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Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
H
Humberto Reyes-Pardo
Post Doctoral Fellow
Also affiliated: Autonomous University of San Luis Potosí (2012); Central Arkansas Veterans Healthcare System (2026); Centro de Investigación y de Estudios Avanzados del Instituto Politécnico Nacional (2019); Tecnológico de Monterrey (2022)
Internal Med, College of Medicine
9 h-index
13 pubs
418 cited
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Humberto Reyes-Pardo's research focuses on bone metabolism and related cellular processes. He has investigated the role of RANKL-mediated osteoclast formation in bone loss associated with Staphylococcus aureus osteomyelitis, utilizing a murine model. His work also explores the mechanisms contributing to rebound bone resorption following the discontinuation of denosumab, specifically examining the expression of osteoprotegerin by osteocytes.
Reyes-Pardo has also contributed to reviews on the applications of microalgae bioactive compounds and photolyase production, as well as technologies for detecting and treating per- and polyfluoroalkyl substances in wastewater. His research interests extend to the mechanical properties of sensory neurons in the context of diabetes mellitus and its relation to neuropathy. He has published 13 papers, with an h-index of 9 and 372 citations.
Metrics
- h-index: 9
- Publications: 13
- Citations: 418
Positions
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Post Doctoral Fellow 2022–presentUniversity of Arkansas for Medical Sciences Internal Med, College of Medicine Institutional directory
Selected Publications
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Mechanistic insights into the pathogenesis and therapeutic recalcitrance of Staphylococcus aureus osteomyelitis (2026)
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Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice (2025)
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RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis (2024)
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Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation (2023)
Collaboration Network
Top Collaborators
Charles A. O’Brien
Central Arkansas Veterans Healthcare System
4 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
- Mechanistic insights into the pathogenesis and therapeutic recalcitrance of Staphylococcus aureus osteomyelitis
Qiang Fu
University of Arkansas for Medical Sciences
2 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
Jeff D. Thostenson
University of Arkansas for Medical Sciences
2 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
Joseph J. Goellner
University of Arkansas for Medical Sciences
2 shared publications
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
Mara J. Campbell
University of Arkansas for Medical Sciences
2 shared publications
In database
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
- Mechanistic insights into the pathogenesis and therapeutic recalcitrance of Staphylococcus aureus osteomyelitis
Cecile Bustamante-Gomez
University of Arkansas for Medical Sciences
2 shared publications
In database
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
Karen E. Beenken
University of Arkansas for Medical Sciences
2 shared publications
In database
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
- Mechanistic insights into the pathogenesis and therapeutic recalcitrance of Staphylococcus aureus osteomyelitis
Mark S. Smeltzer
University of Arkansas for Medical Sciences
2 shared publications
In database
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
- Mechanistic insights into the pathogenesis and therapeutic recalcitrance of Staphylococcus aureus osteomyelitis
Nancy Cecile Bustamante-Gomez
University of Arkansas for Medical Sciences
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
Igor Gubrij
University of Arkansas for Medical Sciences (US)
1 shared publication
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
Diana Escalona‐Vargas
Pediatrics and Genetics
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
Michela Palmieri
University of Arkansas for Medical Sciences
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
Intawat Nookaew
University of Arkansas for Medical Sciences
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
C. Lowry Barnes
University of Arkansas for Medical Sciences
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
Jeffrey B. Stambough
University of Arkansas for Medical Sciences
1 shared publication
In database
- Reduced osteoprotegerin expression by osteocytes may contribute to rebound resorption after denosumab discontinuation
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