Melody L. Allensworth‐James
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Research Areas
Biomedical Subjects
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Biography and Research Information
OverviewAI-generated summary
Melody Allensworth‐James studies the role of leptin as a metabolic signal in the differentiation of pituitary cells. Her research has investigated how maternal undernutrition can lead to transcript changes in male offspring that may promote resistance to high-fat diet-induced weight gain. Allensworth‐James has published 23 papers, with a total of 329 citations, and holds an h-index of 10. She has collaborated with Gwen V. Childs, Angela K. Odle, Melanie C MacNicol, and Angus M. MacNicol, all from the University of Arkansas for Medical Sciences, with whom she shares two publications each.
Metrics
- h-index: 10
- Publications: 23
- Citations: 347
Selected Publications
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Maternal undernutrition results in transcript changes in male offspring that may promote resistance to high fat diet induced weight gain (2024)
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Leptin: A Metabolic Signal for the Differentiation of Pituitary Cells (2021)
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Control of the Anterior Pituitary Cell Lineage Regulator POU1F1 by the Stem Cell Determinant Musashi (2020)
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Sex differences in somatotrope response to fasting: biphasic responses in male mice (2020)
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Metabolic signalling to somatotrophs: Transcriptional and post‐transcriptional mediators (2020)
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Somatotrope Responses to Acute and Prolonged Loss of Leptin Signaling (2020)
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SAT-417 The Gonadotrope Leptin Signal Is Critical for the Early-Morning Estrus Rise in FSH in Female Mice (2019)
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OR24-3 Persistence of Progenitor Cell Markers Following the Selective Ablation of Musashi in Somatotropes (2019)
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Sex-specific changes in postnatal GH and PRL secretion in somatotrope LEPR-null mice (2018)
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The War on the Placenta: The Differing Battles of High-Fat Diet and Obesity (2018)
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Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence (2018)
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Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression (2017)
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Purified Somatotrope Populations Include Cells with Multihormonal Potential That Produce GH, TSH and Prolactin in Response to Leptin Signals (2016)
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A Sex-Dependent, Tropic Role for Leptin in the Somatotrope as a Regulator of POU1F1 and POU1F1-Dependent Hormones (2016)
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Adipocyte Versus Somatotrope Leptin: Regulation of Metabolic Functions in the Mouse (2016)
Collaboration Network
Top Collaborators
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Adipocyte Versus Pituitary Leptin in the Regulation of Pituitary Hormones: Somatotropes Develop Normally in the Absence of Circulating Leptin
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Sex Differences in Somatotrope Dependency on Leptin Receptors in Young Mice: Ablation of LEPR Causes Severe Growth Hormone Deficiency and Abdominal Obesity in Males
Showing 5 of 20 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Adipocyte Versus Pituitary Leptin in the Regulation of Pituitary Hormones: Somatotropes Develop Normally in the Absence of Circulating Leptin
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Sex Differences in Somatotrope Dependency on Leptin Receptors in Young Mice: Ablation of LEPR Causes Severe Growth Hormone Deficiency and Abdominal Obesity in Males
Showing 5 of 20 shared publications
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Adipocyte Versus Pituitary Leptin in the Regulation of Pituitary Hormones: Somatotropes Develop Normally in the Absence of Circulating Leptin
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Sex Differences in Somatotrope Dependency on Leptin Receptors in Young Mice: Ablation of LEPR Causes Severe Growth Hormone Deficiency and Abdominal Obesity in Males
- Ablation of Leptin Signaling to Somatotropes: Changes in Metabolic Factors that Cause Obesity
Showing 5 of 16 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- A Sex-Dependent, Tropic Role for Leptin in the Somatotrope as a Regulator of POU1F1 and POU1F1-Dependent Hormones
- Control of the Anterior Pituitary Cell Lineage Regulator POU1F1 by the Stem Cell Determinant Musashi
- Metabolic signalling to somatotrophs: Transcriptional and post‐transcriptional mediators
Showing 5 of 13 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- A Sex-Dependent, Tropic Role for Leptin in the Somatotrope as a Regulator of POU1F1 and POU1F1-Dependent Hormones
- Control of the Anterior Pituitary Cell Lineage Regulator POU1F1 by the Stem Cell Determinant Musashi
- Metabolic signalling to somatotrophs: Transcriptional and post‐transcriptional mediators
Showing 5 of 12 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Adipocyte Versus Pituitary Leptin in the Regulation of Pituitary Hormones: Somatotropes Develop Normally in the Absence of Circulating Leptin
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Ablation of Leptin Signaling to Somatotropes: Changes in Metabolic Factors that Cause Obesity
Showing 5 of 10 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Ablation of Leptin Signaling to Somatotropes: Changes in Metabolic Factors that Cause Obesity
- A Sex-Dependent, Tropic Role for Leptin in the Somatotrope as a Regulator of POU1F1 and POU1F1-Dependent Hormones
Showing 5 of 9 shared publications
- Metabolic signalling to somatotrophs: Transcriptional and post‐transcriptional mediators
- Sex differences in somatotrope response to fasting: biphasic responses in male mice
- Maternal undernutrition results in transcript changes in male offspring that may promote resistance to high fat diet induced weight gain
- Leptin: A Metabolic Signal for the Differentiation of Pituitary Cells
- OR24-3 Persistence of Progenitor Cell Markers Following the Selective Ablation of Musashi in Somatotropes
Showing 5 of 7 shared publications
- Control of the Anterior Pituitary Cell Lineage Regulator POU1F1 by the Stem Cell Determinant Musashi
- Sex differences in somatotrope response to fasting: biphasic responses in male mice
- Maternal undernutrition results in transcript changes in male offspring that may promote resistance to high fat diet induced weight gain
- OR24-3 Persistence of Progenitor Cell Markers Following the Selective Ablation of Musashi in Somatotropes
- SAT-417 The Gonadotrope Leptin Signal Is Critical for the Early-Morning Estrus Rise in FSH in Female Mice
Showing 5 of 6 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Purified Somatotrope Populations Include Cells with Multihormonal Potential That Produce GH, TSH and Prolactin in Response to Leptin Signals
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Ablation of Leptin Signaling to Somatotropes: Changes in Metabolic Factors that Cause Obesity
- Ghrelin Restoration of Function In Vitro in Somatotropes from Male Mice Lacking the Janus Kinase (JAK)-Binding Site of the Leptin Receptor
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Control of the Anterior Pituitary Cell Lineage Regulator POU1F1 by the Stem Cell Determinant Musashi
- Metabolic signalling to somatotrophs: Transcriptional and post‐transcriptional mediators
- Control of the Anterior Pituitary Cell Lineage Regulator POU1F1 by the Stem Cell Determinant Musashi
- Metabolic signalling to somatotrophs: Transcriptional and post‐transcriptional mediators
- Maternal undernutrition results in transcript changes in male offspring that may promote resistance to high fat diet induced weight gain
- Control of the Anterior Pituitary Cell Lineage Regulator POU1F1 by the Stem Cell Determinant Musashi
- OR24-3 Persistence of Progenitor Cell Markers Following the Selective Ablation of Musashi in Somatotropes
- Control of the Anterior Pituitary Cell Lineage Regulator POU1F1 by the Stem Cell Determinant Musashi
- OR24-3 Persistence of Progenitor Cell Markers Following the Selective Ablation of Musashi in Somatotropes
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