G. Venugopal

Research Assistant Professor

UAMS

12 h-index 80 pubs 620 cited

Research Areas

Biomedical Subjects

Links

ORCID Lab Website Research Group

OverviewAI-generated summary

G. Venugopal's research focuses on understanding disease mechanisms, particularly in the context of parasitic infections and muscle physiology. Recent work has investigated the host-pathogen interactions in Leishmania major infection, including the role of hypoxia-inducible factor signaling in macrophages and the cellular heterogeneity revealed by single-cell transcriptional analysis in infected mice. Venugopal has also studied immune responses, such as IL-10 dependent suppression of cytokines in pulmonary tuberculosis and the regulatory phenotype induced by Brugia malayi microfilariae in monocytes/macrophages. Additionally, Venugopal has explored the analysis of surface electromyography (sEMG) signals to identify features associated with muscle fatigue progression and conditions. Venugopal's scholarship metrics include an h-index of 12, with 80 total publications and 620 total citations.

Metrics

h-index: 12
Publications: 80
Citations: 620

Positions

Research Assistant Professor 2024–present

East Carolina University Brody School of Medicine, Department of Microbiology and Immunology ORCID
Research Assistant Professor

University of Arkansas for Medical Sciences ORCID

Selected Publications

Heat shock protein 72 is a druggable target during cold storage to improve graft outcome after kidney transplantation (2026)

American Journal of Physiology-Renal Physiology DOI OpenAlex
Guanylate-binding proteins balance iNOS/Arg-1 in myeloid cells during L. major infection and promote host defense to infection (2026)

mBio DOI OpenAlex
Enhanced translational activity is linked to lymphatic endothelial cell activation in cutaneous leishmaniasis 4267 (2025)

The Journal of Immunology DOI OpenAlex
Post‐ischemia and reperfusion kidney injury is mitigated in a novel complement 5 knockout rat (2025)

Physiological Reports 4 citations DOI OpenAlex
Guanylate-Binding Proteins Promote Host Defense Against Leishmania major by Balancing iNOS/Arg-1 in Myeloid Cells (2025)

bioRxiv (Cold Spring Harbor Laboratory) DOI OpenAlex
Enhanced Translational Activity Is Linked to Lymphatic Endothelial Cell Activation in Cutaneous Leishmaniasis (2025)

Lymphatic Research and Biology 1 citation DOI OpenAlex
HIF-α signaling regulates the macrophage inflammatory response during Leishmania major infection (2025)

Frontiers in Immunology 2 citations DOI OpenAlex
Cold Storage Disrupts the Proteome and Phosphoproteome Landscape in Rat Kidney Transplants (2024)

Transplantation 3 citations DOI OpenAlex
HIF-α signaling regulates the macrophage inflammatory response during Leishmania major infection (2024)

bioRxiv (Cold Spring Harbor Laboratory) DOI OpenAlex
Enhanced translational activity is linked to lymphatic endothelial cell activation in cutaneous leishmaniasis (2024)

bioRxiv (Cold Spring Harbor Laboratory) 2 citations DOI OpenAlex
Yersinia pestis and pneumonic plague: Insight into how a lethal pathogen interfaces with innate immune populations in the lung to cause severe disease (2024)

Cellular Immunology 8 citations DOI OpenAlex
Both the Infection Status and Inflammatory Microenvironment Induce Transcriptional Remodeling in Macrophages in Murine Leishmanial Lesions (2023)

Journal of Parasitology 3 citations DOI OpenAlex
In vivo transcriptional analysis of mice infected with Leishmania major unveils cellular heterogeneity and altered transcriptomic profiling at single-cell resolution (2022)

PLoS neglected tropical diseases 29 citations DOI OpenAlex