Match tier
Likely match
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
S
Stavroula Tsitkanou
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Post Doctoral Research Fellow
Also affiliated: Beth Israel Deaconess Medical Center (2025–2026); Harvard University (2025–2026); Deakin University (2019–2024); National and Kapodistrian University of Athens (2016–2018)
10 h-index
41 pubs
542 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Stavroula Tsitkanou's research focuses on the physiological impacts of disease and exercise, particularly concerning muscle function and atrophy. Her work investigates the mechanisms behind cancer cachexia, examining how it affects muscle weakness and atrophy, and exploring the role of exercise as a counteracting therapeutic approach. Tsitkanou has studied the differential effects of various exercise types on muscle strength, hypertrophy, and mitochondrial stress in both healthy individuals and disease models. Her publications also address the involvement of skeletal muscle satellite cells, mitochondria, and microRNAs in the pathogenesis of conditions like Amyotrophic Lateral Sclerosis (ALS). Tsitkanou collaborates with researchers at the University of Arkansas at Fayetteville, including Tyrone A. Washington, Eleanor R. Schrems, Francielly Morena da Silva, and Ana Regina Cabrera.
Metrics
- h-index: 10
- Publications: 41
- Citations: 542
Positions
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Instructor - Research Faculty 2026–presentHarvard University Neurology ORCID
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Post Doctoral Research Fellow 2024–presentBeth Israel Deaconess Medical Center Neurology ORCID
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Post Doctoral Research Fellow publications 2022–2026University of Arkansas at Fayetteville ORCID
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Post-Doc Research Fellow 2021–2024University of Arkansas Department of Health, Human Performance and Recreation ORCID
Selected Publications
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Differential impact of cancer- and chemotherapy-induced cachexia: a comparative analysis in a preclinical model of colorectal cancer by biological sex (2026)
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Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia (2026)
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Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females (2025)
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Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer (2025)
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Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice (2025)
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Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice (2025)
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Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice (2024)
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Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice (2024)
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Exercise training induces mild skeletal muscle adaptations without altering disease progression in a TDP-43 mouse model (2024)
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Proteomic analysis of the TDP-43-associated insoluble fraction from NEFH-TDP-43 mouse brain suggests sustained stress granule formation, CLUH granule recruitment and impaired mitochondrial metabolism (2024)
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Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy (2023)
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Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia (2023)
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Mitochondria-Targeted Antioxidant SkQ1 Improves Muscle Contractility in Female C26 Tumor-Bearing Mice (2023)
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Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress (2022)
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Vastus Lateralis and Vastus Intermedius as Predictors of Quadriceps Femoris Muscle Hypertrophy after Strength Training (2022)
Collaboration Network
Top Collaborators
Nicholas P. Greene
University of Arkansas at Fayetteville
11 shared publications
In database
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Exercise Counteracts the Deleterious Effects of Cancer Cachexia
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
Showing 5 of 11 shared publications
Kevin A. Murach
University of Arkansas at Fayetteville
8 shared publications
In database
- Exercise Counteracts the Deleterious Effects of Cancer Cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
Showing 5 of 8 shared publications
Tyrone A. Washington
University of Arkansas at Fayetteville
8 shared publications
In database
- Exercise Counteracts the Deleterious Effects of Cancer Cachexia
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Showing 5 of 8 shared publications
Francielly Morena da Silva
University of Arkansas at Fayetteville
7 shared publications
In database
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
Showing 5 of 7 shared publications
Ana Regina Cabrera
University of Arkansas at Fayetteville
7 shared publications
In database
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
Showing 5 of 7 shared publications
Sabin Khadgi
University of Arkansas at Fayetteville
5 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Mitochondria-Targeted Antioxidant SkQ1 Improves Muscle Contractility in Female C26 Tumor-Bearing Mice
Seongkyun Lim
University of Arkansas at Fayetteville
5 shared publications
In database
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Mitochondria-Targeted Antioxidant SkQ1 Improves Muscle Contractility in Female C26 Tumor-Bearing Mice
Eleanor R. Schrems
University of Arkansas at Fayetteville
5 shared publications
In database
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
Ruqaiza Muhyudin
University of Arkansas at Fayetteville
5 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
Luca J. Delfinis
York University (CA)
4 shared publications
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer
- Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice
Calvin Peterson
University of Arkansas at Fayetteville
4 shared publications
In database
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
Shivam Gandhi
York University (CA)
3 shared publications
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer
- Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice
Madison C. Garibotti
York University (CA)
3 shared publications
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer
- Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice
Megan E. Rosa‐Caldwell
Beth Israel Deaconess Medical Center
3 shared publications
In database
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
Arthur J. Cheng
York University (CA)
3 shared publications
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Mitochondrial-targeted plastoquinone therapy prevents early onset muscle weakness that occurs before atrophy during ovarian cancer
- Mitochondrial-targeted plastoquinone therapy ameliorates early onset muscle weakness that precedes ovarian cancer cachexia in mice
Similar Researchers
Based on overlapping research topics
Megan E. Rosa‐Caldwell
University of Arkansas at Fayetteville
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