Match tier
Listed
Presence
Current · Arkansas
Last published
2024
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
Biography and Research Information
OverviewAI-generated summary
Regina Lichti Binz investigates cytogenetic and epigenetic alterations in human and murine cells. Her research includes studying the effects of dietary methionine deficiency on genetic instability in immune cells and evaluating the impact of simulated galactic cosmic rays on cognitive performance in mice. Binz has also examined cytogenetic and epigenetic aberrations in peripheral lymphocytes of the Marshallese population in northwest Arkansas. She has published protocols for chromosome preparation and staining for cytogenetic analysis, and her work has utilized molecular cytogenetics to identify mosaicism and novel translocations in human endothelial cells. Binz collaborates with several researchers at the University of Arkansas for Medical Sciences, including Rupak Pathak, Igor Koturbash, Martin Hauer‐Jensen, and Laura Ewing, with whom she shares multiple publications. Her work has a h-index of 11 with 451 total citations across 25 publications.
Metrics
- h-index: 11
- Publications: 25
- Citations: 462
Positions
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Cytogenetic Specialist 2018–presentUniversity of Arkansas for Medical Sciences Pharmaceutical Sciences, Radiation Health ORCID
Selected Publications
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G-Banding and Molecular Cytogenetics Detect Novel Translocations and Cryptic Aberrations in Human Immortal Endothelial Cells (2024)
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Protocol for preparation and staining of chromosomes isolated from mouse and human tissues for conventional and molecular cytogenetic analysis (2024)
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Evaluating the effects of low-dose simulated galactic cosmic rays on murine hippocampal-dependent cognitive performance (2022)
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Cytogenetic and epigenetic aberrations in peripheral lymphocytes of northwest Arkansas Marshallese (2022)
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Molecular Cytogenetics Reveals Mosaicism in Human Umbilical Vein Endothelial Cells (2022)
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Dietary Methionine Deficiency Enhances Genetic Instability in Murine Immune Cells (2021)
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Identification of novel breakpoints for locus- and region-specific translocations in 293 cells by molecular cytogenetics before and after irradiation (2019)
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Concurrent Amplification of MYC and 1q21 in Multiple Myeloma: Focal and Segmental Jumping Translocations of MYC (2016)
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The Vitamin E Analog Gamma-Tocotrienol (GT3) Suppresses Radiation-Induced Cytogenetic Damage (2016)
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Jumping Translocations of 1q12 in Multiple Myeloma: Unexpected Intra-clonal Heterogeneity of Copy Number Aberrations in the 1q21-31 Region (2015)
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Evidence of an Epigenetic Origin for High-Risk 1q21 Copy Number Aberrations in Multiple Myeloma (2014)
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Hyperhaploid Multiple Myeloma (MM): A Rare Karyotypic Subgroup Retaining Disomy 18 and 1q12∼23 Amplification (2012)
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Jumping Translocations 1q12 Contribute to Copy Number (CN) Alterations in Multiple Myeloma (MM): Unexpected Focal Amplifications of Receptor Chromosomes (RC) (2011)
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Evidence for a novel mechanism for gene amplification in multiple myeloma: 1q12 pericentromeric heterochromatin mediates breakage‐fusion‐bridge cycles of a 1q12∼23 amplicon (2009)
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A Novel Mechanism for Intrachromosomal Gene Amplification in Multiple Myeloma: 1q12 Pericentromeric Heterochromatin Mediates Breakage- Fusion-Bridge Cycles of the 1q12~23 Amplicon (2008)
Collaboration Network
Top Collaborators
Jeffrey R. Sawyer
University of Arkansas for Medical Sciences (US)
10 shared publications
- De novo proximal duplication of 1(q12q22) in a female infant with multiple congenital anomalies
- Evidence for a novel mechanism for gene amplification in multiple myeloma: 1q12 pericentromeric heterochromatin mediates breakage‐fusion‐bridge cycles of a 1q12∼23 amplicon
- Evidence for telomeric fusions as a mechanism for recurring structural aberrations of chromosome 11 in giant cell tumor of bone
- The Vitamin E Analog Gamma-Tocotrienol (GT3) Suppresses Radiation-Induced Cytogenetic Damage
- Multicolor spectral karyotyping of the L5178YTk+/−-3.7.2C mouse lymphoma cell line
Showing 5 of 10 shared publications
Charles M. Swanson
University of Arkansas for Medical Sciences (US)
9 shared publications
- De novo proximal duplication of 1(q12q22) in a female infant with multiple congenital anomalies
- Evidence for a novel mechanism for gene amplification in multiple myeloma: 1q12 pericentromeric heterochromatin mediates breakage‐fusion‐bridge cycles of a 1q12∼23 amplicon
- Evidence for telomeric fusions as a mechanism for recurring structural aberrations of chromosome 11 in giant cell tumor of bone
- Concurrent Amplification of MYC and 1q21 in Multiple Myeloma: Focal and Segmental Jumping Translocations of MYC
- Jumping Translocations of 1q12 in Multiple Myeloma: Unexpected Intra-clonal Heterogeneity of Copy Number Aberrations in the 1q21-31 Region
Showing 5 of 9 shared publications
Rupak Pathak
University of Arkansas for Medical Sciences
8 shared publications
In database
- Identification of novel breakpoints for locus- and region-specific translocations in 293 cells by molecular cytogenetics before and after irradiation
- The Vitamin E Analog Gamma-Tocotrienol (GT3) Suppresses Radiation-Induced Cytogenetic Damage
- Dietary Methionine Deficiency Enhances Genetic Instability in Murine Immune Cells
- Cytogenetic and epigenetic aberrations in peripheral lymphocytes of northwest Arkansas Marshallese
- Evaluating the effects of low-dose simulated galactic cosmic rays on murine hippocampal-dependent cognitive performance
Showing 5 of 8 shared publications
Erming Tian
University of Arkansas for Medical Sciences
7 shared publications
In database
- Evidence for a novel mechanism for gene amplification in multiple myeloma: 1q12 pericentromeric heterochromatin mediates breakage‐fusion‐bridge cycles of a 1q12∼23 amplicon
- Identification of novel breakpoints for locus- and region-specific translocations in 293 cells by molecular cytogenetics before and after irradiation
- Concurrent Amplification of MYC and 1q21 in Multiple Myeloma: Focal and Segmental Jumping Translocations of MYC
- Hyperhaploid Multiple Myeloma (MM): A Rare Karyotypic Subgroup Retaining Disomy 18 and 1q12∼23 Amplification
- Jumping Translocations 1q12 Contribute to Copy Number (CN) Alterations in Multiple Myeloma (MM): Unexpected Focal Amplifications of Receptor Chromosomes (RC)
Showing 5 of 7 shared publications
Gael Sammartino
University of Arkansas for Medical Sciences
7 shared publications
In database
- Evidence for a novel mechanism for gene amplification in multiple myeloma: 1q12 pericentromeric heterochromatin mediates breakage‐fusion‐bridge cycles of a 1q12∼23 amplicon
- Concurrent Amplification of MYC and 1q21 in Multiple Myeloma: Focal and Segmental Jumping Translocations of MYC
- Jumping Translocations of 1q12 in Multiple Myeloma: Unexpected Intra-clonal Heterogeneity of Copy Number Aberrations in the 1q21-31 Region
- Hyperhaploid Multiple Myeloma (MM): A Rare Karyotypic Subgroup Retaining Disomy 18 and 1q12∼23 Amplification
- Jumping Translocations 1q12 Contribute to Copy Number (CN) Alterations in Multiple Myeloma (MM): Unexpected Focal Amplifications of Receptor Chromosomes (RC)
Showing 5 of 7 shared publications
Bart Barlogie
University of Arkansas for Medical Sciences
5 shared publications
In database
- Evidence for a novel mechanism for gene amplification in multiple myeloma: 1q12 pericentromeric heterochromatin mediates breakage‐fusion‐bridge cycles of a 1q12∼23 amplicon
- Jumping Translocations of 1q12 in Multiple Myeloma: Unexpected Intra-clonal Heterogeneity of Copy Number Aberrations in the 1q21-31 Region
- Hyperhaploid Multiple Myeloma (MM): A Rare Karyotypic Subgroup Retaining Disomy 18 and 1q12∼23 Amplification
- Jumping Translocations 1q12 Contribute to Copy Number (CN) Alterations in Multiple Myeloma (MM): Unexpected Focal Amplifications of Receptor Chromosomes (RC)
- A Novel Mechanism for Intrachromosomal Gene Amplification in Multiple Myeloma: 1q12 Pericentromeric Heterochromatin Mediates Breakage- Fusion-Bridge Cycles of the 1q12~23 Amplicon
Janet L. Lukacs
University of Arkansas for Medical Sciences
5 shared publications
In database
- Concurrent Amplification of MYC and 1q21 in Multiple Myeloma: Focal and Segmental Jumping Translocations of MYC
- Jumping Translocations of 1q12 in Multiple Myeloma: Unexpected Intra-clonal Heterogeneity of Copy Number Aberrations in the 1q21-31 Region
- Hyperhaploid Multiple Myeloma (MM): A Rare Karyotypic Subgroup Retaining Disomy 18 and 1q12∼23 Amplification
- Jumping Translocations 1q12 Contribute to Copy Number (CN) Alterations in Multiple Myeloma (MM): Unexpected Focal Amplifications of Receptor Chromosomes (RC)
- Evidence of an Epigenetic Origin for High-Risk 1q21 Copy Number Aberrations in Multiple Myeloma
Frits van Rhee
University of Arkansas for Medical Sciences
5 shared publications
In database
- Concurrent Amplification of MYC and 1q21 in Multiple Myeloma: Focal and Segmental Jumping Translocations of MYC
- Jumping Translocations of 1q12 in Multiple Myeloma: Unexpected Intra-clonal Heterogeneity of Copy Number Aberrations in the 1q21-31 Region
- Hyperhaploid Multiple Myeloma (MM): A Rare Karyotypic Subgroup Retaining Disomy 18 and 1q12∼23 Amplification
- Jumping Translocations 1q12 Contribute to Copy Number (CN) Alterations in Multiple Myeloma (MM): Unexpected Focal Amplifications of Receptor Chromosomes (RC)
- Evidence of an Epigenetic Origin for High-Risk 1q21 Copy Number Aberrations in Multiple Myeloma
Edward L. Thomas
University of Arkansas for Medical Sciences (US)
4 shared publications
- Evidence for a novel mechanism for gene amplification in multiple myeloma: 1q12 pericentromeric heterochromatin mediates breakage‐fusion‐bridge cycles of a 1q12∼23 amplicon
- Hyperhaploid Multiple Myeloma (MM): A Rare Karyotypic Subgroup Retaining Disomy 18 and 1q12∼23 Amplification
- Jumping Translocations 1q12 Contribute to Copy Number (CN) Alterations in Multiple Myeloma (MM): Unexpected Focal Amplifications of Receptor Chromosomes (RC)
- A Novel Mechanism for Intrachromosomal Gene Amplification in Multiple Myeloma: 1q12 Pericentromeric Heterochromatin Mediates Breakage- Fusion-Bridge Cycles of the 1q12~23 Amplicon
Christoph Heuck
University of Arkansas for Medical Sciences (US)
4 shared publications
- Jumping Translocations of 1q12 in Multiple Myeloma: Unexpected Intra-clonal Heterogeneity of Copy Number Aberrations in the 1q21-31 Region
- Hyperhaploid Multiple Myeloma (MM): A Rare Karyotypic Subgroup Retaining Disomy 18 and 1q12∼23 Amplification
- Jumping Translocations 1q12 Contribute to Copy Number (CN) Alterations in Multiple Myeloma (MM): Unexpected Focal Amplifications of Receptor Chromosomes (RC)
- Evidence of an Epigenetic Origin for High-Risk 1q21 Copy Number Aberrations in Multiple Myeloma
Martin Hauer‐Jensen
University of Arkansas for Medical Sciences
4 shared publications
In database
- Identification of novel breakpoints for locus- and region-specific translocations in 293 cells by molecular cytogenetics before and after irradiation
- The Vitamin E Analog Gamma-Tocotrienol (GT3) Suppresses Radiation-Induced Cytogenetic Damage
- Dietary Methionine Deficiency Enhances Genetic Instability in Murine Immune Cells
- Cytogenetic and epigenetic aberrations in peripheral lymphocytes of northwest Arkansas Marshallese
Linda S. Goosen
University of Arkansas for Medical Sciences (US)
3 shared publications
- Evidence for a novel mechanism for gene amplification in multiple myeloma: 1q12 pericentromeric heterochromatin mediates breakage‐fusion‐bridge cycles of a 1q12∼23 amplicon
- Evidence for telomeric fusions as a mechanism for recurring structural aberrations of chromosome 11 in giant cell tumor of bone
- A Novel Mechanism for Intrachromosomal Gene Amplification in Multiple Myeloma: 1q12 Pericentromeric Heterochromatin Mediates Breakage- Fusion-Bridge Cycles of the 1q12~23 Amplicon
Maurizio Zangari
University of Arkansas for Medical Sciences
3 shared publications
In database
- Concurrent Amplification of MYC and 1q21 in Multiple Myeloma: Focal and Segmental Jumping Translocations of MYC
- Jumping Translocations of 1q12 in Multiple Myeloma: Unexpected Intra-clonal Heterogeneity of Copy Number Aberrations in the 1q21-31 Region
- Evidence of an Epigenetic Origin for High-Risk 1q21 Copy Number Aberrations in Multiple Myeloma
Igor Koturbash
University of Arkansas for Medical Sciences
3 shared publications
In database
- The Vitamin E Analog Gamma-Tocotrienol (GT3) Suppresses Radiation-Induced Cytogenetic Damage
- Dietary Methionine Deficiency Enhances Genetic Instability in Murine Immune Cells
- Cytogenetic and epigenetic aberrations in peripheral lymphocytes of northwest Arkansas Marshallese
Mark Koller
University of Arkansas for Medical Sciences (US)
2 shared publications
- Evidence for a novel mechanism for gene amplification in multiple myeloma: 1q12 pericentromeric heterochromatin mediates breakage‐fusion‐bridge cycles of a 1q12∼23 amplicon
- A Novel Mechanism for Intrachromosomal Gene Amplification in Multiple Myeloma: 1q12 Pericentromeric Heterochromatin Mediates Breakage- Fusion-Bridge Cycles of the 1q12~23 Amplicon
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