Sabin Khadgi Source Confirmed
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Researcher
University of Arkansas at Fayetteville
faculty
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Biography and Research Information
OverviewAI-generated summary
Sabin Khadgi's research investigates molecular and cellular adaptations in skeletal muscle, particularly in response to exercise and aging. His work has explored the role of the MYC gene in regulating muscle growth, identifying it as a key hypertrophic factor sufficient for muscle mass increase. Khadgi has also studied the molecular landscape of exercise responses over a 24-hour period and examined how aging affects satellite cell function in muscle. His recent publications also address the impact of mitochondrial function and antioxidant interventions on cancer-induced muscle wasting (cachexia) in both male and female mice, exploring potential therapeutic strategies.
Metrics
- h-index: 6
- Publications: 14
- Citations: 155
Selected Publications
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females (2025) DOI
- Satellite cells choreograph an immune cell-fibrogenic cell circuit during mechanical loading in geriatric skeletal muscle (2025) DOI
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice (2025) DOI
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice (2025) DOI
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth (2024) DOI
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice (2024) DOI
- Precision and efficacy of RNA-guided DNA integration in high-expressing muscle loci (2024) DOI
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth (2024) DOI
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise (2023) DOI
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle (2022) DOI
- Multi-transcriptome analysis following an acute skeletal muscle growth stimulus yields tools for discerning global and MYC regulatory networks (2022) DOI
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