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Confirmed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-06
Q
Qiang Gu
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
High Impact
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Also affiliated: University of British Columbia Hospital (2000–2002); United States Food and Drug Administration (2012–2026); Harvard University (2016); University of British Columbia (1992–2005); The University of Queensland (2008); University of Calgary (2000); Vancouver Hospital and Health Sciences Centre (2000–2002); Tianjin Medical University General Hospital (2013–2017); Massachusetts General Hospital (2016); Athinoula A. Martinos Center for Biomedical Imaging (2016); Max Planck Institute for Brain Research (1989–1995); Entry Exit Inspection and Quarantine Bureau (2016); Wake Forest University Health Sciences (2003–2014); Wake Forest University (2003–2014); Tianjin Medical University (2009–2017)
27 h-index
109 pubs
3,574 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Qiang Gu's research has focused on the neurobiology of cortical plasticity, particularly within the visual cortex. His work has investigated the role of neuromodulatory transmitter systems in cortical plasticity and the impact of NMDA receptor antagonists on experience-dependent synaptic modifications. Studies have examined how blocking these receptors affects ocularity changes in the visual cortex following monocular deprivation in kittens.
Gu's research also includes work with laboratory animals such as mice and zebrafish, exploring various biological processes. He has contributed to the development and updates of the Galaxy platform, a tool for biomedical analyses that emphasizes accessibility, reproducibility, and collaboration. His scholarly output includes over 240 publications, with a citation count exceeding 7,300, and an h-index of 38, designating him as a highly cited researcher. He leads a research group and maintains an active lab website.
Metrics
- h-index: 27
- Publications: 109
- Citations: 3,574
Selected Publications
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N-Acetylcysteine Prevents Arsenic-Induced Apoptosis but Not Supernumerary Motor Neuron Development in Zebrafish Embryos: Assessment of Protein Carbonylation and the p53 Pathway (2026)
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The effects of cannabidiol and its main metabolites on human neural stem cells (2025)
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Neurogenic Effects of Inorganic Arsenic and Cdk5 Knockdown in Zebrafish Embryos: A Perspective on Modeling Autism (2024)
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Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish (2023)
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Combining tissue clearing and Fluoro-Jade C labeling for neurotoxicity assessments (2023)
Collaboration Network
Top Collaborators
Jyotshna Kanungo
National Center for Toxicological Research
16 shared publications
In database
- Downregulation of 14-3-3 Proteins in Alzheimer’s Disease
- Distinct effects of ketamine and acetyl l-carnitine on the dopamine system in zebrafish
- Mechanistic studies on ketamine-induced mitochondrial toxicity in zebrafish embryos
- Downregulation of 14-3-3 Proteins in a Kainic Acid-Induced Neurotoxicity Model
- Ketamine-induced attenuation of reactive oxygen species in zebrafish is prevented by acetyl l-carnitine in vivo
Showing 5 of 16 shared publications
Merle G. Paule
National Center for Toxicological Research
10 shared publications
In database
- Distinct effects of ketamine and acetyl l-carnitine on the dopamine system in zebrafish
- Mechanistic studies on ketamine-induced mitochondrial toxicity in zebrafish embryos
- Downregulation of 14-3-3 Proteins in a Kainic Acid-Induced Neurotoxicity Model
- Lipidomics reveals a systemic energy deficient state that precedes neurotoxicity in neonatal monkeys after sevoflurane exposure
- Cyclosporine exacerbates ketamine toxicity in zebrafish: Mechanistic studies on drug–drug interaction
Showing 5 of 10 shared publications
Syed F. Ali
National Center for Toxicological Research
9 shared publications
In database
- Distinct effects of ketamine and acetyl l-carnitine on the dopamine system in zebrafish
- Mechanistic studies on ketamine-induced mitochondrial toxicity in zebrafish embryos
- Ketamine-induced attenuation of reactive oxygen species in zebrafish is prevented by acetyl l-carnitine in vivo
- Cyclosporine exacerbates ketamine toxicity in zebrafish: Mechanistic studies on drug–drug interaction
- Acetyl L ‐carnitine targets adenosine triphosphate synthase in protecting zebrafish embryos from toxicities induced by verapamil and ketamine: An in vivo assessment
Showing 5 of 9 shared publications
Bonnie Robinson
National Center for Toxicological Research
9 shared publications
In database
- Distinct effects of ketamine and acetyl l-carnitine on the dopamine system in zebrafish
- Mechanistic studies on ketamine-induced mitochondrial toxicity in zebrafish embryos
- Ketamine-induced attenuation of reactive oxygen species in zebrafish is prevented by acetyl l-carnitine in vivo
- Cyclosporine exacerbates ketamine toxicity in zebrafish: Mechanistic studies on drug–drug interaction
- N-acetylcysteine prevents ketamine-induced adverse effects on development, heart rate and monoaminergic neurons in zebrafish
Showing 5 of 9 shared publications
Sumit Sarkar
National Center for Toxicological Research
7 shared publications
In database
- Downregulation of 14-3-3 Proteins in Alzheimer’s Disease
- Downregulation of 14-3-3 Proteins in a Kainic Acid-Induced Neurotoxicity Model
- One-step labeling of degenerative neurons in unfixed brain tissue samples using Fluoro-Jade C
- ERK/MAP Kinase Activation is Evident in Activated Microglia of the Striatum and Substantia Nigra in an Acute and Chronically-Induced Mouse Model of Parkinson’s Disease
- In vitro detection of cytotoxicity using FluoroJade-C
Showing 5 of 7 shared publications
Melanie Dumas
Bionetics (United States) (US)
7 shared publications
- Distinct effects of ketamine and acetyl l-carnitine on the dopamine system in zebrafish
- Mechanistic studies on ketamine-induced mitochondrial toxicity in zebrafish embryos
- Ketamine-induced attenuation of reactive oxygen species in zebrafish is prevented by acetyl l-carnitine in vivo
- Cyclosporine exacerbates ketamine toxicity in zebrafish: Mechanistic studies on drug–drug interaction
- N-acetylcysteine prevents ketamine-induced adverse effects on development, heart rate and monoaminergic neurons in zebrafish
Showing 5 of 7 shared publications
Elvis Cuevas
National Center for Toxicological Research (US)
4 shared publications
- Downregulation of 14-3-3 Proteins in Alzheimer’s Disease
- Distinct effects of ketamine and acetyl l-carnitine on the dopamine system in zebrafish
- In vitro detection of cytotoxicity using FluoroJade-C
- An Alternative In Vitro Method for Examining Nanoparticle-Induced Cytotoxicity
James Raymick
National Center for Toxicological Research
4 shared publications
In database
- Downregulation of 14-3-3 Proteins in Alzheimer’s Disease
- Downregulation of 14-3-3 Proteins in a Kainic Acid-Induced Neurotoxicity Model
- ERK/MAP Kinase Activation is Evident in Activated Microglia of the Striatum and Substantia Nigra in an Acute and Chronically-Induced Mouse Model of Parkinson’s Disease
- Decreased Mcl-1 protein level in the striatum of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-treated mice
Cheng Wang
National Center for Toxicological Research
4 shared publications
In database
- Lipidomics reveals a systemic energy deficient state that precedes neurotoxicity in neonatal monkeys after sevoflurane exposure
- Application of microRNA profiling to understand sevoflurane-induced adverse effects on developing monkey brain
- The effects of cannabidiol and its main metabolites on human neural stem cells
- The NMDA Receptors: Physiology and Neurotoxicity in the Developing Brain
Fang Liu
National Center for Toxicological Research
3 shared publications
In database
- Lipidomics reveals a systemic energy deficient state that precedes neurotoxicity in neonatal monkeys after sevoflurane exposure
- Application of microRNA profiling to understand sevoflurane-induced adverse effects on developing monkey brain
- The effects of cannabidiol and its main metabolites on human neural stem cells
Shuliang Liu
National Center for Toxicological Research
3 shared publications
In database
- Lipidomics reveals a systemic energy deficient state that precedes neurotoxicity in neonatal monkeys after sevoflurane exposure
- Application of microRNA profiling to understand sevoflurane-induced adverse effects on developing monkey brain
- The effects of cannabidiol and its main metabolites on human neural stem cells
Joseph P. Hanig
Center for Drug Evaluation and Research (US)
3 shared publications
- Lipidomics reveals a systemic energy deficient state that precedes neurotoxicity in neonatal monkeys after sevoflurane exposure
- ERK/MAP Kinase Activation is Evident in Activated Microglia of the Striatum and Substantia Nigra in an Acute and Chronically-Induced Mouse Model of Parkinson’s Disease
- Application of microRNA profiling to understand sevoflurane-induced adverse effects on developing monkey brain
Bryan Raymick
National Center for Toxicological Research
2 shared publications
In database
- One-step labeling of degenerative neurons in unfixed brain tissue samples using Fluoro-Jade C
- Combining tissue clearing and Fluoro-Jade C labeling for neurotoxicity assessments
Edward Lu
University of New Mexico (US)
2 shared publications
- ERK/MAP Kinase Activation is Evident in Activated Microglia of the Striatum and Substantia Nigra in an Acute and Chronically-Induced Mouse Model of Parkinson’s Disease
- Decreased Mcl-1 protein level in the striatum of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-treated mice
Tucker A. Patterson
National Center for Toxicological Research
2 shared publications
In database
- Lipidomics reveals a systemic energy deficient state that precedes neurotoxicity in neonatal monkeys after sevoflurane exposure
- Application of microRNA profiling to understand sevoflurane-induced adverse effects on developing monkey brain
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