Joseph Asante
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Researcher
Also affiliated: Institute on Aging (2024)
Unknown Researcher
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Joseph Asante's research focuses on the intersection of drug transport mechanisms and neurological disorders, particularly Alzheimer's disease. His work investigates the role of P-glycoprotein (P-gp) substrates in influencing the risk and progression of Alzheimer's and related dementias. Asante has explored how P-gp affects the transendothelial transport of amyloid-beta peptide, a key protein implicated in Alzheimer's pathology.
Further research by Asante examines the potential of specific medications, such as ezetimibe, to mitigate Alzheimer's risk by interfering with protein interactions in model systems. He also investigates the structural and functional implications of mutations in viral proteins, such as the SARS-CoV-2 spike protein, using predictive modeling. Asante has published six papers with 21 citations and holds an h-index of 3. He collaborates with researchers at the University of Arkansas for Medical Sciences, including Steven W. Barger and Corey Nagel.
Metrics
- h-index: 3
- Publications: 6
- Citations: 22
Selected Publications
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Impact of P-glycoprotein substrates on transendothelial transport of amyloid-β peptide in an in vitro model (2026)
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Correction: Structural and Functional Impacts of SARS-CoV-2 Spike Protein Mutations: Insights From Predictive Modeling and Analytics (2025)
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Impact of P-Glycoprotein Substrates on Transendothelial Transport of Amyloid-β Peptide (2025)
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Structural and Functional Impacts of SARS-CoV-2 Spike Protein Mutations: Insights From Predictive Modeling and Analytics (2025)
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Prescription-based association of P-glycoprotein substrates with Alzheimer's disease risk: A nested case-control study (2025)
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P-glycoprotein and Alzheimer’s Disease: Threats and Opportunities (2025)
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Ezetimibe Lowers Risk of Alzheimer’s and Related Dementias over Sevenfold, Reducing Aggregation in Model Systems by Inhibiting 14-3-3G::Hexokinase Interaction (2024)
Collaboration Network
Top Collaborators
- Prescription-based association of P-glycoprotein substrates with Alzheimer's disease risk: A nested case-control study
- P-glycoprotein and Alzheimer’s Disease: Threats and Opportunities
- Impact of P-Glycoprotein Substrates on Transendothelial Transport of Amyloid-β Peptide
- Ezetimibe Lowers Risk of Alzheimer’s and Related Dementias over Sevenfold, Reducing Aggregation in Model Systems by Inhibiting 14-3-3G::Hexokinase Interaction
- Prescription-based association of P-glycoprotein substrates with Alzheimer's disease risk: A nested case-control study
- Ezetimibe Lowers Risk of Alzheimer’s and Related Dementias over Sevenfold, Reducing Aggregation in Model Systems by Inhibiting 14-3-3G::Hexokinase Interaction
- Ezetimibe Lowers Risk of Alzheimer’s and Related Dementias over Sevenfold, Reducing Aggregation in Model Systems by Inhibiting 14-3-3G::Hexokinase Interaction
- Ezetimibe Lowers Risk of Alzheimer’s and Related Dementias over Sevenfold, Reducing Aggregation in Model Systems by Inhibiting 14-3-3G::Hexokinase Interaction
- Ezetimibe Lowers Risk of Alzheimer’s and Related Dementias over Sevenfold, Reducing Aggregation in Model Systems by Inhibiting 14-3-3G::Hexokinase Interaction
- Ezetimibe Lowers Risk of Alzheimer’s and Related Dementias over Sevenfold, Reducing Aggregation in Model Systems by Inhibiting 14-3-3G::Hexokinase Interaction
- Ezetimibe Lowers Risk of Alzheimer’s and Related Dementias over Sevenfold, Reducing Aggregation in Model Systems by Inhibiting 14-3-3G::Hexokinase Interaction
- Ezetimibe Lowers Risk of Alzheimer’s and Related Dementias over Sevenfold, Reducing Aggregation in Model Systems by Inhibiting 14-3-3G::Hexokinase Interaction
- Ezetimibe Lowers Risk of Alzheimer’s and Related Dementias over Sevenfold, Reducing Aggregation in Model Systems by Inhibiting 14-3-3G::Hexokinase Interaction
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