Match tier
Likely match
Presence
Current · Arkansas
Last published
2023
Sources
OpenAlex · ORCID
Refreshed
2026-08-08
L
Landen W. Saling
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Researcher
Faculty Researcher
4 h-index
10 pubs
91 cited
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Landen W. Saling's research focuses on the biological sex differences in the development of cancer cachexia, a complex metabolic syndrome characterized by involuntary weight loss and muscle wasting. His work investigates the molecular and physiological alterations that occur in skeletal muscle during this condition, utilizing rodent models. Saling has published studies examining fibrosis development, mitochondrial function, and protein turnover regulators in response to cancer cachexia, highlighting differential impacts between biological sexes. His research also explores the role of specific microRNAs, such as miR-16, in regulating insulin sensitivity and muscle contractile function in a sex-dependent manner. Saling collaborates with several researchers at the University of Arkansas at Fayetteville, including Tyrone A. Washington, Seongkyun Lim, Eleanor R. Schrems, and Nicholas P. Greene, with whom he has co-authored multiple publications.
Metrics
- h-index: 4
- Publications: 10
- Citations: 91
Selected Publications
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Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia (2023)
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Development of skeletal muscle fibrosis in a rodent model of cancer cachexia (2023)
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Biological Sex Differences of Fibrosis During the Development of Cancer Cachexia (2023)
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Differential Induction Of Regulators Of Protein Turnover During C26-induced Cancer Cachexia Between Biological Sexes (2022)
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Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner (2022)
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Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice (2021)
Collaboration Network
Top Collaborators
Seongkyun Lim
University of Arkansas at Fayetteville
6 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Differential Induction Of Regulators Of Protein Turnover During C26-induced Cancer Cachexia Between Biological Sexes
Showing 5 of 6 shared publications
Eleanor R. Schrems
University of Arkansas at Fayetteville
6 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Differential Induction Of Regulators Of Protein Turnover During C26-induced Cancer Cachexia Between Biological Sexes
Showing 5 of 6 shared publications
Tyrone A. Washington
University of Arkansas at Fayetteville
6 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Differential Induction Of Regulators Of Protein Turnover During C26-induced Cancer Cachexia Between Biological Sexes
Showing 5 of 6 shared publications
Nicholas P. Greene
University of Arkansas at Fayetteville
6 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Differential Induction Of Regulators Of Protein Turnover During C26-induced Cancer Cachexia Between Biological Sexes
Showing 5 of 6 shared publications
Megan E. Rosa‐Caldwell
Harvard University
5 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Biological Sex Differences of Fibrosis During the Development of Cancer Cachexia
J. William Deaver
University of Arkansas at Fayetteville (US)
4 shared publications
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Differential Induction Of Regulators Of Protein Turnover During C26-induced Cancer Cachexia Between Biological Sexes
Francielly Morena da Silva
University of Arkansas at Fayetteville
4 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Differential Induction Of Regulators Of Protein Turnover During C26-induced Cancer Cachexia Between Biological Sexes
Ana Regina Cabrera
University of Arkansas at Fayetteville
4 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Differential Induction Of Regulators Of Protein Turnover During C26-induced Cancer Cachexia Between Biological Sexes
Wesley S. Haynie
University of Arkansas at Fayetteville
3 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Biological Sex Differences of Fibrosis During the Development of Cancer Cachexia
Michael P. Wiggs
Baylor University (US)
2 shared publications
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia
David E. Lee
University of Arkansas at Fayetteville (US)
2 shared publications
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
Jacob L. Brown
University of Arkansas at Fayetteville (US)
2 shared publications
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Biological Sex Differences of Fibrosis During the Development of Cancer Cachexia
Richard Perry
University of Arkansas at Fayetteville (US)
2 shared publications
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Biological Sex Differences of Fibrosis During the Development of Cancer Cachexia
Lemuel A. Brown
University of Arkansas at Fayetteville
2 shared publications
In database
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- Biological Sex Differences of Fibrosis During the Development of Cancer Cachexia
Lisa T. Jansen
University of Arkansas at Fayetteville
1 shared publication
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
Similar Researchers
Based on overlapping research topics
Nicholas P. Greene
University of Arkansas at Fayetteville
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders
Mitochondrial Function and Pathology
Megan E. Rosa‐Caldwell
University of Arkansas at Fayetteville
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders
Mitochondrial Function and Pathology
Alan B. Diekman
University of Arkansas for Medical Sciences
Cancer-related molecular mechanisms research
Metabolism and Genetic Disorders
Health disparities and outcomes
Calvin Peterson
University of Arkansas at Fayetteville
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders
Mitochondrial Function and Pathology
Hannah Stovall
Hendrix College
Cancer-related molecular mechanisms research
Mitochondrial Function and Pathology
Metabolism and Genetic Disorders
Thomas S. Risch
Arkansas State University
Cancer-related molecular mechanisms research
Metabolism and Genetic Disorders
Animal Behavior and Welfare Studies