Match tier
Listed
Presence
Current · Arkansas
Last published
2025
Sources
OpenAlex · ORCID
Refreshed
2026-10-08
J
Jeffrey M. Sifford
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Also affiliated: St. Jude Children's Research Hospital (2017–2020)
7 h-index
11 pubs
890 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Jeffrey M. Sifford investigates host-pathogen interactions, with a focus on viral infections and their impact on cellular processes. His research has explored the intrinsic activation of p53 as a mechanism to restrict gammaherpesvirus-driven B cell expansion during the establishment of latency. Sifford's work has been published in peer-reviewed journals, and he has contributed to 11 publications with a total of 839 citations, holding an h-index of 7. He has collaborated with researchers at the University of Arkansas for Medical Sciences, including Jason S. Stumhofer, J. Craig Forrest, Mark Manzano, and Shana M. Owens, with whom he shares multiple publications. Sifford's recent activity indicates ongoing research in his areas of expertise.
Metrics
- h-index: 7
- Publications: 11
- Citations: 890
Selected Publications
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Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment (2025)
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p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated <i>IgH/c-Myc</i> Translocations (2020)
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Conditional mutagenesis in vivo reveals cell type- and infection stage-specific requirements for LANA in chronic MHV68 infection (2018)
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Murine Gammaherpesvirus 68 LANA and SOX Homologs Counteract ATM-Driven p53 Activity during Lytic Viral Replication (2015)
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Phosphoproteomic Analyses Reveal Signaling Pathways That Facilitate Lytic Gammaherpesvirus Replication (2013)
Collaboration Network
Top Collaborators
J. Craig Forrest
Winthrop Rockefeller Foundation
5 shared publications
In database
- Phosphoproteomic Analyses Reveal Signaling Pathways That Facilitate Lytic Gammaherpesvirus Replication
- Conditional mutagenesis in vivo reveals cell type- and infection stage-specific requirements for LANA in chronic MHV68 infection
- Murine Gammaherpesvirus 68 LANA and SOX Homologs Counteract ATM-Driven p53 Activity during Lytic Viral Replication
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated <i>IgH/c-Myc</i> Translocations
Eduardo Salinas
University of Arkansas for Medical Sciences
4 shared publications
In database
- Conditional mutagenesis in vivo reveals cell type- and infection stage-specific requirements for LANA in chronic MHV68 infection
- Murine Gammaherpesvirus 68 LANA and SOX Homologs Counteract ATM-Driven p53 Activity during Lytic Viral Replication
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated <i>IgH/c-Myc</i> Translocations
James A. Stahl
University of Arkansas for Medical Sciences (US)
2 shared publications
- Phosphoproteomic Analyses Reveal Signaling Pathways That Facilitate Lytic Gammaherpesvirus Replication
- Murine Gammaherpesvirus 68 LANA and SOX Homologs Counteract ATM-Driven p53 Activity during Lytic Viral Replication
Darby G. Oldenburg
Gundersen Health System (US)
2 shared publications
- Conditional mutagenesis in vivo reveals cell type- and infection stage-specific requirements for LANA in chronic MHV68 infection
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
Shana M. Owens
University of Arkansas for Medical Sciences
2 shared publications
In database
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated <i>IgH/c-Myc</i> Translocations
Debopam Ghosh
University of Arkansas for Medical Sciences
2 shared publications
In database
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated <i>IgH/c-Myc</i> Translocations
Jason S. Stumhofer
University of Arkansas for Medical Sciences
2 shared publications
In database
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated <i>IgH/c-Myc</i> Translocations
Shweta S. Chavan
University of Arkansas for Medical Sciences (US)
1 shared publication
- Phosphoproteomic Analyses Reveal Signaling Pathways That Facilitate Lytic Gammaherpesvirus Replication
Veronica MacLeod
University of Arkansas for Medical Sciences (US)
1 shared publication
- Phosphoproteomic Analyses Reveal Signaling Pathways That Facilitate Lytic Gammaherpesvirus Replication
Daniel E. Voth
University of Arkansas for Medical Sciences
1 shared publication
In database
- Phosphoproteomic Analyses Reveal Signaling Pathways That Facilitate Lytic Gammaherpesvirus Replication
Ricky D. Edmondson
University of Arkansas for Medical Sciences
1 shared publication
In database
- Phosphoproteomic Analyses Reveal Signaling Pathways That Facilitate Lytic Gammaherpesvirus Replication
Arundhati Gupta
University of Arkansas for Medical Sciences (US)
1 shared publication
- Conditional mutagenesis in vivo reveals cell type- and infection stage-specific requirements for LANA in chronic MHV68 infection
Douglas W. White
Gundersen Health System (US)
1 shared publication
- Conditional mutagenesis in vivo reveals cell type- and infection stage-specific requirements for LANA in chronic MHV68 infection
Gang Li
University of Arkansas for Medical Sciences (US)
1 shared publication
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated <i>IgH/c-Myc</i> Translocations
Andrew D. Miller
Cornell University (US)
1 shared publication
- p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated <i>IgH/c-Myc</i> Translocations
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