Match tier
Confirmed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
M
Megan E. Rosa‐Caldwell
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
High Impact
Assistant Professor of Exercise Science
Also affiliated: Beth Israel Deaconess Medical Center (2020–2025); Harvard University (2020–2025); Beth Israel Deaconess Hospital (2023); Baker University (2018)
21 h-index
105 pubs
1,647 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Megan E. Rosa‐Caldwell's research focuses on understanding the mechanisms underlying skeletal muscle wasting, particularly in the context of cancer cachexia. Her work has investigated the role of mitochondrial dysfunction, protein imbalance, and alterations in microRNAs in the progression of muscle atrophy in tumor-bearing mice. She has explored how muscle weakness can precede observable atrophy and is linked to cellular stress within muscle mitochondria.
Her publications also delve into broader aspects of muscle metabolism, including sex-based differences, and examine hepatic alterations during cachexia development. Additionally, her research has touched upon the effects of physical activity on hepatic autophagy in diet-induced obese mice. Rosa‐Caldwell's scholarly contributions are reflected in her h-index of 21 and over 1,640 citations across 104 publications.
She collaborates with several researchers at the University of Arkansas at Fayetteville, including Tyrone A. Washington, Eleanor R. Schrems, Nicholas P. Greene, and Francielly Morena da Silva, with whom she has co-authored multiple publications. Rosa‐Caldwell maintains an active laboratory website to showcase her research.
Metrics
- h-index: 21
- Publications: 105
- Citations: 1,647
Positions
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Assistant Professor of Exercise Science 2024–presentUniversity of Arkansas at Fayetteville Human Health, Performance, and Recreation ORCID
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Post-Doctoral Research Fellow 2020–2023Beth Israel Deaconess Medical Center Neurology ORCID
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Graduate Assisstant 2014–2020University of Arkansas Fayetteville Health Human Performance and Recreation ORCID
Selected Publications
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Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa (2025)
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Divergent alterations in the skeletal muscle and serum proteome in a rodent model of anorexia nervosa and weight recovery (2025)
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Changes in muscle strength and moderators of protein turnover in a rodent model of anorexia nervosa and recovery (2025)
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Muscular Proteomic Alterations During Simulated Anorexia Nervosa Are Not Resolved Following Weight Recovery (2025)
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Electrical impedance myography as a marker of muscle mass in rats with simulated Anorexia Nervosa (2025)
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Brain Matter Alterations during Simulated Anorexia Nervosa and Following Weight Recovery in Rats (2025)
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Effects of Anorexia Nervosa on Muscle Quality Following Long-Term Recovery in Rats (2025)
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Systemic Proteomic Alterations in a Rodent Model of Anorexia Nervosa (2025)
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Muscle Alterations Persist Beyond Weight Restoration In A Rodent Model Of Anorexia Nervosa (2024)
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Predicting muscle function and mass with electrical impedance myography: A study in rat analogs of micro- and partial gravity (2024)
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A refined rodent model of anorexia nervosa: Simulating state‐specific effects of caloric restriction and weight restoration (2024)
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Supplemental table 6 (2023)
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Supplemental table 1 (2023)
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Supplemental table 3 (2023)
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Supplemental table 2 (2023)
Collaboration Network
Top Collaborators
Nicholas P. Greene
University of Arkansas at Fayetteville
62 shared publications
In database
- Mitochondrial degeneration precedes the development of muscle atrophy in progression of cancer cachexia in tumour‐bearing mice
- Muscle metabolism and atrophy: let’s talk about sex
- Protein imbalance in the development of skeletal muscle wasting in tumour‐bearing mice
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Cancer cachexia-induced muscle atrophy: evidence for alterations in microRNAs important for muscle size
Showing 5 of 62 shared publications
Tyrone A. Washington
University of Arkansas at Fayetteville
59 shared publications
In database
- Mitochondrial degeneration precedes the development of muscle atrophy in progression of cancer cachexia in tumour‐bearing mice
- Protein imbalance in the development of skeletal muscle wasting in tumour‐bearing mice
- Cancer cachexia-induced muscle atrophy: evidence for alterations in microRNAs important for muscle size
- Hepatic alterations during the development and progression of cancer cachexia
- Transcriptomic analysis of the development of skeletal muscle atrophy in cancer-cachexia in tumor-bearing mice
Showing 5 of 59 shared publications
Jacob L. Brown
University of Arkansas at Fayetteville (US)
38 shared publications
- Mitochondrial degeneration precedes the development of muscle atrophy in progression of cancer cachexia in tumour‐bearing mice
- Protein imbalance in the development of skeletal muscle wasting in tumour‐bearing mice
- Cancer cachexia-induced muscle atrophy: evidence for alterations in microRNAs important for muscle size
- Hepatic alterations during the development and progression of cancer cachexia
- Transcriptomic analysis of the development of skeletal muscle atrophy in cancer-cachexia in tumor-bearing mice
Showing 5 of 38 shared publications
Richard Perry
University of Arkansas at Fayetteville (US)
33 shared publications
- Mitochondrial degeneration precedes the development of muscle atrophy in progression of cancer cachexia in tumour‐bearing mice
- Protein imbalance in the development of skeletal muscle wasting in tumour‐bearing mice
- Cancer cachexia-induced muscle atrophy: evidence for alterations in microRNAs important for muscle size
- Hepatic alterations during the development and progression of cancer cachexia
- Transcriptomic analysis of the development of skeletal muscle atrophy in cancer-cachexia in tumor-bearing mice
Showing 5 of 33 shared publications
David E. Lee
University of Arkansas at Fayetteville (US)
32 shared publications
- Mitochondrial degeneration precedes the development of muscle atrophy in progression of cancer cachexia in tumour‐bearing mice
- Protein imbalance in the development of skeletal muscle wasting in tumour‐bearing mice
- Cancer cachexia-induced muscle atrophy: evidence for alterations in microRNAs important for muscle size
- Hepatic alterations during the development and progression of cancer cachexia
- Transcriptomic analysis of the development of skeletal muscle atrophy in cancer-cachexia in tumor-bearing mice
Showing 5 of 32 shared publications
Wesley S. Haynie
University of Arkansas at Fayetteville
31 shared publications
In database
- Mitochondrial degeneration precedes the development of muscle atrophy in progression of cancer cachexia in tumour‐bearing mice
- Protein imbalance in the development of skeletal muscle wasting in tumour‐bearing mice
- Hepatic alterations during the development and progression of cancer cachexia
- Transcriptomic analysis of the development of skeletal muscle atrophy in cancer-cachexia in tumor-bearing mice
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
Showing 5 of 31 shared publications
Lemuel A. Brown
University of Michigan
25 shared publications
In database
- Mitochondrial degeneration precedes the development of muscle atrophy in progression of cancer cachexia in tumour‐bearing mice
- Protein imbalance in the development of skeletal muscle wasting in tumour‐bearing mice
- Cancer cachexia-induced muscle atrophy: evidence for alterations in microRNAs important for muscle size
- Transcriptomic analysis of the development of skeletal muscle atrophy in cancer-cachexia in tumor-bearing mice
- Moderate physical activity promotes basal hepatic autophagy in diet-induced obese mice
Showing 5 of 25 shared publications
Michael P. Wiggs
Baylor University (US)
22 shared publications
- Mitochondrial degeneration precedes the development of muscle atrophy in progression of cancer cachexia in tumour‐bearing mice
- Protein imbalance in the development of skeletal muscle wasting in tumour‐bearing mice
- Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress
- Cancer cachexia-induced muscle atrophy: evidence for alterations in microRNAs important for muscle size
- Hepatic alterations during the development and progression of cancer cachexia
Showing 5 of 22 shared publications
Seongkyun Lim
University of Arkansas at Fayetteville
18 shared publications
In database
- Female mice may have exacerbated catabolic signalling response compared to male mice during development and progression of disuse atrophy
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Mitochondrial aberrations during the progression of disuse atrophy differentially affect male and female mice
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Altering aspects of mitochondrial quality to improve musculoskeletal outcomes in disuse atrophy
Showing 5 of 18 shared publications
Eleanor R. Schrems
University of Arkansas at Fayetteville
17 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- Development of skeletal muscle fibrosis in a rodent model of cancer cachexia
- The effect of diet-induced obesity on extracellular matrix remodeling during skeletal muscle regeneration
Showing 5 of 17 shared publications
Francielly Morena da Silva
University of Arkansas at Fayetteville
14 shared publications
In database
- Female mice may have exacerbated catabolic signalling response compared to male mice during development and progression of disuse atrophy
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- PGC-1α overexpression is not sufficient to mitigate cancer cachexia in either male or female mice
Showing 5 of 14 shared publications
Ana Regina Cabrera
13 shared publications
In database
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Muscle miR-16 deletion results in impaired insulin sensitivity and contractile function in a sex-dependent manner
- PGC-1α overexpression is not sufficient to mitigate cancer cachexia in either male or female mice
- Leucine Supplementation Exacerbates Morbidity in Male but Not Female Mice with Colorectal Cancer-Induced Cachexia
Showing 5 of 13 shared publications
Kevin A. Murach
University of Arkansas at Fayetteville
13 shared publications
In database
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Biological sex divergence in transcriptomic profiles during the onset of hindlimb unloading-induced atrophy
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa
- Divergent alterations in the skeletal muscle and serum proteome in a rodent model of anorexia nervosa and weight recovery
Showing 5 of 13 shared publications
Lisa T. Jansen
University of Arkansas at Fayetteville
12 shared publications
In database
- Female mice may have exacerbated catabolic signalling response compared to male mice during development and progression of disuse atrophy
- Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice
- Mitochondrial aberrations during the progression of disuse atrophy differentially affect male and female mice
- Altering aspects of mitochondrial quality to improve musculoskeletal outcomes in disuse atrophy
- Comparative plasma proteomics in muscle atrophy during cancer‐cachexia and disuse: The search for atrokines
Showing 5 of 12 shared publications
Aaron R. Caldwell
University of Arkansas at Fayetteville
11 shared publications
In database
- Hepatic alterations during the development and progression of cancer cachexia
- Prolonged standing increases lower limb arterial stiffness
- Moving Sport and Exercise Science Forward: A Call for the Adoption of More Transparent Research Practices
- Combined Effects of Standing and Passive Heating on Attenuating Sitting-Induced Vascular Dysfunction
- Confidence in eating disorder knowledge does not predict actual knowledge in collegiate female athletes
Showing 5 of 11 shared publications
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