Match tier
Listed
Presence
Current · Arkansas
Last published
2025
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
A
Ana I. Coelho
Graduate Assistant
Also affiliated: Boston Children's Hospital (2015); Harvard University (2015); University of Lisbon (2013–2015); Universidade do Porto (2023); Maastricht University Medical Centre (2015–2022); Maastricht University (2015–2022); Centro de Investigação em Actividade Física Saúde e Lazer (2023); Manton Center for Orphan Disease Research (2015)
13 h-index
20 pubs
952 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Ana I. Coelho's research focuses on hereditary galactosemia, a group of inherited metabolic disorders affecting galactose metabolism. Her work has involved studying the pathophysiology of classic galactosemia, including an examination of animal and cellular models to identify potential treatment targets. Coelho has also investigated the functional and structural impacts of specific missense mutations associated with classic galactosemia. Her publications address current and future therapeutic strategies for the condition, as well as its natural history, drawing on data from registries. She has also contributed to research on bone metabolism, specifically the role of RANK ligand in osteoclast gene expression.
Metrics
- h-index: 13
- Publications: 20
- Citations: 952
Positions
-
Graduate Assistant 2022–presentUniversity of Arkansas for Medical Sciences Division of Endocrinology and Metabolism ORCID
Selected Publications
-
The Aging Landscape by scRNAseq of Mesenchymal Lineage Cells in Mouse Bone (2025)
-
Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells (2025)
-
Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts (2025)
-
The adverse effects of chemotherapy on bone mass are not prevented by senolytics (2025)
-
Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells (2025)
-
Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness (2025)
-
Differences In Femur And Lumbar Vertebra Adaptations To Running And Swimming In Wistar Rat Models (2024)
-
RANK ligand converts the NCoR/HDAC3 co-repressor to a PGC1β- and RNA-dependent co-activator of osteoclast gene expression (2023)
Collaboration Network
Top Collaborators
Maria Almeida
University of Arkansas for Medical Sciences
7 shared publications
In database
- RANK ligand converts the NCoR/HDAC3 co-repressor to a PGC1β- and RNA-dependent co-activator of osteoclast gene expression
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Showing 5 of 7 shared publications
Aaron Warren
University of Arkansas for Medical Sciences
6 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The adverse effects of chemotherapy on bone mass are not prevented by senolytics
Showing 5 of 6 shared publications
Ha‐Neui Kim
University of Arkansas for Medical Sciences
5 shared publications
In database
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
Md Mohsin Ali
University of Arkansas for Medical Sciences (US)
4 shared publications
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The adverse effects of chemotherapy on bone mass are not prevented by senolytics
Qiang Fu
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Intawat Nookaew
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
Charles A. O’Brien
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Mechanisms of mitochondrial reactive oxygen species action in bone mesenchymal cells
A. Gordon James
University of Arkansas for Medical Sciences
2 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
Landon Gatrell
University of Arkansas for Medical Sciences
2 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
Jinhu Xiong
University of Arkansas for Medical Sciences
2 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
Melda Onal
University of Arkansas for Medical Sciences
2 shared publications
In database
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
Ankita Chalke
University of Arkansas for Medical Sciences
2 shared publications
In database
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
Olivia Reyes‐Castro
University of Arkansas for Medical Sciences
2 shared publications
In database
- The Aging Landscape by <scp>scRNAseq</scp> of Mesenchymal Lineage Cells in Mouse Bone
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
Claudia C.S. Chini
University of Arkansas for Medical Sciences
2 shared publications
In database
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
Farah Alturkmani
University of Arkansas for Medical Sciences
2 shared publications
In database
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
- Estrogens protect bone mass by inhibiting NAD <sup>+</sup> metabolism in osteoclasts
Similar Researchers
Based on overlapping research topics
Grace Wood
University of Arkansas at Fayetteville
Metabolism and Genetic Disorders
Disease Models, Animal
Diet and metabolism studies
Oscar Vicen
University of Arkansas at Fayetteville
Metabolism and Genetic Disorders
Disease Models, Animal
Bone health and treatments
Mahtab Toulany
University of Arkansas for Medical Sciences
Metabolism and Genetic Disorders
Disease Models, Animal
Medical Research
Jin-Ran Chen
University of Arkansas for Medical Sciences
Metabolism and Genetic Disorders
Bone health and treatments
Medical Research
Nell I. Mock
University of Arkansas for Medical Sciences
Metabolism and Genetic Disorders
Disease Models, Animal
Diet and metabolism studies
Amanda L Gwin
University of Arkansas for Medical Sciences
Metabolism and Genetic Disorders
Diet and metabolism studies
Medical Research