Zijing Zhang
Postdoctoral Fellow
Also affiliated: Providence College (2020); Cornell University (2017–2018); Brown University (2019); MEP Equine Solutions (United States) (2014); First Affiliated Hospital of Guangzhou University of Chinese Medicine (2024); Winthrop Rockefeller Foundation (2024–2025); Women & Infants Hospital of Rhode Island (2019–2020)
Postdoc Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Zijing Zhang's research focuses on understanding cellular and molecular mechanisms underlying disease and aging. Zhang has investigated the role of metabolically active neutrophils in supporting Mycobacterium tuberculosis infection and explored how β-glucan-induced macrophages confer protection against this bacterium. Their work also examines age-associated changes in ovarian myeloid cells, specifically macrophages and their signaling dynamics, using single-cell analysis techniques. Additionally, Zhang has studied the impact of SARS-CoV-2 infection on disease severity in mouse models of Down syndrome and analyzed the molecular characteristics and immune microenvironment of gastrointestinal stromal tumors to identify therapeutic targets. Zhang has 12 publications with 326 citations and an h-index of 7, and collaborates with researchers at the University of Arkansas for Medical Sciences, including Lu Huang, Lin‐Xi Li, J. Tucker Andrews, and Ananya Ranaraja.
Metrics
- h-index: 7
- Publications: 12
- Citations: 333
Selected Publications
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β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395 (2025)
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Single-cell analysis of ovarian myeloid cells identifies age-associated changes in macrophages and signaling dynamics (2025)
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Single-cell analysis of ovarian myeloid cells identifies aging associated changes in macrophages and signaling dynamics (2024)
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Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis (2024)
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SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome (2024)
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Macrophages: an indispensable piece of ovarian health (2020)
Collaboration Network
Top Collaborators
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Single-cell analysis of ovarian myeloid cells identifies age-associated changes in macrophages and signaling dynamics
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Single-cell analysis of ovarian myeloid cells identifies age-associated changes in macrophages and signaling dynamics
- Single-cell analysis of ovarian myeloid cells identifies aging associated changes in macrophages and signaling dynamics
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
- Single-cell analysis of ovarian myeloid cells identifies age-associated changes in macrophages and signaling dynamics
- Single-cell analysis of ovarian myeloid cells identifies aging associated changes in macrophages and signaling dynamics
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
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