph J. Goellner investigates bone remodeling and the cellular mechanisms that regulate it. His research focuses on the roles of osteoblasts, osteocytes, and osteoclasts in bone maintenance and the potential contributions of these cells to conditions like rebound resorption following drug discontinuation. Goellner's work also explores the impact of therapeutic agents on bone metabolism, specifically examining how potent suppressors of bone remodeling interact with anabolic responses. He has published 29 papers with 3,986 citations and an h-index of 19. Goellner collaborates with researchers at the University of Arkansas for Medical Sciences, including Charles A. O’Brien and Jeff D. Thostenson, with whom he has co-authored multiple publications.
Metrics
- h-index: 19
- Publications: 29
- Citations: 4,012
Selected Publications
-
Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice (2025)
-
Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation (2023)
Grants & Funding
- Center for Musculoskeletal Disease Research (CMDR) NIH/Nat. Inst. of General Medical Sciences Co-Investigator
- Center for Musculoskeletal Disease Research (CMDR) NIH/Nat. Inst. of General Medical Sciences Principal Investigator
Collaboration Network
Top Collaborators
Jeff D. Thostenson
University of Arkansas for Medical Sciences
3 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
- Local Production of Osteoprotegerin by Osteoblasts Suppresses Bone Resorption
Charles A. O’Brien
University of Arkansas for Medical Sciences
3 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
- Local Production of Osteoprotegerin by Osteoblasts Suppresses Bone Resorption
Qiang Fu
University of Arkansas for Medical Sciences
2 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
Nancy Cecile Bustamante-Gomez
2 shared publications
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Local Production of Osteoprotegerin by Osteoblasts Suppresses Bone Resorption
Humberto Reyes-Pardo
University of Arkansas for Medical Sciences
2 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
Igor Gubrij
2 shared publications
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Local Production of Osteoprotegerin by Osteoblasts Suppresses Bone Resorption
Michela Palmieri
2 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Local Production of Osteoprotegerin by Osteoblasts Suppresses Bone Resorption
Diana Escalona‐Vargas
University of Arkansas for Medical Sciences
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
Intawat Nookaew
Institute for Musculoskeletal Health
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
C. Lowry Barnes
Institute for Musculoskeletal Health
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
Jeffrey B. Stambough
Institute for Musculoskeletal Health
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
Elena Ambrogini
Central Arkansas Veterans Healthcare System
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
Keisha M. Cawley
1 shared publication
- Local Production of Osteoprotegerin by Osteoblasts Suppresses Bone Resorption
Anveshi G. Guha
1 shared publication
- Local Production of Osteoprotegerin by Osteoblasts Suppresses Bone Resorption
Ryan S. MacLeod
1 shared publication
- Local Production of Osteoprotegerin by Osteoblasts Suppresses Bone Resorption
Similar Researchers
Based on overlapping research topics
Scott A. Stuart
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Pharmacological Effects and Toxicity Studies
John T. Sherrill
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Pharmacological Effects and Toxicity Studies
Nisreen Akel
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Pharmacological Effects and Toxicity Studies
Amy Y. Sato
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Pharmacological Effects and Toxicity Studies
Cecile Bustamante-Gomez
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Pharmacological Effects and Toxicity Studies
Gareeballah Osman Adam
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Pharmacological Effects and Toxicity Studies