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OverviewAI-generated summary
Filipa Ponte's research focuses on the molecular mechanisms underlying bone loss, particularly in the context of aging and estrogen deficiency. Her work investigates the role of specific cellular components and signaling pathways in maintaining bone homeostasis. Ponte has studied the contribution of mitochondrial Sirt3 to age-related bone loss and the impact of decreased NAD+ levels on osteoprogenitor cells and bone mass. She has also explored the function of RANK ligand in regulating osteoclast gene expression and the effect of Mmp13 deletion in mesenchymal cells on bone mass and cortical bone loss in mice. Ponte has published 27 papers, with a total of 420 citations and an h-index of 9. She has collaborated with researchers at the University of Arkansas for Medical Sciences, including Maria Almeida, Stavros C. Manolagas, Aaron Warren, and Ha‐Neui Kim.
Metrics
- h-index: 9
- Publications: 27
- Citations: 433
Selected Publications
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RANK ligand converts the NCoR/HDAC3 co-repressor to a PGC1β- and RNA-dependent co-activator of osteoclast gene expression (2023)
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Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency (2022)
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Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency (2022)
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<i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice (2021)
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A decrease in NAD+ contributes to the loss of osteoprogenitors and bone mass with aging (2021)
Collaboration Network
Top Collaborators
Maria Almeida
Central Arkansas Veterans Healthcare System
6 shared publications
In database
- Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency
- A decrease in NAD+ contributes to the loss of osteoprogenitors and bone mass with aging
- RANK ligand converts the NCoR/HDAC3 co-repressor to a PGC1β- and RNA-dependent co-activator of osteoclast gene expression
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
Showing 5 of 6 shared publications
Ha‐Neui Kim
University of Arkansas for Medical Sciences
5 shared publications
In database
- Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency
- A decrease in NAD+ contributes to the loss of osteoprogenitors and bone mass with aging
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Aaron Warren
University of Arkansas for Medical Sciences
5 shared publications
In database
- Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency
- A decrease in NAD+ contributes to the loss of osteoprogenitors and bone mass with aging
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Stavros C. Manolagas
University of Arkansas for Medical Sciences
5 shared publications
In database
- Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency
- RANK ligand converts the NCoR/HDAC3 co-repressor to a PGC1β- and RNA-dependent co-activator of osteoclast gene expression
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Srividhya Iyer
University of Arkansas for Medical Sciences (US)
4 shared publications
- A decrease in NAD+ contributes to the loss of osteoprogenitors and bone mass with aging
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Erin M. Mannen
University of Arkansas for Medical Sciences (US)
3 shared publications
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Horacio Gómez-Acevedo
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Intawat Nookaew
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Li Han
University of Arkansas for Medical Sciences (US)
2 shared publications
- A decrease in NAD+ contributes to the loss of osteoprogenitors and bone mass with aging
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
Rebecca Ring
University of Arkansas for Medical Sciences
1 shared publication
In database
- A decrease in NAD+ contributes to the loss of osteoprogenitors and bone mass with aging
Wen Ling
1 shared publication
In database
- Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency
Kimberly J. Krager
University of Arkansas for Medical Sciences
1 shared publication
In database
- Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency
Kimberly Richardson
1 shared publication
- Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency
Nükhet Aykin‐Burns
University of Arkansas for Medical Sciences
1 shared publication
In database
- Mitochondrial Sirt3 contributes to the bone loss caused by aging or estrogen deficiency
Han Li
University of Arkansas for Medical Sciences (US)
1 shared publication
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
Similar Researchers
Based on overlapping research topics
Stavros C. Manolagas
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Estrogen and related hormone effects
Mitochondrial Function and Pathology
Ilham Kadhim
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Mitochondrial Function and Pathology
Aging and Longevity in Model Organisms
Jinhu Xiong
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Mitochondrial Function and Pathology
Aging and Longevity in Model Organisms
Maria Almeida
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Mitochondrial Function and Pathology
Aging and Longevity in Model Organisms
Jacob Kordsmeier
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Mitochondrial Function and Pathology
Bone health and treatments
Ankita Chalke
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Mitochondrial Function and Pathology
Bone health and treatments