Biography and Research Information
OverviewAI-generated summary
Zachary Waldrip's research focuses on the application of medical imaging techniques, specifically lung ultrasound, to quantify regional lung strain in mechanically ventilated subjects. His recent work has investigated this approach in pigs, contributing to the understanding of lung mechanics under artificial ventilation. Waldrip has also explored feature tracking within ultrasound images to derive quantitative measures of lung deformation.
Waldrip is affiliated with the University of Arkansas for Medical Sciences. His scholarly output includes 23 publications with 171 citations and an h-index of 5. He has collaborated with researchers from both the University of Arkansas at Fayetteville and within his own institution, including R.A. Walters, Haley Scheen, and Cassidy Beam.
Metrics
- Publications: 1
Positions
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Instructor 2022–presentUniversity of Arkansas for Medical Sciences Surgery ORCID
Selected Publications
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Lung ultrasound feature tracking to quantify regional lung strain in mechanically ventilated pigs (2026)
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Lung Ultrasound Feature Tracking to Quantify Regional Lung Strain in Mechanically Ventilated Pigs (2026)
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A step-by-step protocol for isolation and flow cytometry analysis of porcine cardiac valve leaflets (2025)
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A functional surface display system in Saccharomyces boulardii for protein absorption in simulated intestinal fluids (2025)
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DNA-PKcs Controls the Cytotoxic T-Cell Response to Cancer and Transplant Allograft Through Regulating LAT-Dependent Signaling (2025)
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DNA-PKcs governs LAT-dependent signaling in CD4 + and CD8 + T cells (2025)
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Intra-peritoneal Povidone-iodine Irrigation Decreases Abscesses in a Perforated Appendicitis Murine Model (2024)
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Discovery of the DNA-PKcs inhibitor DA-143 which exhibits enhanced solubility relative to NU7441 (2024)
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Chemical inhibition of DNA‐PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells (2023)
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DNA-PKcs is required for the activation and cytotoxicity of CD8+ effector T cells (2022)
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Inhibition of DNA-PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells (2022)
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DNA-PKcs Inhibition Extends Allogeneic Skin Graft Survival (2021)
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DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells (2021)
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DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells (2021)
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10399 DNA-PK(cs) Regulates Stability of Egr1 During T Cell Activation (2021)
Collaboration Network
Top Collaborators
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
- DNA-PKcs Inhibition Extends Allogeneic Skin Graft Survival
- Chemical inhibition of DNA‐PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
- Discovery of the DNA-PKcs inhibitor DA-143 which exhibits enhanced solubility relative to NU7441
- Intra-peritoneal Povidone-iodine Irrigation Decreases Abscesses in a Perforated Appendicitis Murine Model
Showing 5 of 16 shared publications
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
- DNA-PKcs Inhibition Extends Allogeneic Skin Graft Survival
- Chemical inhibition of DNA‐PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
- Discovery of the DNA-PKcs inhibitor DA-143 which exhibits enhanced solubility relative to NU7441
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
Showing 5 of 14 shared publications
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
- Chemical inhibition of DNA‐PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
- Discovery of the DNA-PKcs inhibitor DA-143 which exhibits enhanced solubility relative to NU7441
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
- Inhibition of DNA-PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
Showing 5 of 8 shared publications
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
- DNA-PKcs Inhibition Extends Allogeneic Skin Graft Survival
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
- 10399 DNA-PK(cs) Regulates Stability of Egr1 During T Cell Activation
- DNA-PKcs Inhibition Extends Allogeneic Skin Graft Survival and Suppresses EGR1 and NFκB-mediated IL6 Production
Showing 5 of 6 shared publications
- A CRISPR-based approach for proteomic analysis of a single genomic locus
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
- DNA-PKcs governs LAT-dependent signaling in CD4 + and CD8 + T cells
- DNA-PKcs Controls the Cytotoxic T-Cell Response to Cancer and Transplant Allograft Through Regulating LAT-Dependent Signaling
- A CRISPR-based approach for proteomic analysis of a single genomic locus
- DEAD-box RNA helicases Dbp2, Ded1 and Mss116 bind to G-quadruplex nucleic acids and destabilize G-quadruplex RNA
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
- DNA-PKcs kinase activity stabilizes the transcription factor Egr1 in activated immune cells
- Chemical inhibition of DNA‐PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
- Intra-peritoneal Povidone-iodine Irrigation Decreases Abscesses in a Perforated Appendicitis Murine Model
- Inhibition of DNA-PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
- DNA-PKcs is required for the activation and cytotoxicity of CD8+ effector T cells
- Chemical inhibition of DNA‐PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
- Inhibition of DNA-PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
- DNA-PKcs governs LAT-dependent signaling in CD4 + and CD8 + T cells
- DNA-PKcs Controls the Cytotoxic T-Cell Response to Cancer and Transplant Allograft Through Regulating LAT-Dependent Signaling
- Chemical inhibition of DNA‐PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
- Discovery of the DNA-PKcs inhibitor DA-143 which exhibits enhanced solubility relative to NU7441
- A functional surface display system in Saccharomyces boulardii for protein absorption in simulated intestinal fluids
- DNA-PKcs governs LAT-dependent signaling in CD4 + and CD8 + T cells
- A CRISPR-based approach for proteomic analysis of a single genomic locus
- DEAD-box RNA helicases Dbp2, Ded1 and Mss116 bind to G-quadruplex nucleic acids and destabilize G-quadruplex RNA
- Genome-wide Cas9 binding specificity in Saccharomyces cerevisiae
- Chemical inhibition of DNA‐PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
- Inhibition of DNA-PKcs impairs the activation and cytotoxicity of CD4 + helper and CD8 + effector T cells
- DNA-PKcs is required for the activation and cytotoxicity of CD8+ effector T cells
- A CRISPR-based approach for proteomic analysis of a single genomic locus
- DEAD-box RNA helicases Dbp2, Ded1 and Mss116 bind to G-quadruplex nucleic acids and destabilize G-quadruplex RNA
- A CRISPR-based approach for proteomic analysis of a single genomic locus
- DEAD-box RNA helicases Dbp2, Ded1 and Mss116 bind to G-quadruplex nucleic acids and destabilize G-quadruplex RNA
- A CRISPR-based approach for proteomic analysis of a single genomic locus
- Genome-wide Cas9 binding specificity in Saccharomyces cerevisiae
- A CRISPR-based approach for proteomic analysis of a single genomic locus
- Genome-wide Cas9 binding specificity in Saccharomyces cerevisiae
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