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Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-08-16
A
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Ana I. Coelho's research investigates metabolic and genetic disorders, with a focus on galactosemia. Her work includes exploring novel therapeutic approaches, such as mRNA therapy, to restore enzyme activity in zebrafish models of classic galactosemia. Coelho also studies bone health and the aging process, examining factors like mitochondrial function, oxidative stress, and autophagy in mesenchymal lineage cells of mouse bone. Her research extends to the association between adipose tissue and bone quality in rats, and the mechanical loading predictions derived from accelerometry data during walking and running.
Coelho's scholarship metrics include an h-index of 13, with 29 total publications and 942 total citations. She has collaborated with researchers at the University of Arkansas for Medical Sciences, including Aaron Warren, Maria Almeida, Ha‐Neui Kim, and Landon B. Gatrell, with whom she shares multiple publications.
Metrics
- h-index: 13
- Publications: 29
- Citations: 942
Selected Publications
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The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone (2025)
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Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells (2025)
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Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts (2025)
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The adverse effects of chemotherapy on bone mass are not prevented by senolytics (2025)
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Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells (2025)
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Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness (2025)
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Differences In Femur And Lumbar Vertebra Adaptations To Running And Swimming In Wistar Rat Models (2024)
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RANK ligand converts the NCoR/HDAC3 co-repressor to a PGC1β- and RNA-dependent co-activator of osteoclast gene expression (2023)
Collaboration Network
Top Collaborators
Maria Almeida
University of Arkansas for Medical Sciences
7 shared publications
In database
- RANK ligand converts the NCoR/HDAC3 co-repressor to a PGC1β- and RNA-dependent co-activator of osteoclast gene expression
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Showing 5 of 7 shared publications
Aaron Warren
University of Arkansas for Medical Sciences
6 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The adverse effects of chemotherapy on bone mass are not prevented by senolytics
Showing 5 of 6 shared publications
Ha‐Neui Kim
University of Arkansas for Medical Sciences
5 shared publications
In database
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
Md Mohsin Ali
University of Arkansas for Medical Sciences (US)
4 shared publications
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The adverse effects of chemotherapy on bone mass are not prevented by senolytics
Qiang Fu
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Intawat Nookaew
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Charles A. O’Brien
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
A. Gordon James
University of Arkansas for Medical Sciences
2 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
Landon Gatrell
University of Arkansas for Medical Sciences
2 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
Jinhu Xiong
University of Arkansas for Medical Sciences
2 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
Melda Onal
University of Arkansas for Medical Sciences
2 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
Ankita Chalke
University of Arkansas for Medical Sciences
2 shared publications
In database
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
Olivia Reyes‐Castro
University of Arkansas for Medical Sciences
2 shared publications
In database
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
Claudia C.S. Chini
University of Arkansas for Medical Sciences
2 shared publications
In database
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
Farah Alturkmani
University of Arkansas for Medical Sciences
2 shared publications
In database
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
Similar Researchers
Based on overlapping research topics
Qiang Fu
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
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University of Arkansas for Medical Sciences
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Kimberly J. Krager
University of Arkansas for Medical Sciences
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Aaron Warren
University of Arkansas for Medical Sciences
Metabolism and Genetic Disorders
Bone Metabolism and Diseases
Mitochondrial Function and Pathology
Nisreen Akel
University of Arkansas for Medical Sciences
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Julienne Daniel
University of Arkansas at Fayetteville
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