Thomas A. Williams
Senior Assistant Professor
faculty
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Biography and Research Information
OverviewAI-generated summary
Thomas A. Williams is a Senior Assistant Professor at the University of Arkansas for Medical Sciences. His research has focused on understanding and reducing stigma, particularly in the context of mental health and through webinar interventions. He has also contributed to research on T-cell function and immunotherapy, investigating mechanisms such as EZH2 inhibition, CD28 costimulation, and the manipulation of DNA damage response pathways to enhance CAR-T cell persistence and combat T-cell exhaustion.
Williams' scholarly output includes 143 publications, with a total of 3,111 citations, and an h-index of 21. He is recognized as a highly cited researcher. His collaborations include work with Brian Koss, Katherine Wallis, Daniel Fil, and Lora J. Rogers, all from the University of Arkansas for Medical Sciences, with whom he has co-authored three publications each. Williams maintains an active lab website to disseminate his research activities. He has been recently active, with publications in 2025.
Metrics
- h-index: 21
- Publications: 144
- Citations: 3,112
Selected Publications
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685 EZH2 inhibition impairs CD8+ CAR-T cell persistence (2025)
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762 CD28 costimulation induces PCK2 to support T cell effector function in metabolically hostile environments (2025)
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236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response (2025)
Collaboration Network
Top Collaborators
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 762 CD28 costimulation induces PCK2 to support T cell effector function in metabolically hostile environments
- 685 EZH2 inhibition impairs CD8+ CAR-T cell persistence
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 762 CD28 costimulation induces PCK2 to support T cell effector function in metabolically hostile environments
- 685 EZH2 inhibition impairs CD8+ CAR-T cell persistence
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 762 CD28 costimulation induces PCK2 to support T cell effector function in metabolically hostile environments
- 685 EZH2 inhibition impairs CD8+ CAR-T cell persistence
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 762 CD28 costimulation induces PCK2 to support T cell effector function in metabolically hostile environments
- 685 EZH2 inhibition impairs CD8+ CAR-T cell persistence
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 762 CD28 costimulation induces PCK2 to support T cell effector function in metabolically hostile environments
- 685 EZH2 inhibition impairs CD8+ CAR-T cell persistence
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 762 CD28 costimulation induces PCK2 to support T cell effector function in metabolically hostile environments
- Webinar Intervention Research to Reduce Stigma Using a Pre–Post Research Design
- Webinar Intervention Research to Reduce Stigma Using a Pre–Post Research Design
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 236 Manipulating the DNA damage response to combat T cell exhaustion and improve immunotherapy response
- 762 CD28 costimulation induces PCK2 to support T cell effector function in metabolically hostile environments
- 685 EZH2 inhibition impairs CD8+ CAR-T cell persistence
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