Ronald G. Jones
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Also affiliated: Georgia State University (1978–1990); Tripler Army Medical Center (1991–1993); Jamestown Education Foundation (1993)
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Ronald G. Jones' research investigates molecular mechanisms underlying skeletal muscle adaptation, aging, and disease states. His work has explored the transcriptomic changes associated with exercise in human skeletal muscle, identifying MYC as a key regulator of muscle growth and an epigenetic reprogramming factor that combats skeletal muscle aging. Jones has also studied cancer cachexia, examining its temporal progression and distinct molecular signatures in male and female mice, and its association with neuromuscular dysfunction and mitochondrial stress in ovarian cancer models. His research utilizes animal models, including mice, to understand these processes, with a focus on the interplay between genetics, molecular biology, and physiological outcomes. Jones has published 60 papers, with an h-index of 17 and over 900 citations, and collaborates frequently with researchers at the University of Arkansas at Fayetteville.
Metrics
- h-index: 15
- Publications: 41
- Citations: 839
Selected Publications
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Differential impact of cancer- and chemotherapy-induced cachexia: a comparative analysis in a preclinical model of colorectal cancer by biological sex (2026)
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Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations (2026)
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TRAIL: A potential new biomarker for muscle health in anorexia nervosa (2026)
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Ribosome dynamics during skeletal muscle repair and regeneration in mice and humans (2026)
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The Age‐Dependent Resident Myonuclear Multi‐Omic Response to an Acute Skeletal Muscle Hypertrophic Stimulus in Mice (2026)
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Myonuclear loss, rather than senescent myonuclei, associates with fiber type-specific atrophy in aging human skeletal muscle (2026)
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Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia (2025)
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The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus (2025)
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Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity (2025)
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Making sense of MYC in skeletal muscle: location, duration, and magnitude (2025)
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The Skeletal Muscle Proteomic Signature of Long-Term Repeated Transient MYC Induction (2025)
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The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth (2024)
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Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex (2024)
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Muscle weakness and mitochondrial stress occur before severe metastasis in a novel mouse model of ovarian cancer cachexia (2024)
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Muscle weakness and mitochondrial stress occur before metastasis in a novel mouse model of ovarian cancer cachexia (2024)
Collaboration Network
Top Collaborators
- A molecular signature defining exercise adaptation with ageing and in vivo partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Neuromuscular Dysfunction Precedes Cognitive Impairment in a Mouse Model of Alzheimer’s Disease
- Exercise-Induced MYC as an Epigenetic Reprogramming Factor That Combats Skeletal Muscle Aging
Showing 5 of 21 shared publications
- A molecular signature defining exercise adaptation with ageing and in vivo partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Muscle weakness and mitochondrial stress occur before severe metastasis in a novel mouse model of ovarian cancer cachexia
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
Showing 5 of 12 shared publications
- A molecular signature defining exercise adaptation with ageing and in vivo partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates MYC as a Hypertrophic Regulator That is Sufficient for Growth
Showing 5 of 10 shared publications
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- The Age‐Dependent Resident Myonuclear Multi‐Omic Response to an Acute Skeletal Muscle Hypertrophic Stimulus in Mice
- Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
Showing 5 of 7 shared publications
- A molecular signature defining exercise adaptation with ageing and in vivo partial reprogramming in skeletal muscle
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates MYC as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia
Showing 5 of 7 shared publications
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Exercise-Induced MYC as an Epigenetic Reprogramming Factor That Combats Skeletal Muscle Aging
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates MYC as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Ribosome dynamics during skeletal muscle repair and regeneration in mice and humans
Showing 5 of 6 shared publications
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity
- A molecular signature defining exercise adaptation with ageing and in vivo partial reprogramming in skeletal muscle
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates MYC as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates MYC as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Ribosome dynamics during skeletal muscle repair and regeneration in mice and humans
- Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations
- A molecular signature defining exercise adaptation with ageing and in vivo partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
- A molecular signature defining exercise adaptation with ageing and in vivo partial reprogramming in skeletal muscle
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates MYC as a Hypertrophic Regulator That is Sufficient for Growth
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
- A molecular signature defining exercise adaptation with ageing and in vivo partial reprogramming in skeletal muscle
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
- A molecular signature defining exercise adaptation with ageing and in vivo partial reprogramming in skeletal muscle
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
- Myonuclear loss, rather than senescent myonuclei, associates with fiber type-specific atrophy in aging human skeletal muscle
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates MYC as a Hypertrophic Regulator That is Sufficient for Growth
- Ribosome dynamics during skeletal muscle repair and regeneration in mice and humans
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