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Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-09
I
Ilham Kadhim
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Also affiliated: University of Arkansas System (2022)
3 h-index
7 pubs
24 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Ilham Kadhim's research focuses on the molecular mechanisms underlying bone health and aging, particularly the role of autophagy in osteoblast lineage cells. Kadhim has investigated how the master autophagy regulator Tfeb influences bone mass and strength, with findings suggesting that its elevation can increase bone density and structural integrity. Further work has examined the relationship between mitochondrial oxidative stress, autophagy, and the age-related decline in bone mechanoresponsiveness, indicating that these factors alone do not fully replicate the effects of aging on bone mechanics. Kadhim's studies also explore the essentiality of autophagy in Osx1-Cre-targeted cells for bone development, growth, and maintenance. Collaborations include work with Melda Onal, A. Gordon James, and Maria Almeida at the University of Arkansas for Medical Sciences.
Metrics
- h-index: 3
- Publications: 7
- Citations: 24
Selected Publications
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Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone (2026)
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Renal hemodynamics underlie hepatorenal physiology in bile duct-ligated rats (2026)
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Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength (2025)
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Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness (2025)
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CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength (2024)
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Encapsulation of Inositol Hexakisphosphate with Chitosan via Gelation to Facilitate Cellular Delivery and Programmed Cell Death in Human Breast Cancer Cells (2024)
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Up-regulation of Osh6 boosts an anti-aging membrane trafficking pathway toward vacuoles (2022)
Collaboration Network
Top Collaborators
A. Gordon James
University of Arkansas for Medical Sciences
4 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
Melda Onal
University of Arkansas for Medical Sciences
4 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
Jacob Laster
University of Arkansas for Medical Sciences
3 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
Maria Almeida
University of Arkansas Medical Center
3 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Jinhu Xiong
University of Arkansas for Medical Sciences
3 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
James A. Hendrixson
University of Arkansas for Medical Sciences
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Julie Crawford
Institute for Musculoskeletal Health
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Jeff Thostenson
University of Arkansas for Medical Sciences
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Amy Y. Sato
University of Arkansas for Medical Sciences
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Intawat Nookaew
University of Arkansas for Medical Sciences
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
Nazneen Begum
University of Arkansas at Little Rock (US)
1 shared publication
- Up-regulation of Osh6 boosts an anti-aging membrane trafficking pathway toward vacuoles
William P. King
University of Arkansas at Little Rock (US)
1 shared publication
- Up-regulation of Osh6 boosts an anti-aging membrane trafficking pathway toward vacuoles
Licheng Xu
University of Arkansas at Little Rock (US)
1 shared publication
- Up-regulation of Osh6 boosts an anti-aging membrane trafficking pathway toward vacuoles
Fusheng Tang
University of Arkansas at Little Rock
1 shared publication
In database
- Up-regulation of Osh6 boosts an anti-aging membrane trafficking pathway toward vacuoles
Adeolu Sunday Oluremi
University of Arkansas at Little Rock
1 shared publication
In database
- Encapsulation of Inositol Hexakisphosphate with Chitosan via Gelation to Facilitate Cellular Delivery and Programmed Cell Death in Human Breast Cancer Cells
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