Jennifer Chen
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Researcher
Also affiliated: Rutgers, The State University of New Jersey (2023); Broad Institute (2021); Brigham Young University–Hawaii (2019); University of Vermont (2024); Brigham and Women's Hospital (2000–2011); San Francisco General Hospital (2023); The University of Sydney (2019–2024); Hartford Hospital (2010); Queensland Health (2022); University of Minnesota (2020–2021); San Francisco VA Medical Center (2006); Gladstone Institutes (2005–2006); University of Nevada, Reno (2001–2004); Harvard University (2001–2018); University of Connecticut (2010–2023); University of British Columbia (2008); Johns Hopkins University (2020); University of Auckland (2015); Central Connecticut State University (2019); University of Alberta (2016); Queensland University of Technology (2008); University of California, San Francisco (2005–2023); Baylor College of Medicine (2023); University of Toronto (2004–2024); University of Washington (2024); Washington University in St. Louis (2008–2023); Cornell University (2017); University of Maryland Eastern Shore (2015); Brown University (2025); University of Michigan (2013); Royal Brisbane and Women's Hospital (2022); Lenox Hill Hospital (2011–2012); Hospital for Sick Children (2011–2016); Harvard University Press (2015); Canada Research Chairs (2004); Johnson University (2023); Baylor University Medical Center (1993); Lankenau Medical Center (2024); Japan Display (Japan) (2021); Yale University (2018–2025); University of California San Diego (2023); University of New Haven (2021); Massachusetts General Hospital (2018); St. Francis Hospital (2023); Blood Systems Research Institute (2006); Rutgers Sexual and Reproductive Health and Rights (2023); Harvard College Observatory (2020); Sidney Kimmel Cancer Center (2016–2020); National Public Radio (2015); Yale New Haven Health System (2021); Creative Commons (2015); Maurice Wilkins Centre (2015); City Eye Centre (2008); Winthrop Rockefeller Foundation (2024); Washington University Medical Center (2008); SickKids Foundation (2011); Westmead Institute for Medical Research (2019–2024); Arkansas Children's Nutrition Center (2024–2025); AstraZeneca (United States) (2010); University of California Davis Medical Center (2006); Saint Louis University (2018); Drexel University (2010–2018); Rice University (2012); UConn Health (2023); Columbia University (2016–2022); University of Pennsylvania (2010–2025); University of California, Davis (2010–2022); The University of Texas at Austin (2023); The University of Texas Southwestern Medical Center (2010); University of California, Berkeley (2003); Stanford University (2023); McMaster University (2022–2023)
Faculty Researcher
Upstream record may be merged OpenAlex, the source of these figures, lists 74 institutions in 6 countries for this author record — a pattern that usually means it combines several researchers with similar names. The totals above may include work by other people.
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Jennifer Chen's research focuses on understanding disease mechanisms and immune responses, with a significant portion of her recent work investigating SARS-CoV-2 infection and its impact. She has published studies on the cellular targets of SARS-CoV-2 in human airway epithelium, identifying alterations in gene expression and cell state changes. Further research has explored the role of nonsteroidal anti-inflammatory drugs in modulating the cytokine and antibody response to SARS-CoV-2, as well as the generation of high-affinity neutralizing antibodies in the absence of T follicular helper cells.
Beyond viral infections, Chen's research interests extend to other areas of human health. She has contributed to work on integrative oncogene-dependency mapping to identify vulnerabilities and synergies in lung cancer, and has investigated the role of Nox4 expression in osteo-progenitors during bone development in mice. Additionally, her work includes a systematic review on the efficacy of treatments in reducing inflammatory lesion count in rosacea. Chen leads a research group and has a substantial publication record, evidenced by an h-index of 34 and over 5,900 citations. She collaborates with researchers from the University of Arkansas at Fayetteville and the University of Arkansas for Medical Sciences.
Metrics
- h-index: 34
- Publications: 153
- Citations: 5,988
Selected Publications
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Soy Isoflavones Prevent Bone Quality Loss Induced by High‐Fat Diet in Rats Through Epigenetic Modifications (2025)
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Soy Isoflavones Prevent Loss of Bone Quality Associated With High Fat Diet in Rats Through Ameliorating Epigenetic Modifications in Bone (2025)
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Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption (2025)
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Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx (2024)
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Phenolic acids prevent sex-steroid deficiency-induced bone loss and bone marrow adipogenesis in mice (2024)
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Nox4 expression in osteo-progenitors controls bone development in mice during early life (2022)
Collaboration Network
Top Collaborators
- Nox4 expression in osteo-progenitors controls bone development in mice during early life
- Phenolic acids prevent sex-steroid deficiency-induced bone loss and bone marrow adipogenesis in mice
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Soy Isoflavones Prevent Bone Quality Loss Induced by High‐Fat Diet in Rats Through Epigenetic Modifications
- Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx
Showing 5 of 6 shared publications
- Nox4 expression in osteo-progenitors controls bone development in mice during early life
- Phenolic acids prevent sex-steroid deficiency-induced bone loss and bone marrow adipogenesis in mice
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Soy Isoflavones Prevent Bone Quality Loss Induced by High‐Fat Diet in Rats Through Epigenetic Modifications
- Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx
Showing 5 of 6 shared publications
- Nox4 expression in osteo-progenitors controls bone development in mice during early life
- Phenolic acids prevent sex-steroid deficiency-induced bone loss and bone marrow adipogenesis in mice
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Soy Isoflavones Prevent Bone Quality Loss Induced by High‐Fat Diet in Rats Through Epigenetic Modifications
- Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx
- Nox4 expression in osteo-progenitors controls bone development in mice during early life
- Phenolic acids prevent sex-steroid deficiency-induced bone loss and bone marrow adipogenesis in mice
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Phenolic acids prevent sex-steroid deficiency-induced bone loss and bone marrow adipogenesis in mice
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Soy Isoflavones Prevent Bone Quality Loss Induced by High‐Fat Diet in Rats Through Epigenetic Modifications
- Nox4 expression in osteo-progenitors controls bone development in mice during early life
- Phenolic acids prevent sex-steroid deficiency-induced bone loss and bone marrow adipogenesis in mice
- Phenolic acids prevent sex-steroid deficiency-induced bone loss and bone marrow adipogenesis in mice
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx
- Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx
- Soy Isoflavones Prevent Loss of Bone Quality Associated With High Fat Diet in Rats Through Ameliorating Epigenetic Modifications in Bone
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx
- Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption
- Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx
- Soy Isoflavones Prevent Bone Quality Loss Induced by High‐Fat Diet in Rats Through Epigenetic Modifications
- Soy Isoflavones Prevent Loss of Bone Quality Associated With High Fat Diet in Rats Through Ameliorating Epigenetic Modifications in Bone
- Nox4 expression in osteo-progenitors controls bone development in mice during early life
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