Biography and Research Information
OverviewAI-generated summary
Lindsey Hazeslip's research focuses on molecular mechanisms underlying gene expression regulation, with a particular emphasis on DNA helicases and their roles in cellular processes. Hazeslip has investigated the structural features of Dda helicase that influence its displacement of DNA-bound proteins, contributing to the understanding of enzyme function. Their work also extends to the multi-omics analysis of triple-negative breast cancer, aiming to identify correlated regulatory features associated with the disease. This research involves the integration of diverse biological data to uncover complex molecular interactions and potential therapeutic targets. Hazeslip collaborates with researchers at the University of Arkansas for Medical Sciences, including Maroof K. Zafar, Alicia K. Byrd, and Matthew D. Thompson, as well as John C. Marecki at the University of Arkansas at Fayetteville. Their scholarship metrics include an h-index of 4, with 5 total publications and 59 total citations.
Metrics
- h-index: 4
- Publications: 5
- Citations: 61
Selected Publications
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Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection (2022)
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A structural feature of Dda helicase which enhances displacement of streptavidin and <i>trp</i> repressor from <scp>DNA</scp> (2021)
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Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer (2021)
Collaboration Network
Top Collaborators
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- A structural feature of Dda helicase which enhances displacement of streptavidin and <i>trp</i> repressor from <scp>DNA</scp>
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- A structural feature of Dda helicase which enhances displacement of streptavidin and <i>trp</i> repressor from <scp>DNA</scp>
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- A structural feature of Dda helicase which enhances displacement of streptavidin and <i>trp</i> repressor from <scp>DNA</scp>
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- Multi-omics data integration reveals correlated regulatory features of triple negative breast cancer
- A structural feature of Dda helicase which enhances displacement of streptavidin and <i>trp</i> repressor from <scp>DNA</scp>
- A structural feature of Dda helicase which enhances displacement of streptavidin and <i>trp</i> repressor from <scp>DNA</scp>
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