Lu Dai
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Assistant Professor
Also affiliated: Changchun University of Science and Technology (2025–2026); Liaoning University (2022–2024); Huaqiao University (2023); Beijing Institute of Technology (2023–2025); Roche (United States) (2021); Zhejiang Sci-Tech University (2022); The University of Texas MD Anderson Cancer Center (1993–1999); Zhejiang Normal University (2026); University of Kentucky (2014–2018); East China University of Science and Technology (2025); Medical University of South Carolina (2009); Sun Yat-sen University (2024); Guangxi Medical University (2016); China Three Gorges University (2024); Hangzhou Normal University (2017); Hohai University (2016); Zhejiang University of Science and Technology (2022); National University of Defense Technology (2000); Nanyang Technological University (2009–2013); Yangtze University (2023–2025); University of Bremen (2012); Genentech (2019–2023); Jilin University (2014–2026); Chinese Academy of Sciences (2025); Peking University (2009–2014); Fondazione Bruno Kessler (2012); Fudan University (2010); San Diego State University (1997–1998); Wenzhou Medical University (2023–2024); China Tobacco (2023); Southern University of Science and Technology (2024–2025); University Town of Shenzhen (2009–2012); Westlake University (2024); Kunming Children's Hospital (2022); Peking University Cancer Hospital (2024); Sun Yat-sen Memorial Hospital (2024); Zhongda Hospital Southeast University (2015–2017); Istituto Nazionale di Ricerca Metrologica (2012); Technical Institute of Physics and Chemistry (2025); Jiangsu Province Hospital (2025); Guangzhou Medical University Cancer Hospital (2015–2021); Guizhou Provincial People's Hospital (2023); Tumor Hospital of Guangxi Medical University (2016); Nanjing Jiangning Hospital (2026); Contemporary Amperex Technology Co., Limited. (2020); Nanjing First Hospital (2018–2023); Southwest Technology and Engineering Research Institute (China) (2022–2023); University of Houston (2021–2026); Hunan Agricultural University (2023); Southeast University (2015–2017); Nanjing Medical University (2018–2026); Guangzhou Medical University (2015–2021); University of California, Berkeley (2017–2025)
Upstream record may be merged OpenAlex, the source of these figures, lists 54 institutions in 5 countries for this author record — a pattern that usually means it combines several researchers with similar names. The totals above may include work by other people.
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Lu Dai's research focuses on virus-associated malignancies, particularly those linked to Kaposi's sarcoma-associated herpesvirus (KSHV). Dai has received significant federal funding to investigate the role of macrophage migration inhibitory factor (MIF) in KSHV-associated malignancies, with a grant from the NIH/National Institute of Dental and Craniofacial Research totaling $306,000. Further funding from the NIH/National Institute of Allergy and Infectious Diseases ($229,500) supports the development of O’PROTACs-based degraders targeting oncogenic viral proteins. Another NIH/National Institute of Dental and Craniofacial Research grant ($152,000) targets IL1RAP in virus-associated malignancies.
With a publication record of 207 articles and an h-index of 24, Dai's work has been cited 1,726 times. Recent publications explore the implications of HIF-1α in pancreatic cancer tumorigenesis and progression, the role of miRNA 17 family in cisplatin-resistant non-small cell lung cancer (NSCLC), and the inhibition of hyaluronan-CD44 interactions in ovarian carcinoma cells and malignant peripheral nerve sheath tumors. Dai collaborates with researchers at the University of Arkansas for Medical Sciences, including Steven R. Post, Shengyu Mu, Lindsey Barrett, and Samantha Kendrick, with whom they have co-authored multiple publications.
Dai leads a research group and maintains an active lab website. Their scholarly output has led to recognition as a high-impact, highly cited researcher and a principal investigator on federal grants.
Metrics
- h-index: 23
- Publications: 192
- Citations: 1,631
Positions
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Assistant Professor publications 2026University of Arkansas for Medical Sciences Institution web page
Selected Publications
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Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation (2026)
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Recent Advances in Development of Kaposi's Sarcoma‐Associated Herpesvirus‐Latency‐Associated Nuclear Antigen Targeted Therapies (2025)
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Developing new ceramide analogs against non-small cell lung cancer (NSCLC) (2024)
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Human endogenous retrovirus type K encoded Np9 oncoprotein induces DNA damage response (2024)
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Development of human endogenous retrovirus type K‐ related treatments for human diseases (2024)
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Role of lymphatic endothelium specific hyaluronan receptor 1 in virus infection and associated diseases (2024)
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Coinfections with additional oncoviruses in HPV+ individuals: Status, function and potential clinical implications (2024)
Federal Grants 3 $687,500 total
Role of Macrophage Migration Inhibitory Factor (MIF) in KSHV-associated Malignancies
Development of O’PROTACs-based degraders targeting an oncogenic viral protein
Collaboration Network
Top Collaborators
- SARS-CoV-2 proteins and anti-COVID-19 drugs induce lytic reactivation of an oncogenic virus
- SARS‐CoV‐2 infection and lytic reactivation of herpesviruses: A potential threat in the postpandemic era?
- Identification of new antiviral agents against Kaposi’s sarcoma-associated herpesvirus (KSHV) by high-throughput drug screening reveals the role of histamine-related signaling in promoting viral lytic reactivation
- Identification of new therapeutic targets and natural compounds against diffuse intrinsic pontine glioma (DIPG)
- Kaposi Sarcoma–Associated Herpesvirus and Staphylococcus aureus Coinfection in Oral Cavities of HIV-Positive Patients: A Unique Niche for Oncogenic Virus Lytic Reactivation
Showing 5 of 23 shared publications
- SARS-CoV-2 proteins and anti-COVID-19 drugs induce lytic reactivation of an oncogenic virus
- SARS‐CoV‐2 infection and lytic reactivation of herpesviruses: A potential threat in the postpandemic era?
- Identification of new antiviral agents against Kaposi’s sarcoma-associated herpesvirus (KSHV) by high-throughput drug screening reveals the role of histamine-related signaling in promoting viral lytic reactivation
- Identification of new therapeutic targets and natural compounds against diffuse intrinsic pontine glioma (DIPG)
- The Anti-COVID-19 Drug Remdesivir Promotes Oncogenic Herpesvirus Reactivation through Regulation of Intracellular Signaling Pathways
Showing 5 of 14 shared publications
- SARS-CoV-2 proteins and anti-COVID-19 drugs induce lytic reactivation of an oncogenic virus
- SARS‐CoV‐2 infection and lytic reactivation of herpesviruses: A potential threat in the postpandemic era?
- Identification of new antiviral agents against Kaposi’s sarcoma-associated herpesvirus (KSHV) by high-throughput drug screening reveals the role of histamine-related signaling in promoting viral lytic reactivation
- Kaposi Sarcoma–Associated Herpesvirus and Staphylococcus aureus Coinfection in Oral Cavities of HIV-Positive Patients: A Unique Niche for Oncogenic Virus Lytic Reactivation
- The Anti-COVID-19 Drug Remdesivir Promotes Oncogenic Herpesvirus Reactivation through Regulation of Intracellular Signaling Pathways
Showing 5 of 12 shared publications
- Identification of new therapeutic targets and natural compounds against diffuse intrinsic pontine glioma (DIPG)
- Role of EIF4G1 network in non‐small cell lung cancers (NSCLC) cell survival and disease progression
- Identification of natural compounds tubercidin and lycorine HCl against small‐cell lung cancer and BCAT1 as a therapeutic target
- Oncolytic strategy using new bifunctional HDACs/BRD4 inhibitors against virus-associated lymphomas
- Developing new ceramide analogs and identifying novel sphingolipid-controlled genes against a virus-associated lymphoma
Showing 5 of 8 shared publications
- SARS-CoV-2 proteins and anti-COVID-19 drugs induce lytic reactivation of an oncogenic virus
- Identification of new therapeutic targets and natural compounds against diffuse intrinsic pontine glioma (DIPG)
- Kaposi's sarcoma‐associated herpesvirus and extracellular vesicles
- Identification of natural compounds tubercidin and lycorine HCl against small‐cell lung cancer and BCAT1 as a therapeutic target
- SARS-CoV-2 proteins and anti-COVID-19 drugs induce lytic reactivation of an oncogenic virus
Showing 5 of 7 shared publications
- SARS-CoV-2 proteins and anti-COVID-19 drugs induce lytic reactivation of an oncogenic virus
- <i>Porphyromonas gingivalis</i> coinfects with KSHV in oral cavities of HIV+ patients and induces viral lytic reactivation
- Role of Interleukin-1 Family Members and Signaling Pathways in KSHV Pathogenesis
- Echinomycin as a promising therapeutic agent against KSHV-related malignancies
- Activation of IL1 signaling molecules by Kaposi’s sarcoma-associated herpesvirus
Showing 5 of 6 shared publications
- Identification of natural compounds tubercidin and lycorine HCl against small‐cell lung cancer and BCAT1 as a therapeutic target
- Echinomycin as a promising therapeutic agent against KSHV-related malignancies
- Targeting Sphingosine Kinase by ABC294640 against Diffuse Intrinsic Pontine Glioma (DIPG)
- Oral Shedding of an Oncogenic Virus Alters the Oral Microbiome in HIV+ Patients
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
- Role of EIF4G1 network in non‐small cell lung cancers (NSCLC) cell survival and disease progression
- Role of Interleukin-1 Family Members and Signaling Pathways in KSHV Pathogenesis
- Developing new ceramide analogs and identifying novel sphingolipid-controlled genes against a virus-associated lymphoma
- SARS-CoV-2 proteins and anti-COVID-19 drugs induce lytic reactivation of an oncogenic virus
- Echinomycin as a promising therapeutic agent against KSHV-related malignancies
- Activation of IL1 signaling molecules by Kaposi’s sarcoma-associated herpesvirus
- The Anti-COVID-19 Drug Remdesivir Promotes Oncogenic Herpesvirus Reactivation through Regulation of Intracellular Signaling Pathways
- Identification of natural compounds tubercidin and lycorine HCl against small‐cell lung cancer and BCAT1 as a therapeutic target
- Oncolytic strategy using new bifunctional HDACs/BRD4 inhibitors against virus-associated lymphomas
- SARS‐CoV‐2 infection and lytic reactivation of herpesviruses: A potential threat in the postpandemic era?
- Echinomycin as a promising therapeutic agent against KSHV-related malignancies
- Activation of IL1 signaling molecules by Kaposi’s sarcoma-associated herpesvirus
- Human endogenous retrovirus type K encoded Np9 oncoprotein induces DNA damage response
- Development of human endogenous retrovirus type K‐ related treatments for human diseases
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
- Identification of new antiviral agents against Kaposi’s sarcoma-associated herpesvirus (KSHV) by high-throughput drug screening reveals the role of histamine-related signaling in promoting viral lytic reactivation
- Kaposi Sarcoma–Associated Herpesvirus and Staphylococcus aureus Coinfection in Oral Cavities of HIV-Positive Patients: A Unique Niche for Oncogenic Virus Lytic Reactivation
- Kaposi Sarcoma–Associated Herpesvirus and Staphylococcus aureus Coinfection in Oral Cavities of HIV-Positive Patients: A Unique Niche for Oncogenic Virus Lytic Reactivation
- Role of EIF4G1 network in non‐small cell lung cancers (NSCLC) cell survival and disease progression
- Identification of new antiviral agents against Kaposi’s sarcoma-associated herpesvirus (KSHV) by high-throughput drug screening reveals the role of histamine-related signaling in promoting viral lytic reactivation
- Oral Shedding of an Oncogenic Virus Alters the Oral Microbiome in HIV+ Patients
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