Iad Alhallak
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Also affiliated: Memorial Hermann (2025–2026); Texas Medical Center (2026); Harvard University (2022–2025); Massachusetts General Hospital (2022–2025); Memorial Hermann–Texas Medical Center (2026); The University of Texas Medical Branch at Galveston (2025); The University of Texas Health Science Center at Houston (2025–2026)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Iad Alhallak's research focuses on metabolic pathways and their roles in disease, particularly in the context of obesity and cancer. Recent work has investigated the role of malic enzyme 1 (ME1) in promoting adiposity, hepatic steatosis, and the induction of circulating insulin and leptin in obese female mice. Other studies have examined Krüppel-like Factor 9 (KLF9) in suppressing hepatocellular carcinoma-promoting oxidative stress and inflammation in mice fed a high-fat diet, and the impact of metformin on anti-tumor biomarkers in human endometrial cancer cells. Alhallak also contributes to research on HIV, with a publication exploring macrophage infiltration related to noncalcified plaque and systemic immune activation in individuals with HIV. Collaborations include work with Rosalia C. M. Simmen and Frank A. Simmen at the University of Arkansas for Medical Sciences, with whom Alhallak has co-authored multiple publications.
Metrics
- h-index: 9
- Publications: 32
- Citations: 252
Positions
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Resident 2024–2027The University of Texas Health Science Center at Houston Internal Medicine ORCID
Selected Publications
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26-CCC-10096-ACC CHEMOTHERAPY-INDUCED CARDIOTOXICITY VS LEFT BUNDLE BRANCH BLOCK CARDIOMYOPATHY: A DIAGNOSTIC AND MANAGEMENT CHALLENGE (2026)
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Krüppel-like Factor-9 and Krüppel-like Factor-13: Highly Related, Multi-Functional, Transcriptional Repressors and Activators of Oncogenesis (2023)
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Prior Statin vs In-Hospital Statin Usage in Severe COVID-19: Review and Meta-Analysis (2023)
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Malic Enzyme 1 (ME1) Promotes Adiposity and Hepatic Steatosis and Induces Circulating Insulin and Leptin in Obese Female Mice (2023)
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A Pilot Study on the Co-existence of Diabetes and Endometriosis in Reproductive-Age Women: Potential for Endometriosis Progression (2023)
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Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet (2022)
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Modelling the adsorption of phospholipid vesicles to a silicon dioxide surface using Langmuir kinetics (2022)
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Lesion Genotype Modifies High-Fat Diet Effects on Endometriosis Development in Mice (2021)
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Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10 (2021)
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Co-Morbidity of Type 1 Diabetes Promotes Endometriosis Status-A Pilot Study (2021)
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Metformin regulation of progesterone receptor isoform‑B expression in human endometrial cancer cells is glucose‑dependent (2020)
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Malic enzyme 1 (ME1) in the biology of cancer: it is not just intermediary metabolism (2020)
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Impact of Student-Run Free Clinics in Urgent Care (2020)
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SUN-126 Distinct Molecular Phenotypes of Non-Diseased Breast Adipose Tissue of Pre-Menopausal Obese and Non-Obese Women May Underlie Differing Breast Cancer Risks (2020)
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Metformin Promotes Anti-tumor Biomarkers in Human Endometrial Cancer Cells (2020)
Collaboration Network
Top Collaborators
- Malic enzyme 1 (ME1) in the biology of cancer: it is not just intermediary metabolism
- Malic Enzyme 1 (ME1) Promotes Adiposity and Hepatic Steatosis and Induces Circulating Insulin and Leptin in Obese Female Mice
- Metformin Promotes Anti-tumor Biomarkers in Human Endometrial Cancer Cells
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
- Krüppel-like Factor-9 and Krüppel-like Factor-13: Highly Related, Multi-Functional, Transcriptional Repressors and Activators of Oncogenesis
Showing 5 of 12 shared publications
- Malic enzyme 1 (ME1) in the biology of cancer: it is not just intermediary metabolism
- Malic Enzyme 1 (ME1) Promotes Adiposity and Hepatic Steatosis and Induces Circulating Insulin and Leptin in Obese Female Mice
- Metformin Promotes Anti-tumor Biomarkers in Human Endometrial Cancer Cells
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
- Krüppel-like Factor-9 and Krüppel-like Factor-13: Highly Related, Multi-Functional, Transcriptional Repressors and Activators of Oncogenesis
Showing 5 of 8 shared publications
- Metformin Promotes Anti-tumor Biomarkers in Human Endometrial Cancer Cells
- Co-morbidity of type 1 diabetes and endometriosis: bringing a new paradigm into focus
- A Pilot Study on the Co-existence of Diabetes and Endometriosis in Reproductive-Age Women: Potential for Endometriosis Progression
- Co-Morbidity of Type 1 Diabetes Promotes Endometriosis Status-A Pilot Study
- Malic Enzyme 1 (ME1) Promotes Adiposity and Hepatic Steatosis and Induces Circulating Insulin and Leptin in Obese Female Mice
- Metformin Promotes Anti-tumor Biomarkers in Human Endometrial Cancer Cells
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
- Metformin Promotes Anti-tumor Biomarkers in Human Endometrial Cancer Cells
- Co-morbidity of type 1 diabetes and endometriosis: bringing a new paradigm into focus
- Metformin Promotes Anti-tumor Biomarkers in Human Endometrial Cancer Cells
- Co-morbidity of type 1 diabetes and endometriosis: bringing a new paradigm into focus
- Metformin Promotes Anti-tumor Biomarkers in Human Endometrial Cancer Cells
- Co-morbidity of type 1 diabetes and endometriosis: bringing a new paradigm into focus
- Malic Enzyme 1 (ME1) Promotes Adiposity and Hepatic Steatosis and Induces Circulating Insulin and Leptin in Obese Female Mice
- Metformin Promotes Anti-tumor Biomarkers in Human Endometrial Cancer Cells
- Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10
- SUN-126 Distinct Molecular Phenotypes of Non-Diseased Breast Adipose Tissue of Pre-Menopausal Obese and Non-Obese Women May Underlie Differing Breast Cancer Risks
- Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10
- SUN-126 Distinct Molecular Phenotypes of Non-Diseased Breast Adipose Tissue of Pre-Menopausal Obese and Non-Obese Women May Underlie Differing Breast Cancer Risks
- Breast adipose regulation of premenopausal breast epithelial phenotype involves interleukin 10
- SUN-126 Distinct Molecular Phenotypes of Non-Diseased Breast Adipose Tissue of Pre-Menopausal Obese and Non-Obese Women May Underlie Differing Breast Cancer Risks
- A Pilot Study on the Co-existence of Diabetes and Endometriosis in Reproductive-Age Women: Potential for Endometriosis Progression
- Co-Morbidity of Type 1 Diabetes Promotes Endometriosis Status-A Pilot Study
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
- Lesion Genotype Modifies High-Fat Diet Effects on Endometriosis Development in Mice
- Krüppel-like Factor 9 (KLF9) Suppresses Hepatocellular Carcinoma (HCC)-Promoting Oxidative Stress and Inflammation in Mice Fed High-Fat Diet
- Lesion Genotype Modifies High-Fat Diet Effects on Endometriosis Development in Mice
- Prior Statin vs In-Hospital Statin Usage in Severe COVID-19: Review and Meta-Analysis
- 26-CCC-10096-ACC CHEMOTHERAPY-INDUCED CARDIOTOXICITY VS LEFT BUNDLE BRANCH BLOCK CARDIOMYOPATHY: A DIAGNOSTIC AND MANAGEMENT CHALLENGE
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