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Biography and Research Information
OverviewAI-generated summary
Jessica L. Kelliher's research focuses on host-pathogen interactions and bacterial gene regulation, particularly involving *Staphylococcus aureus*. Her work investigates how bacterial proteins and metabolic pathways, such as those involving manganese and phosphates, influence the host-pathogen relationship. Kelliher has also explored the role of specific genes, like PCK2, in metabolic plasticity within the context of glioblastoma and CAR-T cell persistence. Her scholarship metrics include an h-index of 8 and 13 total publications, with 689 citations. Kelliher collaborates with researchers at the University of Arkansas for Medical Sciences, including Brian Koss, Daniel Fil, and Lora J. Rogers, with whom she shares multiple publications.
Metrics
- h-index: 8
- Publications: 15
- Citations: 705
Selected Publications
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762 CD28 costimulation induces PCK2 to support T cell effector function in metabolically hostile environments (2025)
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236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response (2025)
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277 Proteome turnover dynamics analysis uncovers E3 ligases that enhance T-cell persistence during exhaustion (2025)
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392 Donor-intrinsic proteomic programs shape CAR-T cell persistence across a longitudinal killing assay (2025)
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Comprehensive Analysis of Proteome Turnover Dynamics During T Cell Exhaustion (2025)
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376 Inhibition of GSK3β-mediated 53BP1 T334 phosphorylation in T cells enhances infiltration and cytotoxicity against head and neck squamous cell carcinoma (2024)
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319 Comprehensive analysis of proteome turnover dynamics during T cell exhaustion (2024)
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929 Proteomic analysis reveals differential modulation of the DNA damage response in exhausted T cells (2024)
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932 Defining the role for PCK2 in T-cell metabolic plasticity (2024)
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Evolved histone tail regulates 53BP1 recruitment at damaged chromatin (2024)
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1020 Proteomic analysis of T cell exhaustion unveils differential modulation of the DNA damage response (2023)
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304 Discovering T cell proteome turnover dynamics to enhance persistence in solid tumors (2023)
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1211 Defining the role of PCK2 in T cell metabolic plasticity in Glioblastoma (2023)
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UBA80 and UBA52 fine-tune RNF168-dependent histone ubiquitination and DNA repair (2023)
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New answers to the old RIDDLE: RNF168 and the DNA damage response pathway (2021)
Collaboration Network
Top Collaborators
Lora J. Rogers
University of Arkansas for Medical Sciences
2 shared publications
In database
- 1211 Defining the role of PCK2 in T cell metabolic plasticity in Glioblastoma
- 392 Donor-intrinsic proteomic programs shape CAR-T cell persistence across a longitudinal killing assay
Daniel Fil
University of Arkansas for Medical Sciences
2 shared publications
In database
- 1211 Defining the role of PCK2 in T cell metabolic plasticity in Glioblastoma
- 392 Donor-intrinsic proteomic programs shape CAR-T cell persistence across a longitudinal killing assay
Sydnye L. Shuttleworth
University of Arkansas for Medical Sciences
2 shared publications
In database
- 1211 Defining the role of PCK2 in T cell metabolic plasticity in Glioblastoma
- 392 Donor-intrinsic proteomic programs shape CAR-T cell persistence across a longitudinal killing assay
Brian Koss
University of Arkansas for Medical Sciences
2 shared publications
In database
- 1211 Defining the role of PCK2 in T cell metabolic plasticity in Glioblastoma
- 392 Donor-intrinsic proteomic programs shape CAR-T cell persistence across a longitudinal killing assay
Latambria Hampton
University of Arkansas for Medical Sciences
1 shared publication
In database
- 1211 Defining the role of PCK2 in T cell metabolic plasticity in Glioblastoma
Michael Bauer
University of Arkansas for Medical Sciences
1 shared publication
In database
- 1211 Defining the role of PCK2 in T cell metabolic plasticity in Glioblastoma
Emilie Darrigues
University of Arkansas for Medical Sciences
1 shared publication
In database
- 1211 Defining the role of PCK2 in T cell metabolic plasticity in Glioblastoma
Analiz Rodriguez
University of Arkansas for Medical Sciences
1 shared publication
In database
- 1211 Defining the role of PCK2 in T cell metabolic plasticity in Glioblastoma
Katherine Cruse
University of Arkansas for Medical Sciences
1 shared publication
In database
- 392 Donor-intrinsic proteomic programs shape CAR-T cell persistence across a longitudinal killing assay
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