Azra Dad Data-verified
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Research Participation Program/ ORISE Postdoctoral Fellow
postdoc
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Biography and Research Information
OverviewAI-generated summary
Azra Dad is a postdoctoral fellow at the National Center for Toxicological Research (NCTR) through the Research Participation Program/ORISE. Her research focuses on genotoxicity assessment, particularly investigating the effects of chemical mutagens in animal models. Dad has published work examining the dose-response relationship of ethyl methanesulfonate-induced genotoxicity across different life stages and tissues in rats. Her research also explores background mutation frequencies in cell lines such as TK6 and L5178Y, with implications for error-corrected sequencing.
Further contributions include studies on *Pig-a* gene mutations in the bone marrow granulocytes of rats treated with procarbazine. Her scholarly output includes 19 publications with 327 citations and an h-index of 10. Dad collaborates with researchers at NCTR, including Javier R. Revollo, Robert H. Heflich, and Mason G. Pearce, with whom she shares multiple publications.
Metrics
- h-index: 10
- Publications: 19
- Citations: 330
Selected Publications
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Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing (2025)
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Effect of life stage and target tissue on dose–response assessment of ethyl methane sulfonate‐induced genotoxicity (2021)
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<i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats (2021)
Collaboration Network
Top Collaborators
- <i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
- Effect of life stage and target tissue on dose–response assessment of ethyl methane sulfonate‐induced genotoxicity
- <i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats
- Effect of life stage and target tissue on dose–response assessment of ethyl methane sulfonate‐induced genotoxicity
- <i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats
- <i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats
- <i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats
- Effect of life stage and target tissue on dose–response assessment of ethyl methane sulfonate‐induced genotoxicity
- Effect of life stage and target tissue on dose–response assessment of ethyl methane sulfonate‐induced genotoxicity
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
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