Match tier
Likely match
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-08-08
R
Ronald G. Jones
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Researcher
Also affiliated: Pennsylvania State University (1963); University of Leeds (2001); Georgia State University (1978–1990); University of Kent (2001–2005); Tripler Army Medical Center (1991–1993); Warwickshire College (1969); Jacobson Holman (United States) (1993); Joint Educational Facilities (1993); Coventry (United Kingdom) (1969); JDSU (United States) (1993)
Faculty Researcher
17 h-index
60 pubs
952 cited
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Ronald G. Jones' research investigates molecular mechanisms underlying skeletal muscle adaptation, aging, and disease states. His work has explored the transcriptomic changes associated with exercise in human skeletal muscle, identifying MYC as a key regulator of muscle growth and an epigenetic reprogramming factor that combats skeletal muscle aging. Jones has also studied cancer cachexia, examining its temporal progression and distinct molecular signatures in male and female mice, and its association with neuromuscular dysfunction and mitochondrial stress in ovarian cancer models. His research utilizes animal models, including mice, to understand these processes, with a focus on the interplay between genetics, molecular biology, and physiological outcomes. Jones has published 60 papers, with an h-index of 17 and over 900 citations, and collaborates frequently with researchers at the University of Arkansas at Fayetteville.
Metrics
- h-index: 17
- Publications: 60
- Citations: 952
Selected Publications
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Differential impact of cancer- and chemotherapy-induced cachexia: a comparative analysis in a preclinical model of colorectal cancer by biological sex (2026)
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Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations (2026)
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TRAIL: A potential new biomarker for muscle health in anorexia nervosa (2026)
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Ribosome dynamics during skeletal muscle repair and regeneration in mice and humans (2026)
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The Age‐Dependent Resident Myonuclear Multi‐Omic Response to an Acute Skeletal Muscle Hypertrophic Stimulus in Mice (2026)
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Myonuclear loss, rather than senescent myonuclei, associates with fiber type-specific atrophy in aging human skeletal muscle (2026)
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Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia (2025)
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The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus (2025)
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Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity (2025)
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Making sense of MYC in skeletal muscle: location, duration, and magnitude (2025)
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The Skeletal Muscle Proteomic Signature of Long-Term Repeated Transient MYC Induction (2025)
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The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth (2024)
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Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex (2024)
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Muscle weakness and mitochondrial stress occur before severe metastasis in a novel mouse model of ovarian cancer cachexia (2024)
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Muscle weakness and mitochondrial stress occur before metastasis in a novel mouse model of ovarian cancer cachexia (2024)
Collaboration Network
Top Collaborators
Kevin A. Murach
University of Arkansas at Fayetteville
19 shared publications
In database
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Neuromuscular Dysfunction Precedes Cognitive Impairment in a Mouse Model of Alzheimer’s Disease
- Muscle weakness and mitochondrial stress occur before severe metastasis in a novel mouse model of ovarian cancer cachexia
Showing 5 of 19 shared publications
Nicholas P. Greene
University of Arkansas at Fayetteville
12 shared publications
In database
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Muscle weakness and mitochondrial stress occur before severe metastasis in a novel mouse model of ovarian cancer cachexia
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
Showing 5 of 12 shared publications
Francielly Morena da Silva
University of Arkansas at Fayetteville
10 shared publications
In database
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
Showing 5 of 10 shared publications
Ana Regina Cabrera
University of Arkansas at Fayetteville
7 shared publications
In database
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
- Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia
Showing 5 of 7 shared publications
Yuan Wen
University of Kentucky (US)
7 shared publications
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
Showing 5 of 7 shared publications
Eleanor R. Schrems
University of Arkansas at Fayetteville
5 shared publications
In database
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia
- Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity
Tyrone A. Washington
University of Arkansas at Fayetteville
5 shared publications
In database
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- Skeletal muscle methylome-transcriptome disruptions during the onset and progression of colorectal cancer-induced cachexia
- Transcriptomic analysis demonstrates moderators of muscle quality are altered in age-related sarcopenic obesity
Christopher S. Fry
University of Kentucky (US)
5 shared publications
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
Ferdinand von Walden
Karolinska Institutet (SE)
5 shared publications
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Exercise-Induced MYC as an Epigenetic Reprogramming Factor That Combats Skeletal Muscle Aging
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- Insight on the loading-mediated regulation of Runx1 in skeletal muscle
Seongkyun Lim
University of Arkansas at Fayetteville
4 shared publications
In database
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
Sabin Khadgi
University of Arkansas at Fayetteville
4 shared publications
In database
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
John J. McCarthy
University of Kentucky (US)
4 shared publications
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
Cory M. Dungan
Baylor University (US)
3 shared publications
- A molecular signature defining exercise adaptation with ageing and <i>in vivo</i> partial reprogramming in skeletal muscle
- A Rejuvenation Signature in Skeletal Muscle That Is Mediated By Late-Life Exercise
- Myonuclear loss, rather than senescent myonuclei, associates with fiber type-specific atrophy in aging human skeletal muscle
Sebastian Edman
Karolinska Institutet (SE)
3 shared publications
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Making sense of MYC in skeletal muscle: location, duration, and magnitude
Megan E. Rosa‐Caldwell
University of Arkansas at Fayetteville
2 shared publications
In database
- The time-course of cancer cachexia onset reveals biphasic transcriptional disruptions in female skeletal muscle distinct from males
- The Time-Course of Cancer Cachexia Onset Reveals Biphasic Transcriptional Disruptions in Female Skeletal Muscle Distinct from Males
Similar Researchers
Based on overlapping research topics
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