Match tier
Likely match
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
E
Eleanor R. Schrems
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Assistant Professor
10 h-index
50 pubs
229 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Eleanor R. Schrems investigates the mechanisms underlying cancer cachexia, a wasting syndrome characterized by muscle loss and metabolic dysfunction. Her research employs animal models, specifically focusing on Lewis Lung carcinoma in mice, to examine the temporal development of cachexia and its impact on skeletal muscle. Schrems' work has identified sex-dependent differences in muscle quality and transcriptional disruptions during cachexia onset. She has explored the role of specific molecules, such as microRNA-16 and the mitochondrial antioxidant SkQ1, in modulating muscle wasting and contractility in the context of cancer. Her publications also address the development of skeletal muscle fibrosis and the overexpression of PGC-1α in muscle regeneration.
Schrems' scholarship metrics include an h-index of 10, with 39 total publications and 224 total citations. She frequently collaborates with researchers at the University of Arkansas at Fayetteville, including Tyrone A. Washington (36 shared publications), Francielly Morena da Silva (28 shared publications), Ana Regina Cabrera (27 shared publications), and Nicholas P. Greene (20 shared publications).
Metrics
- h-index: 10
- Publications: 50
- Citations: 229
Positions
-
Assistant Professor 2026–presentAlice L. Walton School of Medicine Medical Education ORCID
-
Assistant Professor publications 2021–2026University of Arkansas at Fayetteville ORCID
Selected Publications
-
Circulating granulocyte colony‐stimulating factor as a sex‐unbiased global marker of lung and colorectal cancer cachexia pre‐clinical models (2026)
-
Differential impact of cancer- and chemotherapy-induced cachexia: a comparative analysis in a preclinical model of colorectal cancer by biological sex (2026)
-
Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia (2026)
-
Combined High-Intensity Exercise and Autologous Muscle Grafting Differentially Modulate Myogenesis and Extracellular Matrix Remodeling by Sex in VML-Injured Muscle (2026)
-
Exercise Intensities Modulates Inflammatory Gene Expression to Minced Muscle Repair Following Volumetric Muscle Loss (2026)
-
Evaluation of Biological Sex in the Pathophysiology of Recovery from Volumetric Muscle Loss with Integrated Regenerative and Rehabilitative Therapies (2025)Journal of the Arkansas Academy of Science OpenAlex
-
Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia (2025)
-
Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity (2025)
-
Aerobic Exercise Training Does Not Attenuate Fibrosis In Autologous Repaired Vml-injured Skeletal Muscle (2025)
-
Skeletal Muscle Damage and Inflammation (2025)
-
Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females (2025)
-
Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice (2025)
-
Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice (2025)
-
Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex (2024)
-
Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice (2024)
Collaboration Network
Top Collaborators
Tyrone A. Washington
University of Arkansas at Fayetteville
30 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
Showing 5 of 30 shared publications
Nicholas P. Greene
University of Arkansas at Fayetteville
28 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
Showing 5 of 28 shared publications
Ana Regina Cabrera
University of Arkansas at Fayetteville
26 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
Showing 5 of 26 shared publications
Francielly Morena da Silva
University of Arkansas at Fayetteville
22 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
Showing 5 of 22 shared publications
Megan E. Rosa‐Caldwell
University of Arkansas at Fayetteville
18 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
Showing 5 of 18 shared publications
Kevin A. Murach
University of Arkansas at Fayetteville
17 shared publications
In database
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
Showing 5 of 17 shared publications
Stavroula Tsitkanou
University of Arkansas at Fayetteville
14 shared publications
In database
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Showing 5 of 14 shared publications
Seongkyun Lim
University of Arkansas at Fayetteville
13 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
Showing 5 of 13 shared publications
Ruqaiza Muhyudin
University of Arkansas at Fayetteville
7 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
Showing 5 of 7 shared publications
J. William Deaver
Vanderbilt University (US)
6 shared publications
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- The effect of diet-induced obesity on extracellular matrix remodeling during skeletal muscle regeneration
- Males, But Not Females, Demonstrate Mitochondrial Dysfunction In The C26 Model Of Cancer Cachexia
Showing 5 of 6 shared publications
Landen W. Saling
University of Arkansas at Fayetteville
6 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Differential Induction Of Regulators Of Protein Turnover During C26-induced Cancer Cachexia Between Biological Sexes
Showing 5 of 6 shared publications
Sepideh Shakeri
University of Arkansas at Fayetteville
6 shared publications
In database
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Aerobic Exercise Training Does Not Attenuate Fibrosis In Autologous Repaired Vml-injured Skeletal Muscle
- Exercise Intensities Modulates Inflammatory Gene Expression to Minced Muscle Repair Following Volumetric Muscle Loss
- Combined High-Intensity Exercise and Autologous Muscle Grafting Differentially Modulate Myogenesis and Extracellular Matrix Remodeling by Sex in VML-Injured Muscle
Showing 5 of 6 shared publications
Richard Perry
University of Arkansas at Fayetteville (US)
5 shared publications
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- The effect of diet-induced obesity on extracellular matrix remodeling during skeletal muscle regeneration
- Effects of PGC-1α overexpression on the myogenic response during skeletal muscle regeneration
- Leucine Supplementation Exacerbates Morbidity in Male but Not Female Mice with Colorectal Cancer-Induced Cachexia
- Biological Sex Differences of Fibrosis During the Development of Cancer Cachexia
Wesley S. Haynie
University of Arkansas at Fayetteville
5 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Effects of PGC-1α overexpression on the myogenic response during skeletal muscle regeneration
- Leucine Supplementation Exacerbates Morbidity in Male but Not Female Mice with Colorectal Cancer-Induced Cachexia
- Biological Sex Differences of Fibrosis During the Development of Cancer Cachexia
Ronald G. Jones
University of Arkansas at Fayetteville
5 shared publications
In database
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity
Similar Researchers
Based on overlapping research topics
Ruqaiza Muhyudin
University of Arkansas at Fayetteville
Cancer-related molecular mechanisms research
Disease Models, Animal
Muscle Physiology and Disorders
Landen W. Saling
University of Arkansas at Fayetteville
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders
Metabolism and Genetic Disorders
Megan E. Rosa‐Caldwell
University of Arkansas at Fayetteville
Cancer-related molecular mechanisms research
Disease Models, Animal
Muscle Physiology and Disorders
Judit K. Megyesi
University of Arkansas for Medical Sciences
Disease Models, Animal
Muscle Physiology and Disorders
Metabolism and Genetic Disorders
Ana Regina Cabrera
University of Arkansas at Fayetteville
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders
Metabolism and Genetic Disorders
Sydney Hilgenbrink
University of Arkansas at Fayetteville
Cancer-related molecular mechanisms research
Disease Models, Animal
Muscle Physiology and Disorders