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Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
A
A. Gordon James
Graduate Student
Also affiliated: European Bioinformatics Institute (2013); Unilever (United Kingdom) (2004–2023); University of Strathclyde (1994); The Ohio State University Wexner Medical Center (1960); The Ohio State University (1960); Wellcome Trust (2013)
12 h-index
22 pubs
701 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
A. Gordon James's research primarily investigates the biochemical and microbiological origins of human body odor, with a specific focus on axillary malodor. His work has explored the role of cutaneous bacteria in metabolizing fatty acids and generating volatile compounds, such as thioalcohols, which contribute to unpleasant odors. James has also examined the structural basis of malodor precursor transport in the human axilla and investigated the metabolic pathways of fungi like Malassezia globosa and M. restricta in relation to scalp conditions. His publications include studies on the generation of volatile fatty acids by axillary bacteria and the molecular basis of thioalcohol production in body odor. James has a citation count of 701 across 22 publications and an h-index of 12. He has collaborated with researchers Melda Onal, Maria Almeida, Julie Crawford, and James A. Hendrixson, all from the University of Arkansas for Medical Sciences, with multiple shared publications.
Metrics
- h-index: 12
- Publications: 22
- Citations: 701
Positions
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Graduate Student publications 2024–2026University of Arkansas for Medical Sciences Physiology and Cell Biology ORCID
Selected Publications
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Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone (2026)
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The Aging Landscape by scRNAseq of Mesenchymal Lineage Cells in Mouse Bone (2025)
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Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength (2025)
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Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness (2025)
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CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength (2024)
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Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice (2024)
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CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system (2023)
Collaboration Network
Top Collaborators
Melda Onal
University of Arkansas for Medical Sciences
7 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- The Aging Landscape by scRNAseq of Mesenchymal Lineage Cells in Mouse Bone
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
Showing 5 of 7 shared publications
James A. Hendrixson
University of Arkansas for Medical Sciences
4 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Julie Crawford
Institute for Musculoskeletal Health
4 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Intawat Nookaew
University of Arkansas for Medical Sciences
4 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- The Aging Landscape by scRNAseq of Mesenchymal Lineage Cells in Mouse Bone
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
Ilham Kadhim
University of Arkansas for Medical Sciences
4 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
Maria Almeida
University of Arkansas for Medical Sciences
4 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- The Aging Landscape by scRNAseq of Mesenchymal Lineage Cells in Mouse Bone
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Jinhu Xiong
University of Arkansas for Medical Sciences
4 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- The Aging Landscape by scRNAseq of Mesenchymal Lineage Cells in Mouse Bone
- Mitochondrial oxidative stress or decreased autophagy in osteoblast lineage cells is not sufficient to mimic the deleterious effects of aging on bone mechanoresponsiveness
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
Jacob Laster
University of Arkansas for Medical Sciences
3 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
Dominique J. Laster
University of Arkansas for Medical Sciences
2 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
Nisreen Akel
University of Arkansas for Medical Sciences
2 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
Stuart B. Berryhill
University of Arkansas for Medical Sciences
2 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Loss of chaperone‐mediated autophagy does not alter age‐related bone loss in male mice
Jeff D. Thostenson
University of Arkansas for Medical Sciences
2 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- Autophagy in Osx1-Cre-targeted cells is essential for development, growth, and maintenance of bone
Charles A. O’Brien
University of Arkansas for Medical Sciences
2 shared publications
In database
- CRISPR interference provides increased cell type-specificity compared to the Cre-loxP system
- The Aging Landscape by scRNAseq of Mesenchymal Lineage Cells in Mouse Bone
Adriana Lelis Carvalho
University of Arkansas for Medical Sciences
2 shared publications
In database
- The Aging Landscape by scRNAseq of Mesenchymal Lineage Cells in Mouse Bone
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
Jeff Thostenson
University of Arkansas for Medical Sciences
2 shared publications
In database
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength
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