Paul E. Gottschall
Researcher
Also affiliated: Semmelweis University (1990); Tulane University (1986–2008); University of Hawaiʻi at Mānoa (2001); University of Hawaii System (2001); Harvard University (2001); Heidelberg University (2012); University of South Florida (1993–2010); Hadassah Medical Center (2001); University of California San Diego (1984); National Institute of Environmental Health Sciences (2012); Queen's Medical Center (2001); Shin Nippon Biomedical Laboratories (Japan) (1991); Shriners Hospitals for Children - Tampa (2005); Biomedical Research Laboratories (United States) (1986–2008); Nagoya University (1991); University of South Alabama (1990); Michigan State University (1982–2008); Universidad Nacional Autónoma de México (2001); Saarland University (2012); Florida College (1993–2008)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Paul E. Gottschall's research has focused on the study of cancer cells and metastasis, as well as the mechanisms of cancer cell death. His work has investigated the role of various signaling pathways and molecules in cancer progression and the development of therapeutic strategies. Gottschall has also explored DNA repair mechanisms and their implications in cancer treatment.
Metrics
- h-index: 50
- Publications: 106
- Citations: 8,474
Selected Publications
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Neuronal cells derived from human induced pluripotent stem cells as a functional tool of melanocortin system (2017)
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Hippocampal administration of chondroitinase ABC increases plaque-adjacent synaptic marker and diminishes amyloid burden in aged APPswe/PS1dE9 mice (2015)
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ADAMTS expression and function in central nervous system injury and disorders (2015)
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Altered Synaptic Marker Abundance in the Hippocampal Stratum Oriens of Ts65Dn Mice is Associated with Exuberant Expression of Versican (2012)
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Selective Decline of Synaptic Protein Levels in the Frontal Cortex of Female Mice Deficient in the Extracellular Metalloproteinase ADAMTS1 (2012)
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Lectican proteoglycans, their cleaving metalloproteinases, and plasticity in the central nervous system extracellular microenvironment (2012)
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Canonical Transient Receptor Channel 5 (TRPC5) and TRPC1/4 Contribute to Seizure and Excitotoxicity by Distinct Cellular Mechanisms (2012)
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Important role for endothelial calveolin‐1 in focal cerebral ischemia‐induced blood–brain barrier injury (2011)
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Trafficking CD11b-Positive Blood Cells Deliver Therapeutic Genes to the Brain of Amyloid-Depositing Transgenic Mice (2010)
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Panel of synaptic protein ELISAs for evaluating neurological phenotype (2010)
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Abnormal post‐translational and extracellular processing of brevican in plaque‐bearing mice over‐expressing APPsw (2010)
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Inhibition of gelatinase activity reduces neural injury in an ex vivo model of hypoxia–ischemia (2009)
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Discordant localization of WFA reactivity and brevican/ADAMTS-derived fragment in rodent brain (2008)
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Delayed administration of a matrix metalloproteinase inhibitor limits progressive brain injury after hypoxia-ischemia in the neonatal rat (2008)
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Adeno-associated Viral (AAV) Serotype 5 Vector Mediated Gene Delivery of Endothelin-converting Enzyme Reduces Aβ Deposits in APP + PS1 Transgenic Mice (2008)
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