Research Areas
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Biography and Research Information
OverviewAI-generated summary
Debopam Ghosh's research focuses on the molecular mechanisms underlying immune responses and cellular processes. His work has investigated the role of B cells as antigen-presenting cells and the regulation of the B cell receptor signalosome by the class II peptide editor H2-M, impacting the development of innate-like B cells. Ghosh has also explored how intrinsic p53 activation restricts gammaherpesvirus-driven germinal center B cell expansion during latency establishment. His publications include studies on NSD2 dimethylation in promoting lung adenocarcinoma pathogenesis and the link between severe delayed hypersensitivity reactions to IL-1 and IL-6 inhibitors and common HLA-DRB1*15 alleles. He has co-authored publications with collaborators Jason S. Stumhofer, J. Craig Forrest, Mark Manzano, and Shana M. Owens, all from the University of Arkansas for Medical Sciences. Ghosh's scholarship metrics include an h-index of 11, with 39 total publications and 574 total citations.
Metrics
- h-index: 11
- Publications: 39
- Citations: 574
Selected Publications
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Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment (2025)
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p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated <i>IgH/c-Myc</i> Translocations (2020)
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IL-17 Promotes Differentiation of Splenic LSK− Lymphoid Progenitors into B Cells following <i>Plasmodium yoelii</i> Infection (2017)
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An Atypical Splenic B Cell Progenitor Population Supports Antibody Production during <i>Plasmodium</i> Infection in Mice (2016)
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The Costimulatory Molecule ICOS Regulates Host Th1 and Follicular Th Cell Differentiation in Response to <i>Plasmodium chabaudi chabaudi</i> AS Infection (2015)
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Signaling Circuits and Regulation of Immune Suppression by Ovarian Tumor-Associated Macrophages (2015)
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Do You See What I See: Recognition of Protozoan Parasites by Toll-Like Receptors (2014)
Collaboration Network
Top Collaborators
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
- Intrinsic p53 activation restricts gammaherpesvirus driven germinal center B cell expansion during latency establishment
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