Research Areas
Links
Biography and Research Information
OverviewAI-generated summary
Taylor McElroy's research investigates the physiological and cognitive impacts of various treatments and environmental factors, with a particular focus on sex differences and the role of mitochondria. McElroy has studied the effects of chemotherapeutic agents such as cyclophosphamide, docetaxel, and doxorubicin on cognitive function and physiological changes, examining implications of the gut microbiome and depressive-like behavior. Further work has explored the neuroprotective potential of piperlongumine against chemotherapy-induced cognitive impairment and the effects of simulated galactic cosmic rays on cognitive performance in mice. Research also extends to comparative toxicities of endocrine-disrupting chemicals in model organisms and the role of mitochondria in neuronal recovery after injury. Studies have also addressed health disparities related to frailty in older adults and iron homeostasis.
Metrics
- h-index: 13
- Publications: 37
- Citations: 485
Selected Publications
-
Changes in gut, microbiome, and cognition after doxorubicin, cyclophosphamide, and paclitaxel chemotherapy treatment (2026)
-
The adverse effects of chemotherapy on bone mass are not prevented by senolytics (2025)
-
Physiological and cognitive changes after treatments of cyclophosphamide, methotrexate, and fluorouracil: implications of the gut microbiome and depressive-like behavior (2023)
-
Piperlongumine as a Neuro-Protectant in Chemotherapy Induced Cognitive Impairment (2022)
-
Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue (2021)
-
Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue (2021)
-
Effects of 16O charged-particle irradiation on cognition, hippocampal morphology and mutagenesis in female mice (2021)
-
Cognitive impairment resulting from treatment with docetaxel, doxorubicin, and cyclophosphamide (2021)
Collaboration Network
Top Collaborators
- Sex differences in frailty among older adults
- Comparative toxicities of BPA, BPS, BPF, and TMBPF in the nematode Caenorhabditis elegans and mammalian fibroblast cells
- The role of mitochondria in the recovery of neurons after injury
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 12 shared publications
- Comparative toxicities of BPA, BPS, BPF, and TMBPF in the nematode Caenorhabditis elegans and mammalian fibroblast cells
- The role of mitochondria in the recovery of neurons after injury
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Bisphenol TMC disturbs mitochondrial activity and biogenesis, reducing lifespan and healthspan in the nematode <i>Caenorhabditis elegans</i>
Showing 5 of 11 shared publications
- Comparative toxicities of BPA, BPS, BPF, and TMBPF in the nematode Caenorhabditis elegans and mammalian fibroblast cells
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Bisphenol TMC disturbs mitochondrial activity and biogenesis, reducing lifespan and healthspan in the nematode <i>Caenorhabditis elegans</i>
- 6-PPD induces mitochondrial dysfunction and reduces healthspan and lifespan through SKN-1 in Caenorhabditis elegans
Showing 5 of 10 shared publications
- Cognitive impairment resulting from treatment with docetaxel, doxorubicin, and cyclophosphamide
- Piperlongumine as a Neuro-Protectant in Chemotherapy Induced Cognitive Impairment
- Physiological and cognitive changes after treatments of cyclophosphamide, methotrexate, and fluorouracil: implications of the gut microbiome and depressive-like behavior
- Evaluating the effects of low-dose simulated galactic cosmic rays on murine hippocampal-dependent cognitive performance
- Effects of 16O charged-particle irradiation on cognition, hippocampal morphology and mutagenesis in female mice
Showing 5 of 9 shared publications
- Cognitive impairment resulting from treatment with docetaxel, doxorubicin, and cyclophosphamide
- Piperlongumine as a Neuro-Protectant in Chemotherapy Induced Cognitive Impairment
- Physiological and cognitive changes after treatments of cyclophosphamide, methotrexate, and fluorouracil: implications of the gut microbiome and depressive-like behavior
- Evaluating the effects of low-dose simulated galactic cosmic rays on murine hippocampal-dependent cognitive performance
- Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue
Showing 5 of 8 shared publications
- Comparative toxicities of BPA, BPS, BPF, and TMBPF in the nematode Caenorhabditis elegans and mammalian fibroblast cells
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #1 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 7 shared publications
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- 6-PPD induces mitochondrial dysfunction and reduces healthspan and lifespan through SKN-1 in Caenorhabditis elegans
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #1 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 7 shared publications
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #1 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #3 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 6 shared publications
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #1 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #3 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 6 shared publications
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #3 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #2 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 6 shared publications
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #1 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #3 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 6 shared publications
- Piperlongumine as a Neuro-Protectant in Chemotherapy Induced Cognitive Impairment
- Physiological and cognitive changes after treatments of cyclophosphamide, methotrexate, and fluorouracil: implications of the gut microbiome and depressive-like behavior
- Evaluating the effects of low-dose simulated galactic cosmic rays on murine hippocampal-dependent cognitive performance
- Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue
- Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #1 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #2 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Cognitive impairment resulting from treatment with docetaxel, doxorubicin, and cyclophosphamide
- Piperlongumine as a Neuro-Protectant in Chemotherapy Induced Cognitive Impairment
- Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue
- Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue
- Cognitive impairment resulting from treatment with docetaxel, doxorubicin, and cyclophosphamide
- Piperlongumine as a Neuro-Protectant in Chemotherapy Induced Cognitive Impairment
- Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue
- Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue
Similar Researchers
Based on overlapping research topics